Shifting FcγRIIA-ITAM from activation to inhibitory configuration ameliorates arthritis - Wikidata - awgldk - TriplyDB

Shifting FcγRIIA-ITAM from activation to inhibitory configuration ameliorates arthritis

Shifting FcγRIIA-ITAM from activation to inhibitory configuration ameliorates arthritis

http://www.wikidata.org/entity/Q34117596

about

Control of cytokine production by human fc gamma receptors: implications for pathogen defense and autoimmunity CD32a antibodies induce thrombocytopenia and type II hypersensitivity reactions in FCGR2A mice.Of ITIMs, ITAMs, and ITAMis: revisiting immunoglobulin Fc receptor signaling.The era of the immunoglobulin A Fc receptor FcαRI; its function and potential as target in disease.Intravenous Immunoglobulin (IVIG) Attenuates TNF-Induced Pathologic Bone Resorption and Suppresses Osteoclastogenesis by Inducing A20 Expression.Enhancing the safety of antibody-based immunomodulatory cancer therapy without compromising therapeutic benefit: Can we have our cake and eat it too?Surveillance of cell and tissue perturbation by receptors in the LRC.Protective role of mouse IgG1 in cryoglobulinaemia; insights from an animal model and relevance to human pathology.Mouse and human FcR effector functions.The many faces of FcγRI: implications for therapeutic antibody function.Lyn and Fyn function as molecular switches that control immunoreceptors to direct homeostasis or inflammation Protein tyrosine phosphatase SHP-1: resurgence as new drug target for human autoimmune disorders.Arthritis models: usefulness and interpretation.Immunoglobulin Glycosylation Effects in Allergy and Immunity.Cross Talk between Inhibitory Immunoreceptor Tyrosine-Based Activation Motif-Signaling and Toll-Like Receptor Pathways in Macrophages and Dendritic Cells.Shp1 function in myeloid cells.Cross-reactive antibodies enhance live attenuated virus infection for increased immunogenicity.Spontaneous germinal centers and autoimmunity.Fc Gamma Receptor IIA (CD32A) R131 Polymorphism as a Marker of Genetic Susceptibility to Sepsis.Leishmania Uses Mincle to Target an Inhibitory ITAM Signaling Pathway in Dendritic Cells that Dampens Adaptive Immunity to Infection FcεRI γ-Chain Negatively Modulates Dectin-1 Responses in Dendritic Cells.B Cell-Extrinsic Myd88 and Fcer1g Negatively Regulate Autoreactive and Normal B Cell Immune Responses.Dendritic Cell-Specific Transmembrane Protein (DC-STAMP) Regulates Osteoclast Differentiation via the Ca2+ /NFATc1 Axis.Flexible Signaling of Myeloid C-Type Lectin Receptors in Immunity and Inflammation.Immunoglobulin A as an anti-inflammatory agent

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P2860

Shifting FcγRIIA-ITAM from activation to inhibitory configuration ameliorates arthritis

http://www.wikidata.org/entity/Q34117596

description

2014 nî lūn-bûn

@nan

2014 թուականի Յուլիսին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի հուլիսին հրատարակված գիտական հոդված

@hy

2014年の論文

@ja

2014年論文

@yue

2014年論文

@zh-hant

2014年論文

@zh-hk

2014年論文

@zh-mo

2014年論文

@zh-tw

2014年论文

@wuu

name

Shifting FcγRIIA-ITAM from activation to inhibitory configuration ameliorates arthritis

@ast

Shifting FcγRIIA-ITAM from activation to inhibitory configuration ameliorates arthritis

@en

Shifting FcγRIIA-ITAM from activation to inhibitory configuration ameliorates arthritis

@nl

type

label

Shifting FcγRIIA-ITAM from activation to inhibitory configuration ameliorates arthritis

@ast

Shifting FcγRIIA-ITAM from activation to inhibitory configuration ameliorates arthritis

@en

Shifting FcγRIIA-ITAM from activation to inhibitory configuration ameliorates arthritis

@nl

prefLabel

Shifting FcγRIIA-ITAM from activation to inhibitory configuration ameliorates arthritis

@ast

Shifting FcγRIIA-ITAM from activation to inhibitory configuration ameliorates arthritis

@en

Shifting FcγRIIA-ITAM from activation to inhibitory configuration ameliorates arthritis

@nl

P1433

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Journal of Clinical Investigation

P1476

Shifting FcγRIIA-ITAM from activation to inhibitory configuration ameliorates arthritis

@en

P2093

Elisabetta Rossato

http://www.w3.org/2001/XMLSchema#string

Erwan Boedec

http://www.w3.org/2001/XMLSchema#string

Friederike Jönsson

http://www.w3.org/2001/XMLSchema#string

Gilles Hayem

http://www.w3.org/2001/XMLSchema#string

Jamel El Benna

http://www.w3.org/2001/XMLSchema#string

Jean-Michel Goujon

http://www.w3.org/2001/XMLSchema#string

Pierre Launay

http://www.w3.org/2001/XMLSchema#string

Tarek Boussetta

http://www.w3.org/2001/XMLSchema#string

P2860

Platelets amplify inflammation in arthritis via collagen-dependent microparticle production

Structural Basis for Fc RIIa Recognition of Human IgG and Formation of Inflammatory Signaling Complexes

IgA Fc receptors

Cytokines in the pathogenesis of rheumatoid arthritis

Downstream signaling molecules bind to different phosphorylated immunoreceptor tyrosine-based activation motif (ITAM) peptides of the high affinity IgE receptor

P-Rex1 and Vav1 cooperate in the regulation of formyl-methionyl-leucyl-phenylalanine-dependent neutrophil responses

Role of substrates and products of PI 3-kinase in regulating activation of Rac-related guanosine triphosphatases by Vav

Neutrophils in a mouse model of autoantibody-mediated arthritis: critical producers of Fc receptor gamma, the receptor for C5a, and lymphocyte function-associated antigen 1

Human neutrophil Fcgamma receptors initiate and play specialized nonredundant roles in antibody-mediated inflammatory diseases

Identification of FcalphaRI as an inhibitory receptor that controls inflammation: dual role of FcRgamma ITAM.

P304

3945-3959

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scholarly article

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10.1172/JCI74572

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P407

P433

9

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P478

124

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P50

Renato Monteiro

Sanae Ben Mkaddem

P577

2014-07-25T00:00:00Z

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264247541

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25061875

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4151227

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