Localization of proliferating cell nuclear antigen, vimentin, c-Fos, and clusterin in the postischemic kidney. Evidence for a heterogenous genetic response among nephron segments, and a large pool of mitotically active and dedifferentiated cells.

Localization of proliferating cell nuclear antigen, vimentin, c-Fos, and clusterin in the postischemic kidney. Evidence for a heterogenous genetic response among nephron segments, and a large pool of mitotically active and dedifferentiated cells.

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http://www.wikidata.org/entity/Q34146136

Q34146136

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The renal papilla is a niche for adult kidney stem cells Mesenchymal-epithelial transition in epithelial response to injury: the role of Foxc2 Chordin-like 1 and twisted gastrulation 1 regulate BMP signaling following kidney injury Role of mitochondrial dysfunction in cellular responses to S-(1,2-dichlorovinyl)-L-cysteine in primary cultures of human proximal tubular cells New tides: using zebrafish to study renal regeneration Stem cells: potential and challenges for kidney repair Renal stem cells: fact or science fiction?Kidney regeneration: common themes from the embryo to the adult Collective epithelial migration drives kidney repair after acute injury MicroRNA expression data reveals a signature of kidney damage following ischemia reperfusion injury Collective cell migration drives morphogenesis of the kidney nephron Role of Oxidative Stress in Drug-Induced Kidney Injury Cell-cycle arrest and acute kidney injury: the light and the dark sides Severe renal mass reduction impairs recovery and promotes fibrosis after AKI CHOP is implicated in programmed cell death in response to impaired function of the endoplasmic reticulum Kidney injury molecule-1 is a phosphatidylserine receptor that confers a phagocytic phenotype on epithelial cells Expression of fibronectin splice variants in the postischemic rat kidney Extracellular signal-regulated kinase activation during renal ischemia/reperfusion mediates focal adhesion dissolution and renal injury Hypertension-related, calcium-regulated gene (HCaRG/COMMD5) and kidney diseases: HCaRG accelerates tubular repair Role of PDGF B-chain and PDGF receptors in rat tubular regeneration after acute injury Evaluation of putative biomarkers of nephrotoxicity after exposure to ochratoxin a in vivo and in vitro.Targeting Endogenous Repair Pathways after AKI.Evaluation of urinary tissue inhibitor of metalloproteinase-2 in acute kidney injury: a prospective observational study.Intrarenal cells, not bone marrow-derived cells, are the major source for regeneration in postischemic kidney.Molecular and functional properties of lung SP cells.Proteomics for biomarker discovery in acute kidney injury Fate tracing reveals the pericyte and not epithelial origin of myofibroblasts in kidney fibrosis Characterization of renal toxicity in mice administered the marine biotoxin domoic Acid.Biomarkers of nephrotoxic acute kidney injury Lessons learned from ischemic and cisplatin-induced nephrotoxicity in animals.Remote Ischemic Preconditioning and Protection of the Kidney--A Novel Therapeutic Option.Dragon enhances BMP signaling and increases transepithelial resistance in kidney epithelial cells.Discovery and validation of cell cycle arrest biomarkers in human acute kidney injury Role of medullary progenitor cells in epithelial cell migration and proliferation.Restoration of tubular epithelial cells during repair of the postischemic kidney occurs independently of bone marrow-derived stem cells.Differentiated kidney epithelial cells repair injured proximal tubule.Kidney tubular epithelium is restored without replacement with bone marrow-derived cells during repair after ischemic injury.Gene expression analysis reveals the cell cycle and kinetochore genes participating in ischemia reperfusion injury and early development in kidney.Defining the acute kidney injury and repair transcriptome G-protein βγ subunit dimers modulate kidney repair after ischemia-reperfusion injury in rats.

P2860

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P2860

Localization of proliferating cell nuclear antigen, vimentin, c-Fos, and clusterin in the postischemic kidney. Evidence for a heterogenous genetic response among nephron segments, and a large pool of mitotically active and dedifferentiated cells.

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http://www.wikidata.org/entity/Q34146136

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1994 nî lūn-bûn

@nan

1994 թուականի Մայիսին հրատարակուած գիտական յօդուած

@hyw

1994 թվականի մայիսին հրատարակված գիտական հոդված

@hy

1994年の論文

@ja

1994年論文

@yue

1994年論文

@zh-hant

1994年論文

@zh-hk

1994年論文

@zh-mo

1994年論文

@zh-tw

1994年论文

@wuu