Signal transduction through CsrRS confers an invasive phenotype in group A Streptococcus.
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The Abi-domain protein Abx1 interacts with the CovS histidine kinase to control virulence gene expression in group B Streptococcus.Dual-site phosphorylation of the control of virulence regulator impacts group a streptococcal global gene expression and pathogenesisStreptolysin O and NAD-glycohydrolase prevent phagolysosome acidification and promote group A Streptococcus survival in macrophages.The small regulatory RNA FasX controls pilus expression and adherence in the human bacterial pathogen group A StreptococcusTwo-component system response regulators involved in virulence of Streptococcus pneumoniae TIGR4 in infective endocarditis.The AgI/II family adhesin AspA is required for respiratory infection by Streptococcus pyogenes.The human antimicrobial peptide LL-37 binds directly to CsrS, a sensor histidine kinase of group A Streptococcus, to activate expression of virulence factors.The two-component system GrvRS (EtaRS) regulates ace expression in Enterococcus faecalis OG1RF.Comparative genomics analysis of Streptococcus isolates from the human small intestine reveals their adaptation to a highly dynamic ecosystemCharacterization of the effect of the histidine kinase CovS on response regulator phosphorylation in group A Streptococcus.Mutations in the control of virulence sensor gene from Streptococcus pyogenes after infection in mice lead to clonal bacterial variants with altered gene regulatory activity and virulence.Cationic antimicrobial peptide resistance mechanisms of streptococcal pathogens.Vitamin D and the human antimicrobial peptide LL-37 enhance group a streptococcus resistance to killing by human cellsRepression of Rgg But Not Upregulation of LacD.1 in emm1-type covS Mutant Mediates the SpeB Repression in Group A Streptococcus.Molecular characterization of an invasive phenotype of group A Streptococcus arising during human infection using whole genome sequencing of multiple isolates from the same patient.Bacterial Mg2+ homeostasis, transport, and virulence.Use of a Phosphorylation Site Mutant To Identify Distinct Modes of Gene Repression by the Control of Virulence Regulator (CovR) in Streptococcus pyogenes.Host Antimicrobial Peptides in Bacterial Homeostasis and Pathogenesis of Disease.Group A Streptococcus encounters with host macrophages.Two-component signal transduction systems in oral bacteria.The C. difficile clnRAB operon initiates adaptations to the host environment in response to LL-37
P2860
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P2860
Signal transduction through CsrRS confers an invasive phenotype in group A Streptococcus.
description
2011 nî lūn-bûn
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2011 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի հոտեմբերին հրատարակված գիտական հոդված
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2011年の論文
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2011年論文
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2011年論文
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2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
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2011年论文
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name
Signal transduction through CsrRS confers an invasive phenotype in group A Streptococcus.
@ast
Signal transduction through CsrRS confers an invasive phenotype in group A Streptococcus.
@en
Signal transduction through CsrRS confers an invasive phenotype in group A Streptococcus.
@nl
type
label
Signal transduction through CsrRS confers an invasive phenotype in group A Streptococcus.
@ast
Signal transduction through CsrRS confers an invasive phenotype in group A Streptococcus.
@en
Signal transduction through CsrRS confers an invasive phenotype in group A Streptococcus.
@nl
prefLabel
Signal transduction through CsrRS confers an invasive phenotype in group A Streptococcus.
@ast
Signal transduction through CsrRS confers an invasive phenotype in group A Streptococcus.
@en
Signal transduction through CsrRS confers an invasive phenotype in group A Streptococcus.
@nl
P2093
P2860
P1433
P1476
Signal transduction through CsrRS confers an invasive phenotype in group A Streptococcus
@en
P2093
Hien J Tran-Winkler
Ioannis Gryllos
Michael R Wessels
P2860
P304
P356
10.1371/JOURNAL.PPAT.1002361
P50
P577
2011-10-27T00:00:00Z