Mitochondrial DNA damage: molecular marker of vulnerable nigral neurons in Parkinson's disease - Wikidata - awgldk - TriplyDB

Mitochondrial DNA damage: molecular marker of vulnerable nigral neurons in Parkinson's disease

Mitochondrial DNA damage: molecular marker of vulnerable nigral neurons in Parkinson's disease

http://www.wikidata.org/entity/Q34090228

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LC/MS analysis of cardiolipins in substantia nigra and plasma of rotenone-treated rats: implication for mitochondrial dysfunction in Parkinson's disease Mitochondrial Biology and Neurological Diseases Live-cell imaging approaches for the investigation of xenobiotic-induced oxidant stress,Mitochondrial DNA damage as a peripheral biomarker for mitochondrial toxin exposure in rats Exposure to mitochondrial genotoxins and dopaminergic neurodegeneration in Caenorhabditis elegans Parkinson Disease and Malignant Disease: Minding Cancer's Own Business.Inefficient DNA Repair Is an Aging-Related Modifier of Parkinson's Disease.Lmx1a and Lmx1b regulate mitochondrial functions and survival of adult midbrain dopaminergic neurons α-Synuclein binds to TOM20 and inhibits mitochondrial protein import in Parkinson's disease.SMN deficiency in severe models of spinal muscular atrophy causes widespread intron retention and DNA damage The Parkinson Disease Mitochondrial Hypothesis: Where Are We at?The role of DNA base excision repair in brain homeostasis and disease.Neuroprotective effects of (E)-3,4-diacetoxystyryl sulfone and sulfoxide derivatives in vitro models of Parkinson's disease.Molecular changes in the postmortem parkinsonian brain.Biological functions of selenium and its potential influence on Parkinson's disease.Calcium and Parkinson's disease.Chronic oxidative damage together with genome repair deficiency in the neurons is a double whammy for neurodegeneration: Is damage response signaling a potential therapeutic target?Risky repair: DNA-protein crosslinks formed by mitochondrial base excision DNA repair enzymes acting on free radical lesions.Base excision repair variants and pesticide exposure increase Parkinson's disease risk.Role of mitochondrial DNA damage and dysfunction in veterans with Gulf War Illness.Mitoapocynin Treatment Protects Against Neuroinflammation and Dopaminergic Neurodegeneration in a Preclinical Animal Model of Parkinson's Disease.From the Cover: Alterations in Optineurin Expression and Localization in Pre-clinical Parkinson's Disease Models DNA strand breaks in peripheral blood leucocytes of Polish blood donors.Relationship between mitochondrial DNA A10398G polymorphism and Parkinson's disease: a meta-analysis.Investigation into experimental toxicological properties of plant protection products having a potential link to Parkinson's disease and childhood leukaemia†Role of Estrogen and Other Sex Hormones in Brain Aging. Neuroprotection and DNA Repair.Mitochondrial DNA depletion by ethidium bromide decreases neuronal mitochondrial creatine kinase: Implications for striatal energy metabolism.RAD52 is required for RNA-templated recombination repair in post-mitotic neurons.Evidence for a Pan-Neurodegenerative Disease Response in Huntington's and Parkinson's Disease Expression Profiles.An adverse outcome pathway for parkinsonian motor deficits associated with mitochondrial complex I inhibition.Botanical Drug Puerarin Attenuates 6-Hydroxydopamine (6-OHDA)-Induced Neurotoxicity via Upregulating Mitochondrial Enzyme Arginase-2.Translational approaches to restoring mitochondrial function in Parkinson's disease.Simplified qPCR method for detecting excessive mtDNA damage induced by exogenous factors.LRRK2 G2019S-induced mitochondrial DNA damage is LRRK2 kinase dependent and inhibition restores mtDNA integrity in Parkinson's disease.Reduced apurinic/apyrimidinic endonuclease 1 activity and increased DNA damage in mitochondria are related to enhanced apoptosis and inflammation in the brain of senescence- accelerated P8 mice (SAMP8).Mitochondrial Dysfunction in Parkinson's Disease: New Mechanistic Insights and Therapeutic Perspectives.Autophagy Disruptions Associated With Altered Optineurin Expression in Extranigral Regions in a Rotenone Model of Parkinson's Disease.AMBRA1-Mediated Mitophagy Counteracts Oxidative Stress and Apoptosis Induced by Neurotoxicity in Human Neuroblastoma SH-SY5Y Cells.Comparative Analysis of Mutant Huntingtin Binding Partners in Yeast Species.The Sources of Reactive Oxygen Species and Its Possible Role in the Pathogenesis of Parkinson's Disease

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Mitochondrial DNA damage: molecular marker of vulnerable nigral neurons in Parkinson's disease

http://www.wikidata.org/entity/Q34090228

description

2014 nî lūn-bûn

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2014 թուականի Յունիսին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի հունիսին հրատարակված գիտական հոդված

@hy

2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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name

Mitochondrial DNA damage: mole ...... neurons in Parkinson's disease

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Mitochondrial DNA damage: mole ...... neurons in Parkinson's disease

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Mitochondrial DNA damage: mole ...... neurons in Parkinson's disease

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Mitochondrial DNA damage: mole ...... neurons in Parkinson's disease

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Mitochondrial DNA damage: mole ...... neurons in Parkinson's disease

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Mitochondrial DNA damage: mole ...... neurons in Parkinson's disease

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Mitochondrial DNA damage: mole ...... neurons in Parkinson's disease

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Mitochondrial DNA damage: mole ...... neurons in Parkinson's disease

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Mitochondrial DNA damage: mole ...... neurons in Parkinson's disease

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J.NBD.2014.06.014

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Neurobiology of Disease

P1476

Mitochondrial DNA damage: mole ...... neurons in Parkinson's disease

@en

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Bennett Van Houten

http://www.w3.org/2001/XMLSchema#string

Bryan C Dickinson

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Christopher J Chang

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Jennifer McCoy

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Laurie H Sanders

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Pier G Mastroberardino

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Xiaoping Hu

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P2860

Instability and decay of the primary structure of DNA

Rotenone, paraquat, and Parkinson's disease

Chemistry and biology of reactive oxygen species in signaling or stress responses

Gene-environment interactions in Parkinson's disease: specific evidence in humans and mammalian models

After the banquet: mitochondrial biogenesis, mitophagy, and cell survival

Oxidant stress evoked by pacemaking in dopaminergic neurons is attenuated by DJ-1.

Chronic systemic pesticide exposure reproduces features of Parkinson's disease

High levels of mitochondrial DNA deletions in substantia nigra neurons in aging and Parkinson disease

A highly reproducible rotenone model of Parkinson's disease

Preparation and use of MitoPY1 for imaging hydrogen peroxide in mitochondria of live cells

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214-223

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scholarly article

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10.1016/J.NBD.2014.06.014

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70

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Charleen T. Chu

J. Timothy Greenamyre

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2014-06-27T00:00:00Z

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263550514

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24981012

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Parkinson's disease

mitochondrial DNA

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4144978

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