An endogenous cannabinoid (2-AG) is neuroprotective after brain injury. - Wikidata - awgldk - TriplyDB

An endogenous cannabinoid (2-AG) is neuroprotective after brain injury.

An endogenous cannabinoid (2-AG) is neuroprotective after brain injury.

http://www.wikidata.org/entity/Q34093755

about

Cannabis and tobacco smoke are not equally carcinogenic.Cloning of the first sn1-DAG lipases points to the spatial and temporal regulation of endocannabinoid signaling in the brain 2-arachidonoylglycerol, an endogenous cannabinoid receptor ligand, induces rapid actin polymerization in HL-60 cells differentiated into macrophage-like cells Identification of a gene encoding MGAT1, a monoacylglycerol acyltransferase.Brain monoglyceride lipase participating in endocannabinoid inactivation N-arachidonyl maleimide potentiates the pharmacological and biochemical effects of the endocannabinoid 2-arachidonylglycerol through inhibition of monoacylglycerol lipase Is lipid signaling through cannabinoid 2 receptors part of a protective system?The endocannabinoid system as an emerging target of pharmacotherapy Parsing the players: 2-arachidonoylglycerol synthesis and degradation in the CNS Monoacylglycerol lipase - a target for drug development?Immune mechanisms in cerebral ischemic tolerance CB2 and GPR55 Receptors as Therapeutic Targets for Systemic Immune Dysregulation Excess cerebral TNF causing glutamate excitotoxicity rationalizes treatment of neurodegenerative diseases and neurogenic pain by anti-TNF agents CB1 and CB2 Cannabinoid Receptor Antagonists Prevent Minocycline-Induced Neuroprotection Following Traumatic Brain Injury in Mice Mass spectrometry imaging of biomarker lipids for phagocytosis and signalling during focal cerebral ischaemia Tetrahydrocannabinol-induced neurotoxicity depends on CB1 receptor-mediated c-Jun N-terminal kinase activation in cultured cortical neurons Differential Expression of Brain Cannabinoid Receptors between Repeatedly Stressed Males and Females may Play a Role in Age and Gender-Related Difference in Traumatic Brain Injury: Implications from Animal Studies.Pursuing paradoxical proconvulsant prophylaxis for epileptogenesis Effects of intra-amygdala infusion of CB1 receptor agonists on the reconsolidation of fear-potentiated startle.Cannabinoid CB2 receptor (CB2R) stimulation delays rubrospinal mitochondrial-dependent degeneration and improves functional recovery after spinal cord hemisection by ERK1/2 inactivation.Cannabinoids in multiple sclerosis (CAMS) study: safety and efficacy data for 12 months follow up.Chronic morphine modulates the contents of the endocannabinoid, 2-arachidonoyl glycerol, in rat brain.Δ9-tetrahydrocannabinol prevents methamphetamine-induced neurotoxicity Anandamide potentiation of miniature spontaneous excitatory synaptic transmission is mediated via IP3 pathway.Endogenous cannabinoid signaling is required for voluntary exercise-induced enhancement of progenitor cell proliferation in the hippocampus.Neuroprotective therapies after perinatal hypoxic-ischemic brain injury.Endocannabinoid metabolism in neurodegenerative diseases CD38 facilitates recovery from traumatic brain injury Targeting the endocannabinoid system in Alzheimer's disease.A new generation fatty acid amide hydrolase inhibitor protects against kainate-induced excitotoxicity.Endocannabinoids in synaptic plasticity and neuroprotection.Quantification of brain endocannabinoid levels: methods, interpretations and pitfalls Early age-related cognitive impairment in mice lacking cannabinoid CB1 receptors.Mutations in ABHD12 cause the neurodegenerative disease PHARC: An inborn error of endocannabinoid metabolism.Cannabis and the brain.Huntington's disease and neurogenesis: FGF-2 to the rescue?Learning and memory performances in adolescent users of alcohol and marijuana: interactive effects.Multiple functions of endocannabinoid signaling in the brain.Δ9-THC-caused synaptic and memory impairments are mediated through COX-2 signaling Synaptic and cognitive improvements by inhibition of 2-AG metabolism are through upregulation of microRNA-188-3p in a mouse model of Alzheimer's disease.

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P2860

An endogenous cannabinoid (2-AG) is neuroprotective after brain injury.

http://www.wikidata.org/entity/Q34093755

description

2001 nî lūn-bûn

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2001 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2001 թվականի հոտեմբերին հրատարակված գիտական հոդված

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2001年の論文

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2001年論文

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2001年論文

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2001年論文

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2001年論文

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2001年論文

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2001年论文

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name

An endogenous cannabinoid (2-AG) is neuroprotective after brain injury.

@ast

An endogenous cannabinoid (2-AG) is neuroprotective after brain injury.

@en

An endogenous cannabinoid

@nl

type

label

An endogenous cannabinoid (2-AG) is neuroprotective after brain injury.

@ast

An endogenous cannabinoid (2-AG) is neuroprotective after brain injury.

@en

An endogenous cannabinoid

@nl

prefLabel

An endogenous cannabinoid (2-AG) is neuroprotective after brain injury.

@ast

An endogenous cannabinoid (2-AG) is neuroprotective after brain injury.

@en

An endogenous cannabinoid

@nl

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An endogenous cannabinoid (2-AG) is neuroprotective after brain injury.

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A Breuer

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C Simeonidou

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D Panikashvili

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E Shohami

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527-531

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scholarly article

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10.1038/35097089

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6855

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413

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Raphael Mechoulam

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2001-10-01T00:00:00Z

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11763987

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