Atopic asthmatic subjects but not atopic subjects without asthma have enhanced inflammatory response to ozone. - Wikidata - awgldk - TriplyDB

Atopic asthmatic subjects but not atopic subjects without asthma have enhanced inflammatory response to ozone.

Atopic asthmatic subjects but not atopic subjects without asthma have enhanced inflammatory response to ozone.

http://www.wikidata.org/entity/Q34171493

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Genes of innate immunity and the biological response to inhaled ozone Addressing human variability in next-generation human health risk assessments of environmental chemicals The effect of environmental oxidative stress on airway inflammation Exposure to ozone modulates human airway protease/antiprotease balance contributing to increased influenza A infection Childhood exposure to ambient polycyclic aromatic hydrocarbons is linked to epigenetic modifications and impaired systemic immunity in T cells Evaluating potential response-modifying factors for associations between ozone and health outcomes: a weight-of-evidence approach.IL-1 receptor antagonist reduces endotoxin-induced airway inflammation in healthy volunteers.Peripheral blood neutrophilia as a biomarker of ozone-induced pulmonary inflammation.Nrf2 expression modifies influenza A entry and replication in nasal epithelial cells.Effects of air pollutants on innate immunity: the role of Toll-like receptors and nucleotide-binding oligomerization domain-like receptors.Interaction with epithelial cells modifies airway macrophage response to ozone Airway purinergic responses in healthy, atopic nonasthmatic, and atopic asthmatic subjects exposed to ozone.Interaction effects of temperature and ozone on lung function and markers of systemic inflammation, coagulation, and fibrinolysis: a crossover study of healthy young volunteers.The IL-1 axis is associated with airway inflammation after O3 exposure in allergic asthmatic patients.Effects of Exposure to Ozone on the Ocular Surface in an Experimental Model of Allergic Conjunctivitis.The glutathione-S-transferase mu 1 (GSTM1) null genotype and increased neutrophil response to low-level ozone (0.06 ppm).Atopic asthmatic patients have reduced airway inflammatory cell recruitment after inhaled endotoxin challenge compared with healthy volunteers.Hyaluronan mediates ozone-induced airway hyperresponsiveness in mice.Outdoor PM2.5, Ambient Air Temperature, and Asthma Symptoms in the Past 14 Days among Adults with Active Asthma.The role of oxidative stress and innate immunity in O(3) and endotoxin-induced human allergic airway disease.Mechanisms and treatments for severe, steroid-resistant allergic airway disease and asthma.Airway cells from atopic asthmatic patients exposed to ozone display an enhanced innate immune gene profile.Neutrophilic inflammation in asthma: mechanisms and therapeutic considerations.Increased antioxidant activity after exposure of ozone in murine asthma model.Ozone primes alveolar macrophage-derived innate immunity in healthy human subjects.Patients with asthma demonstrate airway inflammation after exposure to concentrated ambient particulate matter.Baseline airway inflammation may be a determinant of the response to ozone exposure in asthmatic patients.Ozone-induced eosinophil recruitment to airways is altered by antigen sensitization and tumor necrosis factor-α blockade.Newly divided eosinophils limit ozone-induced airway hyperreactivity in nonsensitized guinea pigs.Limonene and its ozone-initiated reaction products attenuate allergic lung inflammation in mice.The role of non-invasive biomarkers in detecting acute respiratory effects of traffic-related air pollution.Residential ozone and lung function in the elderly

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Atopic asthmatic subjects but not atopic subjects without asthma have enhanced inflammatory response to ozone.

http://www.wikidata.org/entity/Q34171493

description

2010 nî lūn-bûn

@nan

2010 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի սեպտեմբերին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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Atopic asthmatic subjects but ...... nflammatory response to ozone.

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Atopic asthmatic subjects but ...... nflammatory response to ozone.

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J.JACI.2010.06.043

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The Journal of Allergy and Clinical Immunology

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Atopic asthmatic subjects but ...... nflammatory response to ozone.

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Bradford Harris

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Charles R Esther

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David B Peden

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David Diaz-Sanchez

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John C Lay

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P2860

Antagonism of microRNA-126 suppresses the effector function of TH2 cells and the development of allergic airways disease

IL-10 inhibits cytokine production by activated macrophages

Association of ambient ozone exposure with airway inflammation and allergy in adults with asthma.

Signalling through the high-affinity IgE receptor Fc epsilonRI.

TLR4 is necessary for hyaluronan-mediated airway hyperresponsiveness after ozone inhalation.

Analyses of asthma severity phenotypes and inflammatory proteins in subjects stratified by sputum granulocytes

Regulation of lung injury and repair by Toll-like receptors and hyaluronan.

The glutathione-S-transferase Mu 1 null genotype modulates ozone-induced airway inflammation in human subjects.

In vivo uptake of inhaled particles by airway phagocytes is enhanced in patients with mild asthma compared with normal volunteers.

Signal transduction pathways of tumor necrosis factor--mediated lung injury induced by ozone in mice.

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