Driver mutations in TP53 are ubiquitous in high grade serous carcinoma of the ovary. - Wikidata - awgldk - TriplyDB

Driver mutations in TP53 are ubiquitous in high grade serous carcinoma of the ovary.

Driver mutations in TP53 are ubiquitous in high grade serous carcinoma of the ovary.

http://www.wikidata.org/entity/Q34103978

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ARID1A mutations in endometriosis-associated ovarian carcinomas Genetic and epigenetic heterogeneity of epithelial ovarian cancer and the clinical implications for molecular targeted therapy Oncolytic Adenovirus: Strategies and Insights for Vector Design and Immuno-Oncolytic Applications Rethinking ovarian cancer II: reducing mortality from high-grade serous ovarian cancer Prognostic Role of Serum Antibody Immunity to p53 Oncogenic Protein in Ovarian Cancer: A Systematic Review and a Meta-Analysis Clinical investigation of receptor and non-receptor tyrosine kinase inhibitors for the treatment of epithelial ovarian cancer Mutant p53 regulates ovarian cancer transformed phenotypes through autocrine matrix deposition.Integrated genomic analyses of ovarian carcinoma Prioritizing therapeutic targets using patient-derived xenograft models Methylation of cell-free circulating DNA in the diagnosis of cancer Type-specific cell line models for type-specific ovarian cancer research Evolution of pre-existing versus acquired resistance to platinum drugs and PARP inhibitors in BRCA-associated cancers Ovarian cancer cell line panel (OCCP): clinical importance of in vitro morphological subtypes Spatial and temporal heterogeneity in high-grade serous ovarian cancer: a phylogenetic analysis High throughput interrogation of somatic mutations in high grade serous cancer of the ovary Detection of somatic TP53 mutations in tampons of patients with high-grade serous ovarian cancer.Predicting survival in head and neck squamous cell carcinoma from TP53 mutation.MuSiC: identifying mutational significance in cancer genomes.Molecular requirements for transformation of fallopian tube epithelial cells into serous carcinoma.TITAN: inference of copy number architectures in clonal cell populations from tumor whole-genome sequence data.Differential role of PKC-induced c-Jun in HTLV-1 LTR activation by 12-O-tetradecanoylphorbol-13-acetate in different human T-cell lines.The ovary is an alternative site of origin for high-grade serous ovarian cancer in mice.Epigenetic control of autophagy by microRNAs in ovarian cancer Bioenergetic analysis of ovarian cancer cell lines: profiling of histological subtypes and identification of a mitochondria-defective cell line.Functional characterisation of a novel ovarian cancer cell line, NUOC-1.Poor survival with wild-type TP53 ovarian cancer?Analysis and comparison of somatic mutations in paired primary and recurrent epithelial ovarian cancer samples.Safety and utility of image-guided research biopsies in relapsed high-grade serous ovarian carcinoma-experience of the BriTROC consortium.Whole exome sequence analysis of serous borderline tumors of the ovary.Two different, mutually exclusively distributed, TP53 mutations in ovarian and peritoneal tumor tissues of a serous ovarian cancer patient: indicative for tumor origin?CD44 and p53 immunoexpression patterns in NF1 neoplasms - indicators of malignancy and infiltration.Phosphoproteomic analysis of gossypol-induced apoptosis in ovarian cancer cell line, HOC1a.Genomic analysis of genetic heterogeneity and evolution in high-grade serous ovarian carcinoma.TP53 mutations in serous tubal intraepithelial carcinoma and concurrent pelvic high-grade serous carcinoma--evidence supporting the clonal relationship of the two lesions.MiR-182 overexpression in tumourigenesis of high-grade serous ovarian carcinoma.Discovery of prognostic factors for diagnosis and treatment of epithelial-derived ovarian cancer from laying hens.VGLL3 expression is associated with a tumor suppressor phenotype in epithelial ovarian cancer Ovarian surface epithelium at the junction area contains a cancer-prone stem cell niche.Loss of BRCA1-A complex function in RAP80 null tumor cells Profiles of genomic instability in high-grade serous ovarian cancer predict treatment outcome

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P2860

Driver mutations in TP53 are ubiquitous in high grade serous carcinoma of the ovary.

http://www.wikidata.org/entity/Q34103978

description

2010 nî lūn-bûn

@nan

2010 թուականի Մայիսին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի մայիսին հրատարակված գիտական հոդված

@hy

2010年の論文

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2010年論文

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2010年論文

@zh-hant

2010年論文

@zh-hk

2010年論文

@zh-mo

2010年論文

@zh-tw

2010年论文

@wuu

name

Driver mutations in TP53 are ubiquitous in high grade serous carcinoma of the ovary.

@ast

Driver mutations in TP53 are ubiquitous in high grade serous carcinoma of the ovary.

@en

Driver mutations in TP53 are ubiquitous in high grade serous carcinoma of the ovary.

@nl

type

label

Driver mutations in TP53 are ubiquitous in high grade serous carcinoma of the ovary.

@ast

Driver mutations in TP53 are ubiquitous in high grade serous carcinoma of the ovary.

@en

Driver mutations in TP53 are ubiquitous in high grade serous carcinoma of the ovary.

@nl

prefLabel

Driver mutations in TP53 are ubiquitous in high grade serous carcinoma of the ovary.

@ast

Driver mutations in TP53 are ubiquitous in high grade serous carcinoma of the ovary.

@en

Driver mutations in TP53 are ubiquitous in high grade serous carcinoma of the ovary.

@nl

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P1433

The Journal of Pathology

P1476

Driver mutations in TP53 are ubiquitous in high grade serous carcinoma of the ovary.

@en

P2093

Ahmed Ashour Ahmed

http://www.w3.org/2001/XMLSchema#string

Colin Stewart

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Dariush Etemadmoghadam

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Jillian Temple

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Mohamed Riad

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Raghwa Sharma

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Sian Fereday

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P2860

Ovarian carcinoma subtypes are different diseases: implications for biomarker studies

Digital karyotyping

An integrated genomic analysis of human glioblastoma multiforme

The clinical value of somatic TP53 gene mutations in 1,794 patients with breast cancer.

Primer3 on the WWW for general users and for biologist programmers

Rescue of embryonic lethality in Mdm4-null mice by loss of Trp53 suggests a nonoverlapping pathway with MDM2 to regulate p53

The ubiquitin ligase COP1 is a critical negative regulator of p53

Amplification of a gene encoding a p53-associated protein in human sarcomas

Pirh2, a p53-induced ubiquitin-protein ligase, promotes p53 degradation

Mice deficient for p53 are developmentally normal but susceptible to spontaneous tumours

P304

49-56

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scholarly article

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10.1002/PATH.2696

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1

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P478

221

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James D Brenton

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2010-05-01T00:00:00Z

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41945024

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20229506

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3262968

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