CXCR4/YY1 inhibition impairs VEGF network and angiogenesis during malignancy. - Wikidata - awgldk - TriplyDB

CXCR4/YY1 inhibition impairs VEGF network and angiogenesis during malignancy.

CXCR4/YY1 inhibition impairs VEGF network and angiogenesis during malignancy.

http://www.wikidata.org/entity/Q34069720

about

Hydrogels to model 3D in vitro microenvironment of tumor vascularization Yin Yang 1 contributes to gastric carcinogenesis and its nuclear expression correlates with shorter survival in patients with early stage gastric adenocarcinoma The oncogenic role of Yin Yang 1 Competitive binding between Seryl-tRNA synthetase/YY1 complex and NFKB1 at the distal segment results in differential regulation of human vegfa promoter activity during angiogenesis.Yin Yang-1 suppresses invasion and metastasis of pancreatic ductal adenocarcinoma by downregulating MMP10 in a MUC4/ErbB2/p38/MEF2C-dependent mechanism.VEGFR2 translocates to the nucleus to regulate its own transcription.YY1 overexpression is associated with poor prognosis and metastasis-free survival in patients suffering osteosarcoma.YY1-MIR372-SQSTM1 regulatory axis in autophagy Dual inhibition of plasminogen kringle 5 on angiogenesis and chemotaxis suppresses tumor metastasis by targeting HIF-1α pathway.Visceral endoderm expression of Yin-Yang1 (YY1) is required for VEGFA maintenance and yolk sac development.CXCL12 (SDF1alpha)-CXCR4/CXCR7 pathway inhibition: an emerging sensitizer for anticancer therapies?Inhibition of angiogenesis, fibrosis and thrombosis by tetramethylpyrazine: mechanisms contributing to the SDF-1/CXCR4 axis.Inflammation-induced endothelial cell-derived extracellular vesicles modulate the cellular status of pericytes.Antiangiogenic Effects and Therapeutic Targets of Azadirachta indica Leaf Extract in Endothelial Cells.Stromal cell-derived factor 1α mediates resistance to mTOR-directed therapy in pancreatic cancer New Cross-Talk Layer between Ultraconserved Non-Coding RNAs, MicroRNAs and Polycomb Protein YY1 in Bladder Cancer CXCR4 downregulation of let-7a drives chemoresistance in acute myeloid leukemia.Ex vivo behaviour of human bone tumor endothelial cells Yin Yang 1 promotes mTORC2-mediated AKT phosphorylation.Directed in vivo angiogenesis assay and the study of systemic neoangiogenesis in cancer.Spontaneous reversion of the angiogenic phenotype to a nonangiogenic and dormant state in human tumors Genome-wide survey by ChIP-seq reveals YY1 regulation of lincRNAs in skeletal myogenesis.Osteosarcoma cells induce endothelial cell proliferation during neo-angiogenesis.Yin Yang-1 suppresses differentiation of hepatocellular carcinoma cells through the downregulation of CCAAT/enhancer-binding protein alpha.Different expression of CD146 in human normal and osteosarcoma cell lines.Regulation of VEGF, MMP-9 and metastasis by CXCR4 in a prostate cancer cell line.Transcription factor Yin Yang 2 is a novel regulator of the p53/p21 axis Involvement of Src in the Adaptation of Cancer Cells under Microenvironmental Stresses.p38/Sp1/Sp4/HDAC4/BDNF Axis Is a Novel Molecular Pathway of the Neurotoxic Effect of the Methylmercury It is not necessarily YY1--the frequently forgotten Yin-Yang-2 transcription factor.Identification of valid reference housekeeping genes for gene expression analysis in tumor neovascularization studies.Reply to Klar: Key role of YY1 in neoangiogenesis.Post-surgery fluids promote transition of cancer stem cell-to-endothelial and AKT/mTOR activity, contributing to relapse of giant cell tumors of bone.YY1 suppresses proliferation and migration of pancreatic ductal adenocarcinoma by regulating the CDKN3/MdM2/P53/P21 signaling pathway.Extract of white button mushroom affects skin healing and angiogenesis.The MiR-135b-BMAL1-YY1 loop disturbs pancreatic clockwork to promote tumourigenesis and chemoresistance.Sarcoma Spheroids and Organoids-Promising Tools in the Era of Personalized Medicine.Association between SDF1-3'A or CXCR4 gene polymorphisms with predisposition to and clinicopathological characteristics of prostate cancer with or without metastases.Dkk-1 promotes angiogenic responses and cartilage matrix proteinase secretion in synovial fibroblasts from osteoarthritic joints.

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P2860

CXCR4/YY1 inhibition impairs VEGF network and angiogenesis during malignancy.

http://www.wikidata.org/entity/Q34069720

description

2010 nî lūn-bûn

@nan

2010 թուականի Յուլիսին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի հուլիսին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

CXCR4/YY1 inhibition impairs VEGF network and angiogenesis during malignancy.

@ast

CXCR4/YY1 inhibition impairs VEGF network and angiogenesis during malignancy.

@en

CXCR4/YY1 inhibition impairs VEGF network and angiogenesis during malignancy.

@nl

type

label

CXCR4/YY1 inhibition impairs VEGF network and angiogenesis during malignancy.

@ast

CXCR4/YY1 inhibition impairs VEGF network and angiogenesis during malignancy.

@en

CXCR4/YY1 inhibition impairs VEGF network and angiogenesis during malignancy.

@nl

prefLabel

CXCR4/YY1 inhibition impairs VEGF network and angiogenesis during malignancy.

@ast

CXCR4/YY1 inhibition impairs VEGF network and angiogenesis during malignancy.

@en

CXCR4/YY1 inhibition impairs VEGF network and angiogenesis during malignancy.

@nl

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PNAS.1008256107

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Proceedings of the National Academy of Sciences of the United States of America

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CXCR4/YY1 inhibition impairs VEGF network and angiogenesis during malignancy.

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Alfredo Siani

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Andrea Fiore

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Diana C Marquez-Garban

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Francesco Cacciatore

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Letizia Cito

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Linda Sommese

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Mohammed Al-Omran

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Toshio Hayashi

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Valeria Crudele

http://www.w3.org/2001/XMLSchema#string

P2860

The biology of VEGF and its receptors

Patterns and emerging mechanisms of the angiogenic switch during tumorigenesis

Tumor angiogenesis

Tumorigenesis and the angiogenic switch

VEGF-targeted therapy: mechanisms of anti-tumour activity.

Adverse effects of anticancer agents that target the VEGF pathway.

CXCR4/CXCL12 axis promotes VEGF-mediated tumor angiogenesis through Akt signaling pathway

Transcription factor YY1: structure, function, and therapeutic implications in cancer biology.

Novel challenges in exploring peptide ligands and corresponding tissue-specific endothelial receptors.

VEGF-A splicing: the key to anti-angiogenic therapeutics?

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14484-14489

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10.1073/PNAS.1008256107

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32

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107

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Antonella Petrillo

Antonio Barbieri

Francesca Pentimalli

Alfonso Giovane

Antonio Giordano

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2010-07-26T00:00:00Z

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45366345

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20660740

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2922579

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