Shear stress regulates the endothelial Kir2.1 ion channel. - Wikidata - awgldk - TriplyDB

Shear stress regulates the endothelial Kir2.1 ion channel.

Shear stress regulates the endothelial Kir2.1 ion channel.

http://www.wikidata.org/entity/Q34065567

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Functional expression of inward rectifier potassium channels in cultured human pulmonary smooth muscle cells: evidence for a major role of Kir2.4 subunits Stop the flow: a paradigm for cell signaling mediated by reactive oxygen species in the pulmonary endothelium Flow-dependent cellular mechanotransduction in atherosclerosis.The role of cellular adaptation to mechanical forces in atherosclerosis.Mechanotransduction in vascular physiology and atherogenesis.Fluid flow facilitates inward rectifier K+ current by convectively restoring [K+] at the cell membrane surface.PDGF-BB and TGF-{beta}1 on cross-talk between endothelial and smooth muscle cells in vascular remodeling induced by low shear stress.Expression and Analysis of Flow-regulated Ion Channels in Xenopus Oocytes.Effect of aging on cellular mechanotransduction Physiological mechanisms of vascular response induced by shear stress and effect of exercise in systemic and placental circulation A microfluidic shear device that accommodates parallel high and low stress zones within the same culturing chamber.Collateralization and the response to obstruction of epicardial coronary arteries.Shear stress-related mechanosignaling with lung ischemia: lessons from basic research can inform lung transplantation.Potassium channels in the peripheral microcirculation.Effect of membrane tension on the electric field and dipole potential of lipid bilayer membrane.Shear stress mediates exocytosis of functional TRPV4 channels in endothelial cells.Fluid flows and forces in development: functions, features and biophysical principles.Cdo Regulates Surface Expression of Kir2.1 K+ Channel in Myoblast Differentiation Ca2+-activated K+ channels in murine endothelial cells: block by intracellular calcium and magnesium.Activation of the Caenorhabditis elegans Degenerin Channel by Shear Stress Requires the MEC-10 Subunit.Molecular mechanisms responsible for the atheroprotective effects of laminar shear stress.Loss of syndecan-1 induces a pro-inflammatory phenotype in endothelial cells with a dysregulated response to atheroprotective flow.Hemodynamic forces in endothelial dysfunction and vascular aging.Mechanotransduction in embryonic vascular development.Mechanotransduction in the endothelium: role of membrane proteins and reactive oxygen species in sensing, transduction, and transmission of the signal with altered blood flow Regulation of the Epithelial Na(+) Channel by Paraoxonase-2.Flow-induced endothelial cell alignment requires the RhoGEF Trio as a scaffold protein to polarize active Rac1 distribution.Boosting the signal: Endothelial inward rectifier K+ channels.Ion Channels in Endothelial Responses to Fluid Shear Stress.Effects of shear stress on endothelial cells: go with the flow.Endothelial Mechanosignaling: Does One Sensor Fit All?Contribution of K(ir)2 potassium channels to ATP-induced cell death in brain capillary endothelial cells and reconstructed HEK293 cell model.Molecular characterization of an inward rectifier channel (IKir) found in avian vestibular hair cells: cloning and expression of pKir2.1.Sodium azide dilates coronary arterioles via activation of inward rectifier K+ channels and Na+-K+-ATPase.Shear stress-induced endothelial cell polarization is mediated by Rho and Rac but not Cdc42 or PI 3-kinases.Differential effects of pulsatile versus steady flow on coronary endothelial membrane potential.Flow-activated chloride channels in vascular endothelium. Shear stress sensitivity, desensitization dynamics, and physiological implications.Adrenergic origin of very low-frequency blood pressure oscillations in the unanesthetized rat.N-linked Glycans are Required on Epithelial Na+ Channel Subunits for Maturation and Surface Expression.Expression and function of inwardly rectifying potassium channels after experimental subarachnoid hemorrhage.

P2860

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P2860

Shear stress regulates the endothelial Kir2.1 ion channel.

http://www.wikidata.org/entity/Q34065567

description

2002 nî lūn-bûn

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2002 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2002 թվականի մայիսին հրատարակված գիտական հոդված

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2002年の論文

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2002年論文

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2002年論文

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2002年論文

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2002年論文

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2002年論文

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2002年论文

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name

Shear stress regulates the endothelial Kir2.1 ion channel.

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Shear stress regulates the endothelial Kir2.1 ion channel.

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Shear stress regulates the endothelial Kir2.1 ion channel.

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type

label

Shear stress regulates the endothelial Kir2.1 ion channel.

@ast

Shear stress regulates the endothelial Kir2.1 ion channel.

@en

Shear stress regulates the endothelial Kir2.1 ion channel.

@nl

prefLabel

Shear stress regulates the endothelial Kir2.1 ion channel.

@ast

Shear stress regulates the endothelial Kir2.1 ion channel.

@en

Shear stress regulates the endothelial Kir2.1 ion channel.

@nl

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Proceedings of the National Academy of Sciences of the United States of America

P1476

Shear stress regulates the endothelial Kir2.1 ion channel.

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P2093

Anne Hoger

http://www.w3.org/2001/XMLSchema#string

Jeff H Hoger

http://www.w3.org/2001/XMLSchema#string

Scott Forsyth

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Victor I Ilyin

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P2860

Improved patch-clamp techniques for high-resolution current recording from cells and cell-free membrane patches

Targeting of an A kinase-anchoring protein, AKAP79, to an inwardly rectifying potassium channel, Kir2.1

Human homologue of the Drosophila discs large tumor suppressor binds to p56lck tyrosine kinase and Shaker type Kv1.3 potassium channel in T lymphocytes

Neuronal inwardly rectifying K(+) channels differentially couple to PDZ proteins of the PSD-95/SAP90 family

Inward rectifier potassium channel Kir2.2 is associated with synapse-associated protein SAP97

Clustering and enhanced activity of an inwardly rectifying potassium channel, Kir4.1, by an anchoring protein, PSD-95/SAP90

Binding of the inward rectifier K+ channel Kir 2.3 to PSD-95 is regulated by protein kinase A phosphorylation

Flow activates an endothelial potassium channel to release an endogenous nitrovasodilator

Flow-mediated endothelial mechanotransduction.

Ca2+ influx through stretch-activated cation channels activates maxi K+ channels in porcine endocardial endothelium

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7780-7785

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10.1073/PNAS.102184999

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11

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99

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12032360

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124350

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