Galactose enhances oxidative metabolism and reveals mitochondrial dysfunction in human primary muscle cells - Wikidata - awgldk - TriplyDB

Galactose enhances oxidative metabolism and reveals mitochondrial dysfunction in human primary muscle cells

Galactose enhances oxidative metabolism and reveals mitochondrial dysfunction in human primary muscle cells

http://www.wikidata.org/entity/Q34110088

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A small molecule with anticancer and antimetastatic activities induces rapid mitochondrial-associated necrosis in breast cancer.OPA1-dependent cristae modulation is essential for cellular adaptation to metabolic demand Glucose controls morphodynamics of LPS-stimulated macrophages.Galactose and its Metabolites Deteriorate Metaphase II Mouse Oocyte Quality and Subsequent Embryo Development by Disrupting the Spindle Structure.Impaired mitochondrial function in human placenta with increased maternal adiposity Membrane-bound CYB5R3 is a common effector of nutritional and oxidative stress response through FOXO3a and Nrf2.Remodeling of oxidative energy metabolism by galactose improves glucose handling and metabolic switching in human skeletal muscle cells.Acylcarnitines: potential implications for skeletal muscle insulin resistance.Low glucose but not galactose enhances oxidative mitochondrial metabolism in C2C12 myoblasts and myotubes.A metabolomic study of the PPARδ agonist GW501516 for enhancing running endurance in Kunming mice.Meclizine Preconditioning Protects the Kidney Against Ischemia-Reperfusion Injury.The Effect of a Novel c.820C>T (Arg274Trp) Mutation in the Mitofusin 2 Gene on Fibroblast Metabolism and Clinical Manifestation in a Patient.Molecular hydrogen protects against oxidative stress-induced SH-SY5Y neuroblastoma cell death through the process of mitohormesis.Effect of serial cell passaging in the retention of fiber type and mitochondrial content in primary human myotubes Oxidative stress contributes to outcome severity in a Drosophila melanogaster model of classic galactosemia.T-cell development of resistance to apoptosis is driven by a metabolic shift in carbon source and altered activation of death pathways Akt3 knockdown induces mitochondrial dysfunction in human cancer cells.Glucocorticoid receptor-PPARα axis in fetal mouse liver prepares neonates for milk lipid catabolism MiR-448 promotes glycolytic metabolism of gastric cancer by downregulating KDM2B Mitochondrial respiratory capacity and content are normal in young insulin-resistant obese humans.Expansion of mesenchymal stem cells under atmospheric carbon dioxide.Modulation of mitochondrial bioenergetics in a skeletal muscle cell line model of mitochondrial toxicity.Glycolysis and oxidative phosphorylation are essential for purinergic receptor-mediated angiogenic responses in vasa vasorum endothelial cells.Are cultured human myotubes far from home?Bromodomain Inhibitors Correct Bioenergetic Deficiency Caused by Mitochondrial Disease Complex I Mutations A real-time fluorometric method for the simultaneous detection of cell death type and rate.The diabetes medication Canagliflozin reduces cancer cell proliferation by inhibiting mitochondrial complex-I supported respiration.An optimized pyrimidinol multifunctional radical quencher.Hypoxia Inducible Factors Modulate Mitochondrial Oxygen Consumption and Transcriptional Regulation of Nuclear-Encoded Electron Transport Chain Genes.Distinct carbon sources affect structural and functional maturation of cardiomyocytes derived from human pluripotent stem cells.Mitochondrial respiration without ubiquinone biosynthesis.Glucose Metabolism and Oxygen Availability Govern Reactivation of the Latent Human Retrovirus HTLV-1.Normalizing glutamine concentration causes mitochondrial uncoupling in an in vitro model of human skeletal muscle.Lipidomics reveals diurnal lipid oscillations in human skeletal muscle persisting in cellular myotubes cultured in vitro.Mitochondrial ATP Depletion Disrupts Caco-2 Monolayer Integrity and Internalizes Claudin 7 MiR-361-5p inhibits glycolytic metabolism, proliferation and invasion of breast cancer by targeting FGFR1 and MMP-1.Interplay between metabolic identities in the intestinal crypt supports stem cell function.Mitochondrial CoQ deficiency is a common driver of mitochondrial oxidants and insulin resistance.Inhibition of Drp1/Fis1 interaction slows progression of amyotrophic lateral sclerosis.Systematic Gene-to-Phenotype Arrays: A High-Throughput Technique for Molecular Phenotyping.

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Galactose enhances oxidative metabolism and reveals mitochondrial dysfunction in human primary muscle cells

http://www.wikidata.org/entity/Q34110088

description

2011 nî lūn-bûn

@nan

2011 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2011 թվականի դեկտեմբերին հրատարակված գիտական հոդված

@hy

2011年の論文

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2011年論文

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2011年論文

@zh-hant

2011年論文

@zh-hk

2011年論文

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2011年論文

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2011年论文

@wuu

name

Galactose enhances oxidative m ...... in human primary muscle cells

@ast

Galactose enhances oxidative m ...... in human primary muscle cells

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Galactose enhances oxidative m ...... in human primary muscle cells

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type

label

Galactose enhances oxidative m ...... in human primary muscle cells

@ast

Galactose enhances oxidative m ...... in human primary muscle cells

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Galactose enhances oxidative m ...... in human primary muscle cells

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prefLabel

Galactose enhances oxidative m ...... in human primary muscle cells

@ast

Galactose enhances oxidative m ...... in human primary muscle cells

@en

Galactose enhances oxidative m ...... in human primary muscle cells

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Galactose enhances oxidative m ...... in human primary muscle cells

@en

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Cynthia Moffat

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Céline Aguer

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Daniela Gambarotta

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Robert Dent

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Ruth McPherson

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P2860

High aerobic glycolysis of rat hepatoma cells in culture: role of mitochondrial hexokinase

Developmental and tissue-specific modulation of rat galactose-1-phosphate uridyltransferase steady state messenger RNA and specific activity levels

Circumventing the Crabtree Effect: Replacing Media Glucose with Galactose Increases Susceptibility of HepG2 Cells to Mitochondrial Toxicants

Impaired mitochondrial activity in the insulin-resistant offspring of patients with type 2 diabetes

Dysfunction of mitochondria in human skeletal muscle in type 2 diabetes

AMP-activated protein kinase (AMPK) action in skeletal muscle via direct phosphorylation of PGC-1alpha

Dynamic changes in fat oxidation in human primary myocytes mirror metabolic characteristics of the donor.

Characterization of the role of AMP-activated protein kinase in the regulation of glucose-activated gene expression using constitutively active and dominant negative forms of the kinase.

Evidence that glutamine, not sugar, is the major energy source for cultured HeLa cells.

Energy substrate modulates mitochondrial structure and oxidative capacity in cancer cells.

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scholarly article

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10.1371/JOURNAL.PONE.0028536

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12

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6

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Mary-Ellen Harper

Ryan J Mailloux

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2011-12-15T00:00:00Z

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51920960

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22194845

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3240634

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