CCL2-expressing astrocytes mediate the extravasation of T lymphocytes in the brain. Evidence from patients with glioma and experimental models in vivo.
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Neuroantibody biomarkers: links and challenges in environmental neurodegeneration and autoimmunityMultiple sclerosis: molecular mechanisms and therapeutic opportunitiesCell-selective knockout and 3D confocal image analysis reveals separate roles for astrocyte-and endothelial-derived CCL2 in neuroinflammation.The role of astrocytes in CNS tumors: pre-clinical models and novel imaging approachesMicroarray data analysis to identify crucial genes regulated by CEBPB in human SNB19 glioma cellsConditional ablation of astroglial CCL2 suppresses CNS accumulation of M1 macrophages and preserves axons in mice with MOG peptide EAE.A role for human brain pericytes in neuroinflammation.Astrocyte CCL2 sustains immune cell infiltration in chronic experimental autoimmune encephalomyelitis.Interaction of astrocytes and T cells in physiological and pathological conditionsTyrosine receptor kinase B is a drug target in astrocytomas.Ectonucleotidase activity and immunosuppression in astrocyte-CD4 T cell bidirectional signaling.Estrogen mediates neuroprotection and anti-inflammatory effects during EAE through ERα signaling on astrocytes but not through ERβ signaling on astrocytes or neurons.Glial-mediated inflammation underlying parkinsonism.Imaging the microanatomy of astrocyte-T-cell interactions in immune-mediated inflammation.Circulating T regulatory cells migration and phenotype in glioblastoma patients: an in vitro study.Central CCL2 signaling onto MCH neurons mediates metabolic and behavioral adaptation to inflammation.CCL2 Produced by the Glioma Microenvironment Is Essential for the Recruitment of Regulatory T Cells and Myeloid-Derived Suppressor Cells.Treatment with polyamine oxidase inhibitor reduces microglial activation and limits vascular injury in ischemic retinopathy.Downregulation of CCL2 induced by the upregulation of microRNA-206 is associated with the severity of HEV71 encephalitis.Astrocytes play a key role in EAE pathophysiology by orchestrating in the CNS the inflammatory response of resident and peripheral immune cells and by suppressing remyelination.Imbalance of immunological synapse-kinapse states reflects tumor escape to immunity in glioblastoma
P2860
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P2860
CCL2-expressing astrocytes mediate the extravasation of T lymphocytes in the brain. Evidence from patients with glioma and experimental models in vivo.
description
2012 nî lūn-bûn
@nan
2012 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2012 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
name
CCL2-expressing astrocytes med ...... d experimental models in vivo.
@ast
CCL2-expressing astrocytes med ...... d experimental models in vivo.
@en
CCL2-expressing astrocytes med ...... d experimental models in vivo.
@nl
type
label
CCL2-expressing astrocytes med ...... d experimental models in vivo.
@ast
CCL2-expressing astrocytes med ...... d experimental models in vivo.
@en
CCL2-expressing astrocytes med ...... d experimental models in vivo.
@nl
prefLabel
CCL2-expressing astrocytes med ...... d experimental models in vivo.
@ast
CCL2-expressing astrocytes med ...... d experimental models in vivo.
@en
CCL2-expressing astrocytes med ...... d experimental models in vivo.
@nl
P2093
P2860
P1433
P1476
CCL2-expressing astrocytes med ...... d experimental models in vivo.
@en
P2093
Ana Perez-Vallés
Aurora Gómez
Carlos Barcia
Carmen María Ros
Emiliano Fernández-Villalba
Francisco Ros-Bernal
José M Gallego-Sanchez
Maria Angeles Carrillo-de Sauvage
Maria-Trinidad Herrero
P2860
P304
P356
10.1371/JOURNAL.PONE.0030762
P407
P577
2012-02-02T00:00:00Z