Aldosterone blunts tubuloglomerular feedback by activating macula densa mineralocorticoid receptors. - Wikidata - awgldk - TriplyDB

Aldosterone blunts tubuloglomerular feedback by activating macula densa mineralocorticoid receptors.

Aldosterone blunts tubuloglomerular feedback by activating macula densa mineralocorticoid receptors.

http://www.wikidata.org/entity/Q34152478

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Recent advances in renal hemodynamics: insights from bench experiments and computer simulations Aldosterone sensitizes connecting tubule glomerular feedback via the aldosterone receptor GPR30.Testosterone enhances tubuloglomerular feedback by increasing superoxide production in the macula densa.Enhanced expression and activity of Nox2 and Nox4 in the macula densa in ANG II-induced hypertensive mice.Oxidative status in the macula densa modulates tubuloglomerular feedback responsiveness in angiotensin II-induced hypertension Iodinated contrast media cause direct tubular cell damage, leading to oxidative stress, low nitric oxide, and impairment of tubuloglomerular feedback.The Association between Glomerular Hyperfiltration and Left Ventricular Structure and Function in Patients with Primary Aldosteronism.Interaction between nitric oxide and superoxide in the macula densa in aldosterone-induced alterations of tubuloglomerular feedback Inhibition of Nitric Oxide Synthase 1 Induces Salt-Sensitive Hypertension in Nitric Oxide Synthase 1α Knockout and Wild-Type Mice Endothelin-1, oxidative stress, and endogenous angiotensin II: mechanisms of angiotensin II type I receptor autoantibody-enhanced renal and blood pressure response during pregnancy Neurohormonal interactions on the renal oxygen delivery and consumption in haemorrhagic shock-induced acute kidney injury.Renal neurohormonal regulation in heart failure decompensation.Mechanisms of connecting tubule glomerular feedback enhancement by aldosterone.Role of intratubular pressure during the ischemic phase in acute kidney injury.Role of the Primary Cilia on the Macula Densa and Thick Ascending Limbs in Regulation of Sodium Excretion and Hemodynamics.Macula Densa Nitric Oxide Synthase 1β Protects against Salt-Sensitive Hypertension.Renin Phenotypes Characterize Vascular Disease, Autonomous Aldosteronism, and Mineralocorticoid Receptor Activity.Renal tubule insulin receptor modestly promotes elevated blood pressure and markedly stimulates glucose reabsorption

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Aldosterone blunts tubuloglomerular feedback by activating macula densa mineralocorticoid receptors.

http://www.wikidata.org/entity/Q34152478

description

2012 nî lūn-bûn

@nan

2012 թուականի Փետրուարին հրատարակուած գիտական յօդուած

@hyw

2012 թվականի փետրվարին հրատարակված գիտական հոդված

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2012年の論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年论文

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name

Aldosterone blunts tubuloglome ...... a mineralocorticoid receptors.

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Aldosterone blunts tubuloglome ...... a mineralocorticoid receptors.

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Aldosterone blunts tubuloglome ...... a mineralocorticoid receptors.

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type

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Aldosterone blunts tubuloglome ...... a mineralocorticoid receptors.

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Aldosterone blunts tubuloglome ...... a mineralocorticoid receptors.

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Aldosterone blunts tubuloglome ...... a mineralocorticoid receptors.

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Aldosterone blunts tubuloglome ...... a mineralocorticoid receptors.

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Aldosterone blunts tubuloglome ...... a mineralocorticoid receptors.

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Aldosterone blunts tubuloglome ...... a mineralocorticoid receptors.

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HYPERTENSIONAHA.111.173195

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Aldosterone blunts tubuloglome ...... sa mineralocorticoid receptors

@en

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Celso E Gomez-Sanchez

http://www.w3.org/2001/XMLSchema#string

Deyin Lu

http://www.w3.org/2001/XMLSchema#string

John E Hall

http://www.w3.org/2001/XMLSchema#string

Liang Cheng

http://www.w3.org/2001/XMLSchema#string

Luis A Juncos

http://www.w3.org/2001/XMLSchema#string

R Davis Manning

http://www.w3.org/2001/XMLSchema#string

Ruisheng Liu

http://www.w3.org/2001/XMLSchema#string

Yiling Fu

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P2860

Isoforms and functions of NAD(P)H oxidase at the macula densa.

Evidence for the role of nitric oxide in macula densa control of glomerular hemodynamics.

The nongenomic actions of aldosterone.

Aldosterone activates Na+/H+ exchange and raises cytoplasmic pH in target cells of the amphibian kidney

Aldosterone stimulates superoxide production in macula densa cells

Nongenomic stimulation of vacuolar H+-ATPases in intercalated renal tubule cells by aldosterone

Rapid activation of Na+/H+ exchange by aldosterone in renal epithelial cells requires Ca2+ and stimulation of a plasma membrane proton conductance.

Immunohistochemical localization of renal mineralocorticoid receptor by using an anti-idiotypic antibody that is an internal image of aldosterone.

The mineralocorticoid aldosterone activates a proton conductance in cultured kidney cells.

Rapid activation of Na+/H+-exchange in MDCK cells by aldosterone involves MAP-kinase ERK1/2.

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599-606

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scholarly article

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10.1161/HYPERTENSIONAHA.111.173195

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2012-02-06T00:00:00Z

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22311906

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3299000

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