Increased store-operated Ca2+ entry in skeletal muscle with reduced calsequestrin-1 expression.
about
The human protein disulfide isomerase gene familyStore-operated Ca2+ entry in muscle physiology and diseasesCalsequestrin (CASQ1) rescues function and structure of calcium release units in skeletal muscles of CASQ1-null miceAAV-mediated knock-down of HRC exacerbates transverse aorta constriction-induced heart failureMice null for calsequestrin 1 exhibit deficits in functional performance and sarcoplasmic reticulum calcium handling.Malignant hyperthermia susceptibility arising from altered resting coupling between the skeletal muscle L-type Ca2+ channel and the type 1 ryanodine receptorMassive alterations of sarcoplasmic reticulum free calcium in skeletal muscle fibers lacking calsequestrin revealed by a genetically encoded probe.The Sick and the Weak: Neuropathies/Myopathies in the Critically Ill.Orai1 mediates exacerbated Ca(2+) entry in dystrophic skeletal muscleMitochondrial Ca2+-handling in fast skeletal muscle fibers from wild type and calsequestrin-null miceAccelerated activation of SOCE current in myotubes from two mouse models of anesthetic- and heat-induced sudden death.Special article: Future directions in malignant hyperthermia research and patient careOxidative stress, mitochondrial damage, and cores in muscle from calsequestrin-1 knockout miceRetrograde regulation of STIM1-Orai1 interaction and store-operated Ca2+ entry by calsequestrin.Temperature and RyR1 regulate the activation rate of store-operated Ca²+ entry current in myotubesDantrolene-induced inhibition of skeletal L-type Ca2+ current requires RyR1 expression.The disorders of the calcium release unit of skeletal muscles: what have we learned from mouse models?Store-operated calcium entry is present in HL-1 cardiomyocytes and contributes to resting calcium.STIM1-Ca(2+) signaling is required for the hypertrophic growth of skeletal muscle in mice.A focus on extracellular Ca2+ entry into skeletal muscle.An association study of CASQ1 gene polymorphisms and heat stroke.Targeted deletion of Kcne3 impairs skeletal muscle function in mice.Strenuous exercise triggers a life-threatening response in mice susceptible to malignant hyperthermia.With the greatest care, stromal interaction molecule (STIM) proteins verify what skeletal muscle is doing
P2860
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P2860
Increased store-operated Ca2+ entry in skeletal muscle with reduced calsequestrin-1 expression.
description
2010 nî lūn-bûn
@nan
2010 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Increased store-operated Ca2+ ...... ed calsequestrin-1 expression.
@ast
Increased store-operated Ca2+ ...... ed calsequestrin-1 expression.
@en
Increased store-operated Ca2+ ...... ed calsequestrin-1 expression.
@nl
type
label
Increased store-operated Ca2+ ...... ed calsequestrin-1 expression.
@ast
Increased store-operated Ca2+ ...... ed calsequestrin-1 expression.
@en
Increased store-operated Ca2+ ...... ed calsequestrin-1 expression.
@nl
prefLabel
Increased store-operated Ca2+ ...... ed calsequestrin-1 expression.
@ast
Increased store-operated Ca2+ ...... ed calsequestrin-1 expression.
@en
Increased store-operated Ca2+ ...... ed calsequestrin-1 expression.
@nl
P2093
P2860
P1433
P1476
Increased store-operated Ca2+ ...... ed calsequestrin-1 expression.
@en
P2093
Choon Kee Min
Do Han Kim
Jae-Kyun Ko
Jerome Parness
Jianjie Ma
Noah Weisleder
Xiaoli Zhao
P2860
P304
P356
10.1016/J.BPJ.2010.06.050
P407
P577
2010-09-01T00:00:00Z