Coronavirus spike proteins in viral entry and pathogenesis.
about
TMPRSS2 and ADAM17 cleave ACE2 differentially and only proteolysis by TMPRSS2 augments entry driven by the severe acute respiratory syndrome coronavirus spike proteinAcquisition of macrophage tropism during the pathogenesis of feline infectious peritonitis is determined by mutations in the feline coronavirus spike proteinThe Severe Acute Respiratory Syndrome (SARS)-coronavirus 3a protein may function as a modulator of the trafficking properties of the spike proteinThe Coronavirus Spike Protein Is a Class I Virus Fusion Protein: Structural and Functional Characterization of the Fusion Core ComplexProteomic approaches to uncovering virus-host protein interactions during the progression of viral infectionA 193-amino acid fragment of the SARS coronavirus S protein efficiently binds angiotensin-converting enzyme 2A recombinant avian infectious bronchitis virus expressing a heterologous spike gene belonging to the 4/91 serotypeBat origin of human coronaviruses.Further Evidence for Bats as the Evolutionary Source of Middle East Respiratory Syndrome Coronavirus.Human neutralizing Fab molecules against severe acute respiratory syndrome coronavirus generated by phage display.MicroRNome analysis unravels the molecular basis of SARS infection in bronchoalveolar stem cells.The structural characterization and antigenicity of the S protein of SARS-CoV.Rapid peptide-based screening on the substrate specificity of severe acute respiratory syndrome (SARS) coronavirus 3C-like protease by matrix-assisted laser desorption/ionization time-of-flight mass spectrometry.Identification of major histocompatibility complex class I C molecule as an attachment factor that facilitates coronavirus HKU1 spike-mediated infection.The expression and antigenicity of a truncated spike-nucleocapsid fusion protein of severe acute respiratory syndrome-associated coronavirus.Identification of a new region of SARS-CoV S protein critical for viral entryA single tyrosine in the severe acute respiratory syndrome coronavirus membrane protein cytoplasmic tail is important for efficient interaction with spike proteinSevere acute respiratory syndrome coronavirus spike protein expressed by attenuated vaccinia virus protectively immunizes miceReceptor-independent spread of a highly neurotropic murine coronavirus JHMV strain from initially infected microglial cells in mixed neural culturesIdentification of a novel coronavirus in bats.Identification and characterization of the putative fusion peptide of the severe acute respiratory syndrome-associated coronavirus spike proteinHuman coronavirus NL63 employs the severe acute respiratory syndrome coronavirus receptor for cellular entry.Murine coronavirus evolution in vivo: functional compensation of a detrimental amino acid substitution in the receptor binding domain of the spike glycoprotein.Feline lectin activity is critical for the cellular entry of feline infectious peritonitis virus.A mechanism of virus-induced demyelination.Palmitoylation of SARS-CoV S protein is necessary for partitioning into detergent-resistant membranes and cell-cell fusion but not interaction with M protein.Pathogenesis of murine coronavirus in the central nervous system.Contribution of trafficking signals in the cytoplasmic tail of the infectious bronchitis virus spike protein to virus infection.Intracellular targeting signals contribute to localization of coronavirus spike proteins near the virus assembly site.A conserved domain in the coronavirus membrane protein tail is important for virus assembly.Coronavirus pathogenesis and the emerging pathogen severe acute respiratory syndrome coronavirus.Coronavirus genotype diversity and prevalence of infection in wild carnivores in the Serengeti National Park, Tanzania.Severe acute respiratory syndrome coronavirus (SARS-CoV) infection inhibition using spike protein heptad repeat-derived peptides.False-positive results in a recombinant severe acute respiratory syndrome-associated coronavirus (SARS-CoV) nucleocapsid-based western blot assay were rectified by the use of two subunits (S1 and S2) of spike for detection of antibody to SARS-CoV.Molecular epidemiology of bovine coronavirus on the basis of comparative analyses of the S geneMouse hepatitis virus infection upregulates genes involved in innate immune responses.Identification of an antigenic determinant on the S2 domain of the severe acute respiratory syndrome coronavirus spike glycoprotein capable of inducing neutralizing antibodies.The avian coronavirus infectious bronchitis virus undergoes direct low-pH-dependent fusion activation during entry into host cellsN-terminal domain of the murine coronavirus receptor CEACAM1 is responsible for fusogenic activation and conformational changes of the spike proteinHuman coronavirus NL63 utilizes heparan sulfate proteoglycans for attachment to target cells
P2860
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P2860
Coronavirus spike proteins in viral entry and pathogenesis.
description
2001 nî lūn-bûn
@nan
2001 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2001 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2001年の論文
@ja
2001年論文
@yue
2001年論文
@zh-hant
2001年論文
@zh-hk
2001年論文
@zh-mo
2001年論文
@zh-tw
2001年论文
@wuu
name
Coronavirus spike proteins in viral entry and pathogenesis.
@ast
Coronavirus spike proteins in viral entry and pathogenesis.
@en
Coronavirus spike proteins in viral entry and pathogenesis.
@nl
type
label
Coronavirus spike proteins in viral entry and pathogenesis.
@ast
Coronavirus spike proteins in viral entry and pathogenesis.
@en
Coronavirus spike proteins in viral entry and pathogenesis.
@nl
prefLabel
Coronavirus spike proteins in viral entry and pathogenesis.
@ast
Coronavirus spike proteins in viral entry and pathogenesis.
@en
Coronavirus spike proteins in viral entry and pathogenesis.
@nl
P356
P1433
P1476
Coronavirus spike proteins in viral entry and pathogenesis.
@en
P2093
Buchmeier MJ
Gallagher TM
P304
P356
10.1006/VIRO.2000.0757
P407
P577
2001-01-01T00:00:00Z