Cytochrome P4501A1 is required for vascular dysfunction and hypertension induced by 2,3,7,8-tetrachlorodibenzo-p-dioxin. - Wikidata - awgldk - TriplyDB

Cytochrome P4501A1 is required for vascular dysfunction and hypertension induced by 2,3,7,8-tetrachlorodibenzo-p-dioxin.

Cytochrome P4501A1 is required for vascular dysfunction and hypertension induced by 2,3,7,8-tetrachlorodibenzo-p-dioxin.

http://www.wikidata.org/entity/Q34131893

about

Can persistent organic pollutants and plastic-associated chemicals cause cardiovascular disease?The aryl hydrocarbon receptor-activating effect of uremic toxins from tryptophan metabolism: a new concept to understand cardiovascular complications of chronic kidney disease Polychlorinated biphenyls and links to cardiovascular disease Nrf2 protects against 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD)-induced oxidative injury and steatohepatitis Exactly the same but different: promiscuity and diversity in the molecular mechanisms of action of the aryl hydrocarbon (dioxin) receptor Generation of reactive oxygen species by polyenylpyrroles derivatives causes DNA damage leading to G2/M arrest and apoptosis in human oral squamous cell carcinoma cells The Influence of α-Tocopherol on Serum Biochemical Markers During Experimentally Induced Pleuritis in Rats Exposed to Dioxin.Dietary Omega-3 Polyunsaturated Fatty Acids Prevent Vascular Dysfunction and Attenuate Cytochrome P4501A1 Expression by 2,3,7,8-Tetrachlorodibenzo-P-Dioxin.Role of CYP1A1 in modulating the vascular and blood pressure benefits of omega-3 polyunsaturated fatty acids Histopathological, ultrastructural, and immunohistochemical assessment of hippocampus structures of rats exposed to TCDD and high doses of tocopherol and acetylsalicylic acid.2,3,7,8-Tetrachlorodibenzo-p-dioxin treatment alters eicosanoid levels in several organs of the mouse in an aryl hydrocarbon receptor-dependent fashion EGCG protects endothelial cells against PCB 126-induced inflammation through inhibition of AhR and induction of Nrf2-regulated genes.Molecular targets that link dioxin exposure to toxicity phenotypes.Ameliorative effect of supplementation with L-glutamine on oxidative stress, DNA damage, cell viability and hepatotoxicity induced by 2,3,7,8-tetrachlorodibenzo-p-dioxin in rat hepatocyte cultures.Elevated blood pressure in cytochrome P4501A1 knockout mice is associated with reduced vasodilation to omega-3 polyunsaturated fatty acids The Effect of Exposure to Persistent Organic Pollutants on Metabolic Health among KOREAN Children during a 1-Year Follow-Up Biological effects of 6-formylindolo[3,2-b]carbazole (FICZ) in vivo are enhanced by loss of CYP1A function in an Ahr2-dependent manner.PCB 77 dechlorination products modulate pro-inflammatory events in vascular endothelial cells An overview of transcriptional regulation in response to toxicological insult.Omega-3 Polyunsaturated Fatty Acids Protect Against Cigarette Smoke-Induced Oxidative Stress and Vascular Dysfunction.miR-23a Regulates Cardiomyocyte Apoptosis by Targeting Manganese Superoxide Dismutase.AhR activation underlies the CYP1A autoinduction by A-998679 in rats.A less stressful alternative to oral gavage for pharmacological and toxicological studies in mice.Involvement of microRNAs in dioxin-induced liver damage in the mouse.The Aryl Hydrocarbon Receptor and Tumor Immunity.

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P2860

Cytochrome P4501A1 is required for vascular dysfunction and hypertension induced by 2,3,7,8-tetrachlorodibenzo-p-dioxin.

http://www.wikidata.org/entity/Q34131893

description

2010 nî lūn-bûn

@nan

2010 թուականի Յուլիսին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի հուլիսին հրատարակված գիտական հոդված

@hy

2010年の論文

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2010年論文

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2010年論文

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2010年論文

@zh-hk

2010年論文

@zh-mo

2010年論文

@zh-tw

2010年论文

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name

Cytochrome P4501A1 is required ...... 8-tetrachlorodibenzo-p-dioxin.

@ast

Cytochrome P4501A1 is required ...... 8-tetrachlorodibenzo-p-dioxin.

@en

Cytochrome P4501A1 is required ...... 8-tetrachlorodibenzo-p-dioxin.

@nl

type

label

Cytochrome P4501A1 is required ...... 8-tetrachlorodibenzo-p-dioxin.

@ast

Cytochrome P4501A1 is required ...... 8-tetrachlorodibenzo-p-dioxin.

@en

Cytochrome P4501A1 is required ...... 8-tetrachlorodibenzo-p-dioxin.

@nl

prefLabel

Cytochrome P4501A1 is required ...... 8-tetrachlorodibenzo-p-dioxin.

@ast

Cytochrome P4501A1 is required ...... 8-tetrachlorodibenzo-p-dioxin.

@en

Cytochrome P4501A1 is required ...... 8-tetrachlorodibenzo-p-dioxin.

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Toxicological Sciences

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Cytochrome P4501A1 is required ...... 8-tetrachlorodibenzo-p-dioxin.

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Janice K Huwe

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Jason A Scott

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Jason R Boberg

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Khalid M Elased

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Larry N Agbor

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Mary K Walker

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Phillip G Kopf

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P2860

Health status of Army Chemical Corps Vietnam veterans who sprayed defoliant in Vietnam

Chemistry and antihypertensive effects of tempol and other nitroxides

Comparison of the use of a physiologically based pharmacokinetic model and a classical pharmacokinetic model for dioxin exposure assessments

Cardiac hypertrophy in aryl hydrocarbon receptor null mice is correlated with elevated angiotensin II, endothelin-1, and mean arterial blood pressure

Evaluation of PCDD/F and dioxin-like PCB serum concentration data from the 2001-2002 National Health and Nutrition Examination Survey of the United States population.

2,3,7,8-tetrachlorodibenzo-p-dioxin increases reactive oxygen species production in human endothelial cells via induction of cytochrome P4501A1.

Relationship between serum concentrations of persistent organic pollutants and the prevalence of metabolic syndrome among non-diabetic adults: results from the National Health and Nutrition Examination Survey 1999-2002.

Comparisons of estimated human body burdens of dioxinlike chemicals and TCDD body burdens in experimentally exposed animals.

New target regions for human hypertension via comparative genomics

Reactive oxygen species, vascular oxidative stress, and redox signaling in hypertension: what is the clinical significance?

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537-546

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10.1093/TOXSCI/KFQ218

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2

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117

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2010-07-15T00:00:00Z

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45199868

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20634294

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