Candesartan reduces the innate immune response to lipopolysaccharide in human monocytes - Wikidata - awgldk - TriplyDB

Candesartan reduces the innate immune response to lipopolysaccharide in human monocytes

Candesartan reduces the innate immune response to lipopolysaccharide in human monocytes

http://www.wikidata.org/entity/Q34087095

about

Candesartan, an angiotensin II AT₁-receptor blocker and PPAR-γ agonist, reduces lesion volume and improves motor and memory function after traumatic brain injury in mice.Angiotensin II AT1 receptor blockade ameliorates brain inflammation.Telmisartan ameliorates lipopolysaccharide-induced innate immune response through peroxisome proliferator-activated receptor-γ activation in human monocytes Angiotensin II AT(1) receptor blockers ameliorate inflammatory stress: a beneficial effect for the treatment of brain disorders.Minocycline ameliorates LPS-induced inflammation in human monocytes by novel mechanisms including LOX-1, Nur77 and LITAF inhibition.An integrative genome-wide transcriptome reveals that candesartan is neuroprotective and a candidate therapeutic for Alzheimer's disease Angiotensin AT2 receptor stimulation is anti-inflammatory in lipopolysaccharide-activated THP-1 macrophages via increased interleukin-10 production.Telmisartan ameliorates glutamate-induced neurotoxicity: roles of AT(1) receptor blockade and PPARγ activation Evidence to Consider Angiotensin II Receptor Blockers for the Treatment of Early Alzheimer's Disease.The effect of high glucose on the inhibitory action of C21, a selective AT2R agonist, of LPS-stimulated tissue factor expression in human mononuclear cells.Different protective and reparative effects of olmesartan in stroke according to time of administration and withdrawal.Presence and regulation of insulin-regulated aminopeptidase in mouse macrophages.Cell separation by countercurrent centrifugal elutriation: recent developments.Angiotensin-(1-7) decreases LPS-induced inflammatory response in macrophages.

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Candesartan reduces the innate immune response to lipopolysaccharide in human monocytes

http://www.wikidata.org/entity/Q34087095

description

2009 nî lūn-bûn

@nan

2009 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2009 թվականի դեկտեմբերին հրատարակված գիտական հոդված

@hy

2009年の論文

@ja

2009年論文

@yue

2009年論文

@zh-hant

2009年論文

@zh-hk

2009年論文

@zh-mo

2009年論文

@zh-tw

2009年论文

@wuu

name

Candesartan reduces the innate immune response to lipopolysaccharide in human monocytes

@ast

Candesartan reduces the innate immune response to lipopolysaccharide in human monocytes

@en

Candesartan reduces the innate immune response to lipopolysaccharide in human monocytes

@nl

type

label

Candesartan reduces the innate immune response to lipopolysaccharide in human monocytes

@ast

Candesartan reduces the innate immune response to lipopolysaccharide in human monocytes

@en

Candesartan reduces the innate immune response to lipopolysaccharide in human monocytes

@nl

prefLabel

Candesartan reduces the innate immune response to lipopolysaccharide in human monocytes

@ast

Candesartan reduces the innate immune response to lipopolysaccharide in human monocytes

@en

Candesartan reduces the innate immune response to lipopolysaccharide in human monocytes

@nl

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Journal of Hypertension

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Candesartan reduces the innate immune response to lipopolysaccharide in human monocytes

@en

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Enrique Sánchez-Lemus

http://www.w3.org/2001/XMLSchema#string

Jaroslav Pavel

http://www.w3.org/2001/XMLSchema#string

Juan M Saavedra

http://www.w3.org/2001/XMLSchema#string

Julius Benicky

http://www.w3.org/2001/XMLSchema#string

Tao Pang

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P2860

Common angiotensin receptor blockers may directly modulate the immune system via VDR, PPAR and CCR2b

Toll-like receptors

Cutting edge: direct interaction of TLR4 with NAD(P)H oxidase 4 isozyme is essential for lipopolysaccharide-induced production of reactive oxygen species and activation of NF-kappa B

Angiotensin II increases human monocyte matrix metalloproteinase-1 through the AT2 receptor and prostaglandin E2: implications for atherosclerotic plaque rupture

Genomic organization and regulation of expression of the lectin-like oxidized low-density lipoprotein receptor (LOX-1) gene

The expression of the lectin-like oxidized low-density lipoprotein receptor (LOX-1) on human vascular smooth muscle cells and monocytes and its down-regulation by lovastatin

Angiotensin II stimulates endothelial vascular cell adhesion molecule-1 via nuclear factor-kappaB activation induced by intracellular oxidative stress

Angiotensin II stimulates intercellular adhesion molecule-1 (ICAM-1) expression by human vascular endothelial cells and increases soluble ICAM-1 release in vivo

Differential effect of insulin treatment on decreased levels of beta-defensins and Toll-like receptors in diabetic rats

NF-kappaB: a key role in inflammatory diseases

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2365-2376

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scholarly article

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10.1097/HJH.0B013E3283314BC7

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12

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27

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Ignacio M Larrayoz

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2009-12-01T00:00:00Z

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19730394

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lipopolysaccharide

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2928995

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