ER stress negatively modulates the expression of the miR-199a/214 cluster to regulates tumor survival and progression in human hepatocellular cancer.
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Micro(RNA)managing endoplasmic reticulum stressOxymiRs in cutaneous development, wound repair and regenerationMicroRNA-214 Is Upregulated in Heart Failure Patients and Suppresses XBP1-Mediated Endothelial Cells AngiogenesisEndoplasmic reticulum stress activates the hepatic activator protein 1 complex via mitogen activated protein kinase-dependent signaling pathwaysMiR-214 promotes the alcohol-induced oxidative stress via down-regulation of glutathione reductase and cytochrome P450 oxidoreductase in liver cellsEvolution of the miR199-214 cluster and vertebrate skeletal developmentEndoplasmic reticulum stress plays a pivotal role in cell death mediated by the pan-deacetylase inhibitor panobinostat in human hepatocellular cancer cells.Mutual Regulation of MiR-199a-5p and HIF-1α Modulates the Warburg Effect in Hepatocellular Carcinoma.UPR-inducible miRNAs contribute to stressful situationsNeuroprotective effect of ischemic preconditioning in focal cerebral infarction: relationship with upregulation of vascular endothelial growth factorCardio-miRNAs and onco-miRNAs: circulating miRNA-based diagnostics for non-cancerous and cancerous diseasesMiR-214 targets β-catenin pathway to suppress invasion, stem-like traits and recurrence of human hepatocellular carcinoma.Disruption of microRNA biogenesis confers resistance to ER stress-induced cell death upstream of the mitochondrion.A systems biology approach identifies a regulatory network in parotid acinar cell terminal differentiation.MicroRNA-214 Promotes Apoptosis in Canine Hemangiosarcoma by Targeting the COP1-p53 Axis.RNA regulators of host immunity and pathogen adaptive responses in the oral cavityCell intrinsic and extrinsic activators of the unfolded protein response in cancer: Mechanisms and targets for therapyPerk-dependent repression of miR-106b-25 cluster is required for ER stress-induced apoptosisTranscriptional regulation of microRNA-100, -146a, and -150 genes by p53 and NFκB p65/RelA in mouse striatal STHdh(Q7)/ Hdh(Q7) cells and human cervical carcinoma HeLa cells.Unveiling the principle of microRNA-mediated redundancy in cellular pathway regulationMicroRNA-125b functions as a tumor suppressor in hepatocellular carcinoma cells.MicroRNA regulation of unfolded protein response transcription factor XBP1 in the progression of cardiac hypertrophy and heart failure in vivo.Human liver stem cell-derived microvesicles inhibit hepatoma growth in SCID mice by delivering antitumor microRNAs.PERK regulated miR-424(322)-503 cluster fine-tunes activation of IRE1 and ATF6 during Unfolded Protein Response.Overexpression of miR-214-3p in esophageal squamous cancer cells enhances sensitivity to cisplatin by targeting survivin directly and indirectly through CUG-BP1.A microRNA-mediated decrease in eukaryotic initiation factor 2α promotes cell survival during PS-341 treatment.miR-214/199a/199a* cluster levels predict poor survival in hepatocellular carcinoma through interference with cell-cycle regulators.Reduction of the ST6 β-galactosamide α-2,6-sialyltransferase 1 (ST6GAL1)-catalyzed sialylation of nectin-like molecule 2/cell adhesion molecule 1 and enhancement of ErbB2/ErbB3 signaling by microRNA-199a.Tumor-suppressing roles of miR-214 and miR-218 in breast cancer.Triptolide induces the expression of miR-142-3p: a negative regulator of heat shock protein 70 and pancreatic cancer cell proliferationTranscriptional suppression of CYP2A13 expression by lipopolysaccharide in cultured human lung cells and the lungs of a CYP2A13-humanized mouse model.MiRNAs which target CD3 subunits could be potential biomarkers for cancers.Regulation of the transcriptome by ER stress: non-canonical mechanisms and physiological consequences.Myocyte-specific enhancer factor 2C: a novel target gene of miR-214-3p in suppressing angiotensin II-induced cardiomyocyte hypertrophyCYP2J2-derived epoxyeicosatrienoic acids suppress endoplasmic reticulum stress in heart failure.Regulation of the unfolded protein response by microRNAs.Overexpression of endothelin 1 triggers hepatocarcinogenesis in zebrafish and promotes cell proliferation and migration through the AKT pathway.Knockdown of miR-214 promotes apoptosis and inhibits cell proliferation in nasopharyngeal carcinomaIntricately Regulated: A Cellular Toolbox for Fine-Tuning XBP1 Expression and Activity.Targeting EZH1 and EZH2 contributes to the suppression of fibrosis-associated genes by miR-214-3p in cardiac myofibroblasts.
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P2860
ER stress negatively modulates the expression of the miR-199a/214 cluster to regulates tumor survival and progression in human hepatocellular cancer.
description
2012 nî lūn-bûn
@nan
2012 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2012 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
name
ER stress negatively modulates ...... n human hepatocellular cancer.
@ast
ER stress negatively modulates ...... n human hepatocellular cancer.
@en
ER stress negatively modulates ...... n human hepatocellular cancer.
@nl
type
label
ER stress negatively modulates ...... n human hepatocellular cancer.
@ast
ER stress negatively modulates ...... n human hepatocellular cancer.
@en
ER stress negatively modulates ...... n human hepatocellular cancer.
@nl
prefLabel
ER stress negatively modulates ...... n human hepatocellular cancer.
@ast
ER stress negatively modulates ...... n human hepatocellular cancer.
@en
ER stress negatively modulates ...... n human hepatocellular cancer.
@nl
P2093
P2860
P1433
P1476
ER stress negatively modulates ...... n human hepatocellular cancer.
@en
P2093
Dao Wen Wang
Fuqiong Chen
Peihua Wang
Quanlu Duan
Xingxing He
Xingxu Wang
P2860
P304
P356
10.1371/JOURNAL.PONE.0031518
P407
P577
2012-02-16T00:00:00Z