Analysis of the biochemical mechanisms for the endocrine actions of fibroblast growth factor-23 - Wikidata - awgldk - TriplyDB

Analysis of the biochemical mechanisms for the endocrine actions of fibroblast growth factor-23

Analysis of the biochemical mechanisms for the endocrine actions of fibroblast growth factor-23

http://www.wikidata.org/entity/Q34075026

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The FGF23-Klotho axis: endocrine regulation of phosphate homeostasis FGF23-mediated regulation of systemic phosphate homeostasis: is Klotho an essential player?Klotho and aging Receptor specificity of the fibroblast growth factor family. The complete mammalian FGF family Klotho in cardiovascular disease: Current and future perspectives Skeletal secretion of FGF-23 regulates phosphate and vitamin D metabolism Regulation and function of the FGF23/klotho endocrine pathways Fibroblast Growth Factor 23: A New Dimension to Diseases of Calcium-Phosphorus Metabolism Role of FGF23 in vitamin D and phosphate metabolism: implications in chronic kidney disease Cardiovascular Risk Factors and Chronic Kidney Disease-FGF23: A Key Molecule in the Cardiovascular Disease Fibroblast growth factor 23 impairs phosphorus and vitamin D metabolism in vivo and suppresses 25-hydroxyvitamin D-1alpha-hydroxylase expression in vitro FGF-23-Klotho signaling stimulates proliferation and prevents vitamin D-induced apoptosis Molecular mechanisms of fibroblast growth factor signaling in physiology and pathology Regulation of fibroblast growth factor-23 signaling by klotho Fibroblast growth factor 23 and Klotho: physiology and pathophysiology of an endocrine network of mineral metabolism Circulating fibroblast growth factor 23 in patients with end-stage renal disease treated by peritoneal dialysis is intact and biologically active Fibroblast growth factor 23 and mortality among patients undergoing hemodialysis Mineral metabolism and aging: the fibroblast growth factor 23 enigma.Fibroblast growth factor 23 and left ventricular hypertrophy in chronic kidney disease.FGF23 decreases renal NaPi-2a and NaPi-2c expression and induces hypophosphatemia in vivo predominantly via FGF receptor 1.The associations of fibroblast growth factor 23 and uncarboxylated matrix Gla protein with mortality in coronary artery disease: the Heart and Soul Study.FGF23 induces left ventricular hypertrophy FGF23 is a novel regulator of intracellular calcium and cardiac contractility in addition to cardiac hypertrophy.Vitamin D: Metabolism, Molecular Mechanism of Action, and Pleiotropic Effects.Klotho inhibits transforming growth factor-beta1 (TGF-beta1) signaling and suppresses renal fibrosis and cancer metastasis in mice FGF-23 as a predictor of renal outcome in diabetic nephropathy Compound deletion of Fgfr3 and Fgfr4 partially rescues the Hyp mouse phenotype.Common genetic variants associate with serum phosphorus concentration.Skeletal overgrowth is mediated by deficiency in a specific isoform of fibroblast growth factor receptor 3.Conditional Deletion of Fgfr1 in the Proximal and Distal Tubule Identifies Distinct Roles in Phosphate and Calcium Transport.Phosphate: known and potential roles during development and regeneration of teeth and supporting structures.A homozygous missense mutation in human KLOTHO causes severe tumoral calcinosis.FGF23 neutralization improves chronic kidney disease-associated hyperparathyroidism yet increases mortality Effects of gonadal steroid withdrawal on serum phosphate and FGF-23 levels in men Conversion of a paracrine fibroblast growth factor into an endocrine fibroblast growth factor.Assessment of 24,25(OH)2D levels does not support FGF23-mediated catabolism of vitamin D metabolites Premature aging in klotho mutant mice: cause or consequence?Pathogenic role of Fgf23 in Dmp1-null mice.The emerging role of the fibroblast growth factor-23-klotho axis in renal regulation of phosphate homeostasis Insights from genetic disorders of phosphate homeostasis

P2860

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P2860

Analysis of the biochemical mechanisms for the endocrine actions of fibroblast growth factor-23

http://www.wikidata.org/entity/Q34075026

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2005 nî lūn-bûn

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2005 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2005 թվականի օգոստոսին հրատարակված գիտական հոդված

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2005年の論文

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2005年論文

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2005年論文

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2005年論文

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2005年論文

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2005年論文

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2005年论文

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name

Analysis of the biochemical me ...... of fibroblast growth factor-23

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Analysis of the biochemical me ...... of fibroblast growth factor-23

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Analysis of the biochemical me ...... of fibroblast growth factor-23

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Analysis of the biochemical me ...... of fibroblast growth factor-23

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Analysis of the biochemical me ...... of fibroblast growth factor-23

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Analysis of the biochemical me ...... of fibroblast growth factor-23

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Analysis of the biochemical me ...... of fibroblast growth factor-23

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Analysis of the biochemical me ...... of fibroblast growth factor-23

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Analysis of the biochemical me ...... of fibroblast growth factor-23

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Analysis of the biochemical me ...... of fibroblast growth factor-23

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Fuming Zhang

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Holly J Garringer

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Kenneth E White

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Omar A Ibrahimi

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Siobhan I Davis

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P2860

Autosomal dominant hypophosphataemic rickets is associated with mutations in FGF23

FGF-23 inhibits renal tubular phosphate transport and is a PHEX substrate

The cytoplasmic region of the erythropoietin receptor contains nonoverlapping positive and negative growth-regulatory domains

FGF-23 is a potent regulator of vitamin D metabolism and phosphate homeostasis

Receptor specificity of the fibroblast growth factor family

Fibroblast growth factor receptor 3 is a negative regulator of bone growth

Cloning and characterization of FGF23 as a causative factor of tumor-induced osteomalacia

Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT Method

Fibroblast growth factor (FGF)-23 inhibits renal phosphate reabsorption by activation of the mitogen-activated protein kinase pathway

The autosomal dominant hypophosphatemic rickets R176Q mutation in fibroblast growth factor 23 resists proteolytic cleavage and enhances in vivo biological potency

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4647-4656

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scholarly article

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10.1210/EN.2005-0670

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11

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146

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Moosa Mohammadi

David M. Ornitz

Robert J. Linhardt

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2005-08-04T00:00:00Z

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16081635

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4140631

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