Metabolic imprinting on genetically predisposed neural circuits perpetuates obesity.
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Metabolic imprinting: critical impact of the perinatal environment on the regulation of energy homeostasisStress exposure, food intake and emotional stateStage-specific inhibition of TrkB activity leads to long-lasting and sexually dimorphic effects on body weight and hypothalamic gene expressionChanges in melanocortin expression and inflammatory pathways in fetal offspring of nonhuman primates fed a high-fat dietPerinatal environment and its influences on metabolic programming of offspring.Developmental gene x environment interactions affecting systems regulating energy homeostasis and obesity.Effects of Chronic Social Stress on ObesityFurther insights into the neurobiology of melanin-concentrating hormone in energy and mood balances.Nutrition and pubertal development.Gene-environment interactions controlling energy and glucose homeostasis and the developmental origins of obesityGCN2 in the brain programs PPARγ2 and triglyceride storage in the liver during perinatal development in response to maternal dietary fat.Increased susceptibility to metabolic alterations in young adult females exposed to early malnutritionSelf-regulation of energy intake in the prevention and treatment of obesity: is it feasible?Central regulation of energy balance: inputs, outputs and leptin resistance.Impairment of acquisition of cocaine self-administration in rats maintained on a high-fat diet.Exposure to smoke during development: fetal programming of adult disease.Prenatal stress or high-fat diet increases susceptibility to diet-induced obesity in rat offspring.Parental influence on children's early eating environments and obesity risk: implications for prevention.Leptin prevents obesity induced by a high-fat diet after diet-induced weight loss in the marsupial S. crassicaudata.Expression of energy balance regulatory genes in the developing ovine fetal hypothalamus at midgestation and the influence of hyperglycemia.Assortative weight gain in mother-daughter and father-son pairs: an emerging source of childhood obesity. Longitudinal study of trios (EarlyBird 43).Factors associated with newborn in-hospital weight loss: comparisons by feeding method, demographics, and birthing procedures.Role of fat amount and type in ameliorating diet-induced obesity: insights at the level of hypothalamic arcuate nucleus leptin receptor, neuropeptide Y and pro-opiomelanocortin mRNA expression.
P2860
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P2860
Metabolic imprinting on genetically predisposed neural circuits perpetuates obesity.
description
2000 nî lūn-bûn
@nan
2000 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2000 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
2000年の論文
@ja
2000年論文
@yue
2000年論文
@zh-hant
2000年論文
@zh-hk
2000年論文
@zh-mo
2000年論文
@zh-tw
2000年论文
@wuu
name
Metabolic imprinting on genetically predisposed neural circuits perpetuates obesity.
@ast
Metabolic imprinting on genetically predisposed neural circuits perpetuates obesity.
@en
Metabolic imprinting on genetically predisposed neural circuits perpetuates obesity.
@nl
type
label
Metabolic imprinting on genetically predisposed neural circuits perpetuates obesity.
@ast
Metabolic imprinting on genetically predisposed neural circuits perpetuates obesity.
@en
Metabolic imprinting on genetically predisposed neural circuits perpetuates obesity.
@nl
prefLabel
Metabolic imprinting on genetically predisposed neural circuits perpetuates obesity.
@ast
Metabolic imprinting on genetically predisposed neural circuits perpetuates obesity.
@en
Metabolic imprinting on genetically predisposed neural circuits perpetuates obesity.
@nl
P1433
P1476
Metabolic imprinting on genetically predisposed neural circuits perpetuates obesity.
@en
P2093
P304
P356
10.1016/S0899-9007(00)00408-1
P577
2000-10-01T00:00:00Z