p38 MAPK inhibition suppresses the TLR-hypersensitive phenotype in FANCC- and FANCA-deficient mononuclear phagocytes - Wikidata - awgldk - TriplyDB

p38 MAPK inhibition suppresses the TLR-hypersensitive phenotype in FANCC- and FANCA-deficient mononuclear phagocytes

p38 MAPK inhibition suppresses the TLR-hypersensitive phenotype in FANCC- and FANCA-deficient mononuclear phagocytes

http://www.wikidata.org/entity/Q34123342

about

Regulation of macrophage biology by lithium: a new look at an old drug Increased red cell distribution width in Fanconi anemia: a novel marker of stress erythropoiesis.Toll-like receptor alterations in myelodysplastic syndrome Modelling Fanconi anemia pathogenesis and therapeutics using integration-free patient-derived iPSCs FANCA and FANCC modulate TLR and p38 MAPK-dependent expression of IL-1β in macrophages.Gene expression profiles in engineered cardiac tissues respond to mechanical loading and inhibition of tyrosine kinases.Endogenous DNA Damage Leads to p53-Independent Deficits in Replicative Fitness in Fetal Murine Fancd2(-/-) Hematopoietic Stem and Progenitor Cells.Cytokine production by bone marrow mononuclear cells in inherited bone marrow failure syndromes Toll-like receptor signaling in hematopoietic homeostasis and the pathogenesis of hematologic diseases.Understanding chronic neutropenia: life is short.Elevated levels of STAT1 in Fanconi anemia group A lymphoblasts correlate with the cells' sensitivity to DNA interstrand crosslinking drugs.How I manage patients with Fanconi anaemia.PlGF enhances TLR-dependent inflammatory responses in human mononuclear phagocytes.Identification of Interleukin-1 by Functional Screening as a Key Mediator of Cellular Expansion and Disease Progression in Acute Myeloid Leukemia.Fanconi Anemia Mesenchymal Stromal Cells-Derived Glycerophospholipids Skew Hematopoietic Stem Cell Differentiation Through Toll-Like Receptor Signaling.Immunological profile of Fanconi anemia: a multicentric retrospective analysis of 61 patients.Recent advances in understanding hematopoiesis in Fanconi Anemia.Cell-Cycle-Specific Function of p53 in Fanconi Anemia Hematopoietic Stem and Progenitor Cell Proliferation.Inhibition of Metalloproteinase Activity in FANCA Is Linked to Altered Oxygen Metabolism

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p38 MAPK inhibition suppresses the TLR-hypersensitive phenotype in FANCC- and FANCA-deficient mononuclear phagocytes

http://www.wikidata.org/entity/Q34123342

description

2012 nî lūn-bûn

@nan

2012 թուականի Յունուարին հրատարակուած գիտական յօդուած

@hyw

2012 թվականի հունվարին հրատարակված գիտական հոդված

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2012年の論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年论文

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name

p38 MAPK inhibition suppresses ...... ficient mononuclear phagocytes

@ast

p38 MAPK inhibition suppresses ...... ficient mononuclear phagocytes

@en

p38 MAPK inhibition suppresses ...... ficient mononuclear phagocytes

@nl

type

label

p38 MAPK inhibition suppresses ...... ficient mononuclear phagocytes

@ast

p38 MAPK inhibition suppresses ...... ficient mononuclear phagocytes

@en

p38 MAPK inhibition suppresses ...... ficient mononuclear phagocytes

@nl

prefLabel

p38 MAPK inhibition suppresses ...... ficient mononuclear phagocytes

@ast

p38 MAPK inhibition suppresses ...... ficient mononuclear phagocytes

@en

p38 MAPK inhibition suppresses ...... ficient mononuclear phagocytes

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p38 MAPK inhibition suppresses ...... ficient mononuclear phagocytes

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Carlo Dufour

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Grover C Bagby

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Jane Yates

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Keaney Rathbun

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Laura E Hays

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Michael R Garbati

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Praveen Anur

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Winifred Keeble

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P2860

Mammalian thioredoxin is a direct inhibitor of apoptosis signal-regulating kinase (ASK) 1.

FANCC interacts with Hsp70 to protect hematopoietic cells from IFN-gamma/TNF-alpha-mediated cytotoxicity.

Mutations of the SLX4 gene in Fanconi anemia

The anti-apoptotic function of Hsp70 in the interferon-inducible double-stranded RNA-dependent protein kinase-mediated death signaling pathway requires the Fanconi anemia protein, FANCC

The Fanconi anemia complementation group C gene product: structural evidence of multifunctionality

A quantitative analysis of kinase inhibitor selectivity

IRF5 promotes inflammatory macrophage polarization and TH1-TH17 responses

MK2 targets AU-rich elements and regulates biosynthesis of tumor necrosis factor and interleukin-6 independently at different post-transcriptional levels

MAPKAP kinase 2 is essential for LPS-induced TNF-alpha biosynthesis

Selective pressure as an essential force in molecular evolution of myeloid leukemic clones: a view from the window of Fanconi anemia.

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1992-2002

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Jeffrey W. Tyner

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221738046

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22234699

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Toll-like receptor

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