Hyperglycemia activates p53 and p53-regulated genes leading to myocyte cell death. - Wikidata - awgldk - TriplyDB

Hyperglycemia activates p53 and p53-regulated genes leading to myocyte cell death.

Hyperglycemia activates p53 and p53-regulated genes leading to myocyte cell death.

http://www.wikidata.org/entity/Q34092744

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MicroRNA dysregulation in diabetic ischemic heart failure patients Diabetic Cardiovascular Disease Induced by Oxidative Stress Update on RAAS Modulation for the Treatment of Diabetic Cardiovascular Disease Carbonic anhydrase activation is associated with worsened pathological remodeling in human ischemic diabetic cardiomyopathy Therapeutic inhibition of mitochondrial reactive oxygen species with mito-TEMPO reduces diabetic cardiomyopathy Enhanced apoptotic propensity in diabetic cardiac mitochondria: influence of subcellular spatial location Small changes huge impact: the role of protein posttranslational modifications in cellular homeostasis and disease Diminished GATA4 protein levels contribute to hyperglycemia-induced cardiomyocyte injury p38 MAP kinases in the heart Influence of glucosamine on glomerular mesangial cell turnover: implications for hyperglycemia and hexosamine pathway flux Sirtuin1-p53, forkhead box O3a, p38 and post-infarct cardiac remodeling in the spontaneously diabetic Goto-Kakizaki rat Hyperglycemic and hyperlipidemic conditions alter cardiac cell biomechanical properties Late-gestation ventricular myocardial reduction in fetuses of hyperglycemic CD1 mice is associated with increased apoptosis.Impact of conditioning hyperglycemic on myocardial infarction rats: Cardiac cell survival factors.A nonerythropoietic derivative of erythropoietin protects the myocardium from ischemia-reperfusion injury O-GlcNAcylation/phosphorylation cycling at Ser10 controls both transcriptional activity and stability of delta-lactoferrin.Cardiac transcription factor Nkx2.5 is downregulated under excessive O-GlcNAcylation condition.Coronary microvascular dysfunction in diabetes mellitus: A review Activation of the hexosamine biosynthesis pathway and protein O-GlcNAcylation modulate hypertrophic and cell signaling pathways in cardiomyocytes from diabetic mice Chronically elevated glucose compromises myocardial mitochondrial DNA integrity by alteration of mitochondrial topoisomerase function.Systematic Analysis of the Multiple Bioactivities of Green Tea through a Network Pharmacology Approach Muscle-specific overexpression of NCOATGK, splice variant of O-GlcNAcase, induces skeletal muscle atrophy.Glucose metabolism and cardiac hypertrophy Recombinant human erythropoietin protects the myocardium from ischemia-reperfusion injury and promotes beneficial remodeling.Knockout of the TauT gene predisposes C57BL/6 mice to streptozotocin-induced diabetic nephropathy.Cardioprotective effects of gallic acid in diabetes-induced myocardial dysfunction in rats Molecular Mechanisms of Retinoid Receptors in Diabetes-Induced Cardiac Remodeling.Unbreak my heart: targeting mitochondrial autophagy in diabetic cardiomyopathy Back to your heart: ubiquitin proteasome system-regulated signal transduction The diabetic heart: too sweet for its own good?Age-related loss of brain volume and T2 relaxation time in youth with type 1 diabetes.The p53 story: layers of complexity.Suppression of autophagy is protective in high glucose-induced cardiomyocyte injury.Cardiac stem cells and mechanisms of myocardial regeneration.In vitro evidence of glucose-induced toxicity in GnRH secreting neurons: high glucose concentrations influence GnRH secretion, impair cell viability, and induce apoptosis in the GT1-1 neuronal cell line.Post-translational protein modification by O-linked N-acetyl-glucosamine: its role in mediating the adverse effects of diabetes on the heart.Role of protein O-linked N-acetyl-glucosamine in mediating cell function and survival in the cardiovascular system.Obesity improves myocardial ischaemic tolerance and RISK signalling in insulin-insensitive rats.Metabolic syndrome influences cardiac gene expression pattern at the transcript level in male ZDF rats Type 1 diabetes mellitus abrogates compensatory augmentation of myocardial neuregulin-1β/ErbB in response to myocardial infarction resulting in worsening heart failure.

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P2860

Hyperglycemia activates p53 and p53-regulated genes leading to myocyte cell death.

http://www.wikidata.org/entity/Q34092744

description

2001 nî lūn-bûn

@nan

2001 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2001 թվականի հոտեմբերին հրատարակված գիտական հոդված

@hy

2001年の論文

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2001年論文

@yue

2001年論文

@zh-hant

2001年論文

@zh-hk

2001年論文

@zh-mo

2001年論文

@zh-tw

2001年论文

@wuu

name

Hyperglycemia activates p53 and p53-regulated genes leading to myocyte cell death.

@ast

Hyperglycemia activates p53 and p53-regulated genes leading to myocyte cell death.

@en

Hyperglycemia activates p53 and p53-regulated genes leading to myocyte cell death.

@nl

type

label

Hyperglycemia activates p53 and p53-regulated genes leading to myocyte cell death.

@ast

Hyperglycemia activates p53 and p53-regulated genes leading to myocyte cell death.

@en

Hyperglycemia activates p53 and p53-regulated genes leading to myocyte cell death.

@nl

prefLabel

Hyperglycemia activates p53 and p53-regulated genes leading to myocyte cell death.

@ast

Hyperglycemia activates p53 and p53-regulated genes leading to myocyte cell death.

@en

Hyperglycemia activates p53 and p53-regulated genes leading to myocyte cell death.

@nl

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