Cholesterol reduction impairs exocytosis of synaptic vesicles. - Wikidata - awgldk - TriplyDB

Cholesterol reduction impairs exocytosis of synaptic vesicles.

Cholesterol reduction impairs exocytosis of synaptic vesicles.

http://www.wikidata.org/entity/Q34094964

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The dynamic architecture of photoreceptor ribbon synapses: cytoskeletal, extracellular matrix, and intramembrane proteins Miglustat Reverts the Impairment of Synaptic Plasticity in a Mouse Model of NPC Disease The role of cholesterol in membrane fusion High cholesterol obviates a prolonged hemifusion intermediate in fast SNARE-mediated membrane fusion Multiphasic effects of cholesterol on influenza fusion kinetics reflect multiple mechanistic roles.Long-term atorvastatin treatment leads to alterations in behavior, cognition, and hippocampal biochemistry.Niemann-Pick C disease and mobilization of lysosomal cholesterol by cyclodextrin Cholesterol in brain disease: sometimes determinant and frequently implicated.Lateral mobility of presynaptic L-type calcium channels at photoreceptor ribbon synapses Membrane cholesterol removal changes mechanical properties of cells and induces secretion of a specific pool of lysosomes.LTP-triggered cholesterol redistribution activates Cdc42 and drives AMPA receptor synaptic delivery Cholesterol metabolism and homeostasis in the brain Cholesterol regulates multiple forms of vesicle endocytosis at a mammalian central synapse.Regulation of presynaptic strength by controlling Ca2+ channel mobility: effects of cholesterol depletion on release at the cone ribbon synapse Energetics of stalk intermediates in membrane fusion are controlled by lipid composition.Effect of cholesterol reduction on receptor signaling in neurons.The regulated expression, intracellular trafficking, and membrane recycling of the P2Y-like receptor GPR17 in Oli-neu oligodendroglial cells.Morphological Differentiation Towards Neuronal Phenotype of SH-SY5Y Neuroblastoma Cells by Estradiol, Retinoic Acid and Cholesterol.Cholesterol Increases the Openness of SNARE-Mediated Flickering Fusion Pores The lipid composition and physical properties of the yeast vacuole affect the hemifusion-fusion transition.Corpus callosum measurements correlate with developmental delay in Smith-Lemli-Opitz syndrome.Oxysterol-binding protein (OSBP) is required for the perinuclear localization of intra-Golgi v-SNAREs.The E1015K variant in the synprint region of the CaV2.1 channel alters channel function and is associated with different migraine phenotypes.SNAP23-Dependent Surface Translocation of Leukotriene B4 (LTB4) Receptor 1 Is Essential for NOX2-Mediated Exocytotic Degranulation in Human Mast Cells Induced by Trichomonas vaginalis-Secreted LTB4.Splice isoform and pharmacological studies reveal that sterol depletion relocalizes α-synuclein and enhances its toxicity Lipid rafts: a signaling platform linking cholesterol metabolism to synaptic deficits in autism spectrum disorders.Lipid Processing in the Brain: A Key Regulator of Systemic Metabolism.The relationship between statins and depression: a review of the literature.Neuropsychiatric adverse events associated with statins: epidemiology, pathophysiology, prevention and management.Lipid metabolism in Alzheimer's disease Role of endolysosomes and cholesterol in the pathogenesis of Alzheimer's disease: Insights into why statins might not provide clinical benefit.The Deleterious Effects of Oxidative and Nitrosative Stress on Palmitoylation, Membrane Lipid Rafts and Lipid-Based Cellular Signalling: New Drug Targets in Neuroimmune Disorders.Cholesterol regulates polymodal sensory transduction in Müller glia.Cellular Cholesterol Facilitates the Postentry Replication Cycle of Herpes Simplex Virus 1.Lovastatin Differentially Affects Neuronal Cholesterol and Amyloid-β Production in vivo and in vitro.Cholesterol and F-actin are required for clustering of recycling synaptic vesicle proteins in the presynaptic plasma membrane.Inhibition of neuronal cholesterol biosynthesis with lovastatin leads to impaired synaptic vesicle release even in the presence of lipoproteins or geranylgeraniol.SNX27, a protein involved in down syndrome, regulates GPR17 trafficking and oligodendrocyte differentiation.Hypothesis: the role of sterols in autism spectrum disorder.Cholesterol-loaded nanoparticles ameliorate synaptic and cognitive function in Huntington's disease mice.

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P2860

Cholesterol reduction impairs exocytosis of synaptic vesicles.

http://www.wikidata.org/entity/Q34094964

description

2010 nî lūn-bûn

@nan

2010 թուականի Յունուարին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի հունվարին հրատարակված գիտական հոդված

@hy

2010年の論文

@ja

2010年論文

@yue

2010年論文

@zh-hant

2010年論文

@zh-hk

2010年論文

@zh-mo

2010年論文

@zh-tw

2010年论文

@wuu

name

Cholesterol reduction impairs exocytosis of synaptic vesicles.

@ast

Cholesterol reduction impairs exocytosis of synaptic vesicles.

@en

Cholesterol reduction impairs exocytosis of synaptic vesicles.

@nl

type

label

Cholesterol reduction impairs exocytosis of synaptic vesicles.

@ast

Cholesterol reduction impairs exocytosis of synaptic vesicles.

@en

Cholesterol reduction impairs exocytosis of synaptic vesicles.

@nl

prefLabel

Cholesterol reduction impairs exocytosis of synaptic vesicles.

@ast

Cholesterol reduction impairs exocytosis of synaptic vesicles.

@en

Cholesterol reduction impairs exocytosis of synaptic vesicles.

@nl

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Journal of Cell Science

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Cholesterol reduction impairs exocytosis of synaptic vesicles.

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P2093

Alessandra Fratangeli

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Anna Linetti

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Elena Taverna

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Maria Passafaro

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Pamela Valnegri

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Patrizia Rosa

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595-605

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scholarly article

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10.1242/JCS.060681

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P433

Pt 4

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123

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Valentina Cappello

Michela Matteoli

Maura Francolini

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2010-01-26T00:00:00Z

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41148530

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20103534

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