Tat and trans-activation-responsive (TAR) RNA-independent induction of HIV-1 long terminal repeat by human and murine cyclin T1 requires Sp1.
about
HIV-1 gene expression: lessons from provirus and non-integrated DNAInhibition of HIV-1 replication in primary human monocytes by the IkappaB-alphaS32/36A repressor of NF-kappaBCOUP-TF interacting protein 2 represses the initial phase of HIV-1 gene transcription in human microglial cellsThe apical loop of the HIV-1 TAR RNA hairpin is stabilized by a cross-loop base pairSmall molecules targeted to a non-catalytic "RVxF" binding site of protein phosphatase-1 inhibit HIV-1Activation of HIV-1 from latent infection via synergy of RUNX1 inhibitor Ro5-3335 and SAHARegulation of human immunodeficiency virus type 1 gene expression by clade-specific Tat proteins.Lower levels of gamma interferon expressed by a pseudotyped single-cycle simian immunodeficiency virus enhance immunogenicity in rats.Discovery of a small molecule Tat-trans-activation-responsive RNA antagonist that potently inhibits human immunodeficiency virus-1 replication.Regulation of HIV-1 transcription in cells of the monocyte-macrophage lineage.HIV-1 reactivation induced by the periodontal pathogens Fusobacterium nucleatum and Porphyromonas gingivalis involves Toll-like receptor 2 [corrected] and 9 activation in monocytes/macrophages.CD4-specific transgenic expression of human cyclin T1 markedly increases human immunodeficiency virus type 1 (HIV-1) production by CD4+ T lymphocytes and myeloid cells in mice transgenic for a provirus encoding a monocyte-tropic HIV-1 isolate.Iron chelators of the di-2-pyridylketone thiosemicarbazone and 2-benzoylpyridine thiosemicarbazone series inhibit HIV-1 transcription: identification of novel cellular targets--iron, cyclin-dependent kinase (CDK) 2, and CDK9Expression of a protein phosphatase 1 inhibitor, cdNIPP1, increases CDK9 threonine 186 phosphorylation and inhibits HIV-1 transcriptionPhenyl-1-Pyridin-2yl-ethanone-based iron chelators increase IκB-α expression, modulate CDK2 and CDK9 activities, and inhibit HIV-1 transcriptionGenetic selection for context-dependent stochastic phenotypes: Sp1 and TATA mutations increase phenotypic noise in HIV-1 gene expression.HIV-1 transcriptional regulation in the central nervous system and implications for HIV cure researchTRIM22 inhibits HIV-1 transcription independently of its E3 ubiquitin ligase activity, Tat, and NF-kappaB-responsive long terminal repeat elements.Expression of human endogenous retrovirus type K (HML-2) is activated by the Tat protein of HIV-1.The presence of HIV-1 Tat protein second exon delays fas protein-mediated apoptosis in CD4+ T lymphocytes: a potential mechanism for persistent viral productionCNS-specific regulatory elements in brain-derived HIV-1 strains affect responses to latency-reversing agents with implications for cure strategies.Inhibition of HIV-1 transcription and replication by a newly identified cyclin T1 splice variant.PU.1 binding to ets motifs within the equine infectious anemia virus long terminal repeat (LTR) enhancer: regulation of LTR activity and virus replication in macrophages.Human immunodeficiency virus type 1 subtypes have a distinct long terminal repeat that determines the replication rate in a host-cell-specific mannerRegulation of HIV-1 transcription at 3% versus 21% oxygen concentrationHIV-1 Tat directly binds to NFkappaB enhancer sequence: role in viral and cellular gene expression.Host Proteins Ku and HMGA1 As Participants of HIV-1 Transcription.HIV-1 Tat and Viral Latency: What We Can Learn from Naturally Occurring Sequence Variations.Underlying mechanisms of HIV-1 latency.O-linked N-acetylglucosaminylation of Sp1 inhibits the human immunodeficiency virus type 1 promoter.Cross-interaction between JC virus agnoprotein and human immunodeficiency virus type 1 (HIV-1) Tat modulates transcription of the HIV-1 long terminal repeat in glial cells.Artificial zinc finger fusions targeting Sp1-binding sites and the trans-activator-responsive element potently repress transcription and replication of HIV-1.Granulin and granulin repeats interact with the Tat.P-TEFb complex and inhibit Tat transactivation.Imperatorin inhibits HIV-1 replication through an Sp1-dependent pathway.Dominant negative mutant cyclin T1 proteins inhibit HIV transcription by specifically degrading Tat.RNA-mediated displacement of an inhibitory snRNP complex activates transcription elongation.Suv39H1 and HP1gamma are responsible for chromatin-mediated HIV-1 transcriptional silencing and post-integration latency.Hexamethylbisacetamide and disruption of human immunodeficiency virus type 1 latency in CD4(+) T cells.High attenuation and immunogenicity of a simian immunodeficiency virus expressing a proteolysis-resistant inhibitor of NF-kappaB.Functional Studies of CCAAT/Enhancer Binding Protein Site Located Downstream of the Transcriptional Start Site.
P2860
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P2860
Tat and trans-activation-responsive (TAR) RNA-independent induction of HIV-1 long terminal repeat by human and murine cyclin T1 requires Sp1.
description
2002 nî lūn-bûn
@nan
2002 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2002 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
2002年の論文
@ja
2002年論文
@yue
2002年論文
@zh-hant
2002年論文
@zh-hk
2002年論文
@zh-mo
2002年論文
@zh-tw
2002年论文
@wuu
name
Tat and trans-activation-respo ...... murine cyclin T1 requires Sp1.
@ast
Tat and trans-activation-respo ...... murine cyclin T1 requires Sp1.
@en
Tat and trans-activation-responsive
@nl
type
label
Tat and trans-activation-respo ...... murine cyclin T1 requires Sp1.
@ast
Tat and trans-activation-respo ...... murine cyclin T1 requires Sp1.
@en
Tat and trans-activation-responsive
@nl
prefLabel
Tat and trans-activation-respo ...... murine cyclin T1 requires Sp1.
@ast
Tat and trans-activation-respo ...... murine cyclin T1 requires Sp1.
@en
Tat and trans-activation-responsive
@nl
P2093
P2860
P356
P1476
Tat and trans-activation-respo ...... murine cyclin T1 requires Sp1.
@en
P2093
Kuan-Teh Jeang
Monsef Benkirane
Venkat S R K Yedavalli
P2860
P304
P356
10.1074/JBC.M209162200
P407
P577
2002-11-27T00:00:00Z