Type I IFN promotes IL-10 production from T cells to suppress Th17 cells and Th17-associated autoimmune inflammation - Wikidata - awgldk - TriplyDB

Type I IFN promotes IL-10 production from T cells to suppress Th17 cells and Th17-associated autoimmune inflammation

Type I IFN promotes IL-10 production from T cells to suppress Th17 cells and Th17-associated autoimmune inflammation

http://www.wikidata.org/entity/Q34097852

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Th17 plasticity and its changes associated with inflammatory bowel disease CD4 T-cell subsets in malaria: TH1/TH2 revisited Lithium controls central nervous system autoimmunity through modulation of IFN-γ signaling Unraveling the convoluted biological roles of type I interferons in infection and immunity: a way forward for therapeutics and vaccine design.Th1-Th17 cells contribute to the development of uropathogenic Escherichia coli-induced chronic pelvic pain Induction of endogenous Type I interferon within the central nervous system plays a protective role in experimental autoimmune encephalomyelitis.Local IL-17 Production Exerts a Protective Role in Murine Experimental Glomerulonephritis.Infected CD8α- dendritic cells are the predominant source of IL-10 during establishment of persistent viral infection.Protection against Th17 cells differentiation by an interleukin-23 receptor cytokine-binding homology region.What do effective treatments for multiple sclerosis tell us about the molecular mechanisms involved in pathogenesis?IFNAR signaling directly modulates T lymphocyte activity, resulting in milder experimental autoimmune encephalomyelitis development.MicroRNA let-7e is associated with the pathogenesis of experimental autoimmune encephalomyelitis Enhanced immunoregulation of mesenchymal stem cells by IL-10-producing type 1 regulatory T cells in collagen-induced arthritis.Type I Interferon Counters or Promotes Coxiella burnetii Replication Dependent on Tissue.Role of toll-like receptors in multiple sclerosis.Interferon-dependent IL-10 production by Tregs limits tumor Th17 inflammation Independent and interdependent immunoregulatory effects of IL-27, IFN-β, and IL-10 in the suppression of human Th17 cells and murine experimental autoimmune encephalomyelitis.Oral delivery of oligomeric procyanidins in Apple Poly® enhances type I IFN responses in vivo.Protective immunity and vaccination against cutaneous leishmaniasis.IL-10-producing lymphocytes in inflammatory disease.Multipronged CD4(+) T-cell effector and memory responses cooperate to provide potent immunity against respiratory virus.Biomarker studies in multiple sclerosis: from proteins to noncoding RNAs.Direct immunomodulatory influence of IFN-β on human astrocytoma cells.Cannabinoids decrease the th17 inflammatory autoimmune phenotype.IL-10: A Multifunctional Cytokine in Viral Infections.Cellular and Molecular Dynamics of Th17 Differentiation and its Developmental Plasticity in the Intestinal Immune Response West Nile virus-infected human dendritic cells fail to fully activate invariant natural killer T cells.Dual Inhibition of TNFR1 and IFNAR1 in Imiquimod-Induced Psoriasiform Skin Inflammation in Mice.Plasmacytoid dendritic cell-derived IFNα modulates Th17 differentiation during early Bordetella pertussis infection in mice.B cells modulate systemic responses to Pneumocystis murina lung infection and protect on-demand hematopoiesis via T cell-independent innate mechanisms when type I interferon signaling is absent.Inflammasome activation has an important role in the development of spontaneous colitis.IL-10 Modulates Th17 Pathogenicity during Autoimmune Diseases.Interleukin 17A as a good predictor of the severity of Mycoplasma pneumoniae pneumonia in children.T-helper and regulatory T-cell cytokines in the peripheral blood of patients with active alopecia areata.Cytokine Signaling in Multiple Sclerosis and Its Therapeutic Applications.Inverse correlation of expression of microRNA-140-5p with progression of multiple sclerosis and differentiation of encephalitogenic T helper type 1 cells.Fasudil mediates cell therapy of EAE by immunomodulating encephalomyelitic T cells and macrophages.Restoration of the type I IFN-IL-1 balance through targeted blockade of PTGER4 inhibits autoimmunity in NOD mice.Insight Into Non-Pathogenic Th17 Cells in Autoimmune Diseases.Nod2 Deficiency Augments Th17 Responses and Exacerbates Autoimmune Arthritis

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Type I IFN promotes IL-10 production from T cells to suppress Th17 cells and Th17-associated autoimmune inflammation

http://www.wikidata.org/entity/Q34097852

description

2011 nî lūn-bûn

@nan

2011 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2011 թվականի դեկտեմբերին հրատարակված գիտական հոդված

@hy

2011年の論文

@ja

2011年学术文章

@wuu

2011年学术文章

@zh-cn

2011年学术文章

@zh-hans

2011年学术文章

@zh-my

2011年学术文章

@zh-sg

2011年學術文章

@yue

name

Type I IFN promotes IL-10 prod ...... ciated autoimmune inflammation

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Type I IFN promotes IL-10 prod ...... ciated autoimmune inflammation

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Type I IFN promotes IL-10 prod ...... ciated autoimmune inflammation

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type

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Type I IFN promotes IL-10 prod ...... ciated autoimmune inflammation

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Type I IFN promotes IL-10 prod ...... ciated autoimmune inflammation

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Type I IFN promotes IL-10 prod ...... ciated autoimmune inflammation

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prefLabel

Type I IFN promotes IL-10 prod ...... ciated autoimmune inflammation

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Type I IFN promotes IL-10 prod ...... ciated autoimmune inflammation

@en

Type I IFN promotes IL-10 prod ...... ciated autoimmune inflammation

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Type I IFN promotes IL-10 prod ...... ciated autoimmune inflammation

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Beichu Guo

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Lixia Zhang

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Shunzong Yuan

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P2860

TGF-beta-induced Foxp3 inhibits T(H)17 cell differentiation by antagonizing RORgammat function

The biological functions of T helper 17 cell effector cytokines in inflammation

Global mapping of H3K4me3 and H3K27me3 reveals specificity and plasticity in lineage fate determination of differentiating CD4+ T cells

Functional and pathogenic differences of Th1 and Th17 cells in experimental autoimmune encephalomyelitis

Th17 and regulatory T cells in mediating and restraining inflammation.

Reciprocal developmental pathways for the generation of pathogenic effector TH17 and regulatory T cells

The orphan nuclear receptor RORgammat directs the differentiation program of proinflammatory IL-17+ T helper cells

Interferons at age 50: past, current and future impact on biomedicine

A brief history of T(H)17, the first major revision in the T(H)1/T(H)2 hypothesis of T cell-mediated tissue damage

TGFbeta in the context of an inflammatory cytokine milieu supports de novo differentiation of IL-17-producing T cells

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