Lymphoid malignancies: the dark side of B-cell differentiation. - Wikidata - awgldk - TriplyDB

Lymphoid malignancies: the dark side of B-cell differentiation.

Lymphoid malignancies: the dark side of B-cell differentiation.

http://www.wikidata.org/entity/Q34162187

about

Selective generation of functional somatically mutated IgM+CD27+, but not Ig isotype-switched, memory B cells in X-linked lymphoproliferative disease Risk of non-Hodgkin lymphoma in association with germline variation in complement genes Molecular subtypes of diffuse large B-cell lymphoma arise by distinct genetic pathways Inactivation of the PRDM1/BLIMP1 gene in diffuse large B cell lymphoma Mutation of chromatin modifiers; an emerging hallmark of germinal center B-cell lymphomas Of mice and women: a comparative tissue biology perspective of breast stem cells and differentiation Fetal Microchimerism in Cancer Protection and Promotion: Current Understanding in Dogs and the Implications for Human Health The c-Rel transcription factor and B-cell proliferation: a deal with the devil CD19 is a major B cell receptor-independent activator of MYC-driven B-lymphomagenesis The copper chelator ATN-224 induces caspase-independent cell death in diffuse large B cell lymphoma Genome-wide association analysis of chronic lymphocytic leukaemia, Hodgkin lymphoma and multiple myeloma identifies pleiotropic risk loci Parallel gene expression profiling of mantle cell lymphoma - how do we transform 'omics data into clinical practice A library of gene expression signatures to illuminate normal and pathological lymphoid biology.Overexpression of an activated REL mutant enhances the transformed state of the human B-lymphoma BJAB cell line and alters its gene expression profile Immunophenotypic analysis of AIDS-related diffuse large B-cell lymphoma and clinical implications in patients from AIDS Malignancies Consortium clinical trials 010 and 034 AID produces DNA double-strand breaks in non-Ig genes and mature B cell lymphomas with reciprocal chromosome translocations.The rise and fall of long-lived humoral immunity: terminal differentiation of plasma cells in health and disease.The regulatory logic of the NF-kappaB signaling system.Intestinal microbiome and lymphoma development NF-kappaB/STAT3/PI3K signaling crosstalk in iMyc E mu B lymphoma.The significance of FOXP1 in diffuse large B-cell lymphoma.Genetic variation in cell death genes and risk of non-Hodgkin lymphoma A gene panel, including LRP12, is frequently hypermethylated in major types of B-cell lymphoma.Inhibiting NF-κB activation by small molecules as a therapeutic strategy.Msh6 protects mature B cells from lymphoma by preserving genomic stability.Organizational structure and the periphery of the gene regulatory network in B-cell lymphoma.The biology of human lymphoid malignancies revealed by gene expression profiling.Lymphoma caused by intestinal microbiota.Tetraspanin CD37 protects against the development of B cell lymphoma Critical role of SHP2 (PTPN11) signaling in germinal center-derived lymphoma.c-FLIP is involved in tumor progression of peripheral T-cell lymphoma and targeted by histone deacetylase inhibitors.Myeloid cell TRAF3 regulates immune responses and inhibits inflammation and tumor development in mice.Follicular dendritic cell-induced microRNA-mediated upregulation of PRDM1 and downregulation of BCL-6 in non-Hodgkin's B-cell lymphomas.Characterizing immunoglobulin repertoire from whole blood by a personal genome sequencer Sex- and subtype-specific analysis of H2AFX polymorphisms in non-Hodgkin lymphoma MicroRNAs are independent predictors of outcome in diffuse large B-cell lymphoma patients treated with R-CHOP.Transcriptional regulation via TF-modifying enzymes: an integrative model-based analysis.Connexin 43 communication channels in follicular dendritic cell development and in follicular lymphomas.T cell receptor-dependent activation of mTOR signaling in T cells is mediated by Carma1 and MALT1, but not Bcl10.The c-Rel Transcription Factor in Development and Disease.

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Lymphoid malignancies: the dark side of B-cell differentiation.

http://www.wikidata.org/entity/Q34162187

description

2002 nî lūn-bûn

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2002 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

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2002 թվականի դեկտեմբերին հրատարակված գիտական հոդված

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2002年の論文

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2002年学术文章

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2002年学术文章

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2002年学术文章

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2002年学术文章

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2002年学术文章

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2002年學術文章

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name

Lymphoid malignancies: the dark side of B-cell differentiation.

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Lymphoid malignancies: the dark side of B-cell differentiation.

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Lymphoid malignancies: the dark side of B-cell differentiation.

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type

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Lymphoid malignancies: the dark side of B-cell differentiation.

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Lymphoid malignancies: the dark side of B-cell differentiation.

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Lymphoid malignancies: the dark side of B-cell differentiation.

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prefLabel

Lymphoid malignancies: the dark side of B-cell differentiation.

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Lymphoid malignancies: the dark side of B-cell differentiation.

@en

Lymphoid malignancies: the dark side of B-cell differentiation.

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Nature Reviews Immunology

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Lymphoid malignancies: the dark side of B-cell differentiation.

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A L Shaffer

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Andreas Rosenwald

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Louis M Staudt

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P2860

A novel gene, MALT1 at 18q21, is involved in t(11;18) (q21;q21) found in low-grade B-cell lymphoma of mucosa-associated lymphoid tissue

Commitment to the B-lymphoid lineage depends on the transcription factor Pax5

Bcl10 is a positive regulator of antigen receptor-induced activation of NF-kappaB and neural tube closure

Plasma cell differentiation requires the transcription factor XBP-1

Ongoing immunoglobulin somatic mutation in germinal center B cell-like but not in activated B cell-like diffuse large cell lymphomas

Relation of gene expression phenotype to immunoglobulin mutation genotype in B cell chronic lymphocytic leukemia

Frequent occurrence of deletions and duplications during somatic hypermutation: implications for oncogene translocations and heavy chain disease

The use of molecular profiling to predict survival after chemotherapy for diffuse large-B-cell lymphoma.

Distinct types of diffuse large B-cell lymphoma identified by gene expression profiling

Commitment of B lymphocytes to a plasma cell fate is associated with Blimp-1 expression in vivo

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