Rho protein inactivation induced apoptosis of cultured human endothelial cells. - Wikidata - awgldk - TriplyDB

Rho protein inactivation induced apoptosis of cultured human endothelial cells.

Rho protein inactivation induced apoptosis of cultured human endothelial cells.

http://www.wikidata.org/entity/Q34149118

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Lactobacillus delbrueckii ssp. bulgaricus B-30892 can inhibit cytotoxic effects and adhesion of pathogenic Clostridium difficile to Caco-2 cells Preadministration of High-Dose Salicylates, Suppressors of NF- B Activation, May Increase the Chemosensitivity of Many Cancers: An Example of Proapoptotic Signal Modulation Therapy Distinct biological activities of C3 and ADP-ribosyltransferase-deficient C3-E174Q Clostridium difficile toxins: mechanism of action and role in disease.A vascular cell-restricted RhoGAP, p73RhoGAP, is a key regulator of angiogenesis Rho activation patterns after spinal cord injury and the role of activated Rho in apoptosis in the central nervous system Clostridium difficile Toxins A and B: Insights into Pathogenic Properties and Extraintestinal Effects The Role of Rho GTPases in Toxicity of Clostridium difficile Toxins Phenylalanine activates the mitochondria-mediated apoptosis through the RhoA/Rho-associated kinase pathway in cortical neurons The Tumor Suppressor, p190RhoGAP, Differentially Initiates Apoptosis and Confers Docetaxel Sensitivity to Breast Cancer Cells.Caspase and bid involvement in Clostridium difficile toxin A-induced apoptosis and modulation of toxin A effects by glutamine and alanyl-glutamine in vivo and in vitro Focal adhesion kinase and endothelial cell apoptosis.A translocated bacterial protein protects vascular endothelial cells from apoptosis.Utility of Clostridium difficile toxin B for inducing anti-tumor immunity.Small G-protein RhoE is underexpressed in prostate cancer and induces cell cycle arrest and apoptosis.Translocation domain mutations affecting cellular toxicity identify the Clostridium difficile toxin B pore.Primary human colonic myofibroblasts are resistant to Clostridium difficile toxin A-induced, but not toxin B-induced, cell death.Enzymatically active Rho and Rac small-GTPases are involved in the establishment of the vacuolar membrane after Toxoplasma gondii invasion of host cells.Toxin-mediated paracellular transport of antitoxin antibodies facilitates protection against Clostridium difficile infection.Protective effects of protocatechuic acid on retinal ganglion cells from oxidative damage induced by H2O2.The Makes Caterpillars Floppy (MCF)-Like Domain of Vibrio vulnificus Induces Mitochondrion-Mediated Apoptosis Toward a structural understanding of Clostridium difficile toxins A and B.Development of a Novel Vaccine Containing Binary Toxin for the Prevention of Clostridium difficile Disease with Enhanced Efficacy against NAP1 Strains Agonist-induced down-regulation of endogenous protein kinase c α through an endolysosomal mechanism.Regulation of Apoptosis by Gram-Positive Bacteria: Mechanistic Diversity and Consequences for Immunity.Streptococcus pneumoniae-induced caspase 6-dependent apoptosis in lung epithelium.The involvement of Bcl-2 family proteins in AKT-regulated cell survival in cisplatin resistant epithelial ovarian cancer.The enterotoxicity of Clostridium difficile toxins.Rho Kinase and Dopaminergic Degeneration: A Promising Therapeutic Target for Parkinson's Disease.Salubrinal protects against Clostridium difficile toxin B-induced CT26 cell death.Enteric glial cells are susceptible to Clostridium difficile toxin B.Pathogenic effects of glucosyltransferase from Clostridium difficile toxins.Lysyl oxidase (lox) gene deficiency affects osteoblastic phenotype.The Rho kinase inhibitor fasudil is involved in p53-mediated apoptosis in human hepatocellular carcinoma cells.Antiapoptotic proteins Bcl-2 and Bcl-XL inhibit Clostridium difficile toxin A-induced cell death in human epithelial cells.Zoledronate has an antitumor effect and induces actin rearrangement in dexamethasone-resistant myeloma cells.Difference in the cytotoxic effects of toxin B from Clostridium difficile strain VPI 10463 and toxin B from variant Clostridium difficile strain 1470.Legionella pneumophila-induced PKCalpha-, MAPK-, and NF-kappaB-dependent COX-2 expression in human lung epithelium.Pneumococci induced TLR- and Rac1-dependent NF-kappaB-recruitment to the IL-8 promoter in lung epithelial cells.Lethal toxin from Clostridium sordellii induces apoptotic cell death by disruption of mitochondrial homeostasis in HL-60 cells.

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P2860

Rho protein inactivation induced apoptosis of cultured human endothelial cells.

http://www.wikidata.org/entity/Q34149118

description

2002 nî lūn-bûn

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2002 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

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2002 թվականի հոտեմբերին հրատարակված գիտական հոդված

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2002年の論文

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2002年論文

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2002年論文

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2002年論文

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2002年論文

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2002年論文

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2002年论文

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name

Rho protein inactivation induced apoptosis of cultured human endothelial cells.

@ast

Rho protein inactivation induced apoptosis of cultured human endothelial cells.

@en

Rho protein inactivation induced apoptosis of cultured human endothelial cells.

@nl

type

label

Rho protein inactivation induced apoptosis of cultured human endothelial cells.

@ast

Rho protein inactivation induced apoptosis of cultured human endothelial cells.

@en

Rho protein inactivation induced apoptosis of cultured human endothelial cells.

@nl

prefLabel

Rho protein inactivation induced apoptosis of cultured human endothelial cells.

@ast

Rho protein inactivation induced apoptosis of cultured human endothelial cells.

@en

Rho protein inactivation induced apoptosis of cultured human endothelial cells.

@nl

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AJPLUNG.00467.2001

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American Journal of Physiology - Lung Cellular and Molecular Physiology

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Rho protein inactivation induced apoptosis of cultured human endothelial cells.

@en

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Bernd Schmeck

http://www.w3.org/2001/XMLSchema#string

Christoph V Eichel-Streiber

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Joachim Seybold

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Matthias Krüll

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Norbert Suttorp

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Phillipe Dje N'Guessan

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Stefan Hippenstiel

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P2860

Mammalian caspases: structure, activation, substrates, and functions during apoptosis

Small GTP-binding proteins

The biochemistry of apoptosis

Inactivation of the small GTPase Rho disrupts cellular attachment and induces adhesion-dependent and adhesion-independent apoptosis.

Cytotoxic necrotizing factor type 1 of uropathogenic Escherichia coli kills cultured human uroepithelial 5637 cells by an apoptotic mechanism

Signaling to Rho GTPases.

Apoptosis in atherosclerosis: beneficial or detrimental?

Regulation of the cytoskeleton and cell adhesion by the Rho family GTPases in mammalian cells.

Mitochondria as the central control point of apoptosis.

Endothelial cell apoptosis in angiogenesis and vessel regression.

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11163600

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12225960

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