Systemic lipopolysaccharide protects the brain from ischemic injury by reprogramming the response of the brain to stroke: a critical role for IRF3
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Astrocytic Toll-like receptor 3 is associated with ischemic preconditioning-induced protection against brain ischemia in rodentsEthanol-Induced Neurodegeneration and Glial Activation in the Developing BrainChemical Conditioning as an Approach to Ischemic Stroke Tolerance: Mitochondria as the TargetTolerance to ischemia - an increasingly complex biologyMechanisms and potential therapeutic applications of microglial activation after brain injuryCytokines: their role in stroke and potential use as biomarkers and therapeutic targetsThe transcriptome of cerebral ischemiaImmune mechanisms in cerebral ischemic toleranceEndogenous IFN-β signaling exerts anti-inflammatory actions in experimentally induced focal cerebral ischemiaAgricultural exposures and stroke mortality in the Agricultural Health StudyNetwork-Based Approach to Identify Potential Targets and Drugs that Promote Neuroprotection and Neurorepair in Acute Ischemic StrokeThe immunology of stroke: from mechanisms to translationSystemic lipopolysaccharide induces cochlear inflammation and exacerbates the synergistic ototoxicity of kanamycin and furosemide.Microglial ablation and lipopolysaccharide preconditioning affects pilocarpine-induced seizures in mice.Activation of inflammatory signaling by lipopolysaccharide produces a prolonged increase of voluntary alcohol intake in mice.The absence of P2X7 receptors (P2rx7) on non-haematopoietic cells leads to selective alteration in mood-related behaviour with dysregulated gene expression and stress reactivity in mice.LPS and IL-1 differentially activate mouse and human astrocytes: role of CD14Innate inflammatory responses in stroke: mechanisms and potential therapeutic targets.Toll-like receptors and ischemic brain injury.Toll-like receptor 4 in CNS pathologiesProlonged, 24-h delayed peripheral inflammation increases short- and long-term functional impairment and histopathological damage after focal ischemia in the rat.Stroke research at a crossroad: asking the brain for directionsThe Immune Response to Acute Focal Cerebral Ischemia and Associated Post-stroke Immunodepression: A Focused Review.Novel role for the innate immune receptor Toll-like receptor 4 (TLR4) in the regulation of the Wnt signaling pathway and photoreceptor apoptosisThe TLR9 ligand, CpG-ODN, induces protection against cerebral ischemia/reperfusion injury via activation of PI3K/Akt signaling.Transcriptional expression patterns triggered by chemically distinct neuroprotective molecules.Modeling dynamic regulatory processes in stroke.Helicobacter infection alters MyD88 and Trif signalling in response to intestinal ischaemia-reperfusion.Reprogramming the response to stroke by preconditioningToll-like receptor tolerance as a mechanism for neuroprotection.Defining the players in higher-order networks: predictive modeling for reverse engineering functional influence networks.Disruption of downstream MyD88 or TRIF Toll-like receptor signaling does not protect against cerebral ischemiaImmune priming and experimental glaucoma: the effect of prior systemic lipopolysaccharide challenge on tissue outcomes after optic nerve injury.Multiple preconditioning paradigms converge on interferon regulatory factor-dependent signaling to promote tolerance to ischemic brain injury.Poly (I:C) therapy decreases cerebral ischaemia/reperfusion injury via TLR3-mediated prevention of Fas/FADD interactionLong-lasting protection in brain trauma by endotoxin preconditioningToll-like receptors in cerebral ischemic inflammatory injury.LPS preconditioning redirects TLR signaling following stroke: TRIF-IRF3 plays a seminal role in mediating tolerance to ischemic injury.Function and mechanism of toll-like receptors in cerebral ischemic tolerance: from preconditioning to treatment.CpG preconditioning regulates miRNA expression that modulates genomic reprogramming associated with neuroprotection against ischemic injury.
P2860
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P2860
Systemic lipopolysaccharide protects the brain from ischemic injury by reprogramming the response of the brain to stroke: a critical role for IRF3
description
2009 nî lūn-bûn
@nan
2009 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
name
Systemic lipopolysaccharide pr ...... roke: a critical role for IRF3
@ast
Systemic lipopolysaccharide pr ...... roke: a critical role for IRF3
@en
Systemic lipopolysaccharide pr ...... roke: a critical role for IRF3
@nl
type
label
Systemic lipopolysaccharide pr ...... roke: a critical role for IRF3
@ast
Systemic lipopolysaccharide pr ...... roke: a critical role for IRF3
@en
Systemic lipopolysaccharide pr ...... roke: a critical role for IRF3
@nl
prefLabel
Systemic lipopolysaccharide pr ...... roke: a critical role for IRF3
@ast
Systemic lipopolysaccharide pr ...... roke: a critical role for IRF3
@en
Systemic lipopolysaccharide pr ...... roke: a critical role for IRF3
@nl
P2093
P2860
P1476
Systemic lipopolysaccharide pr ...... roke: a critical role for IRF3
@en
P2093
Banu Gopalan
Brian Hunter
Christina A Harrington
Mary P Stenzel-Poore
Philberta Y Leung
Susan L Stevens
P2860
P304
P356
10.1523/JNEUROSCI.2496-09.2009
P407
P577
2009-08-01T00:00:00Z