Targeting Epstein-Barr virus oncoprotein LMP1-mediated glycolysis sensitizes nasopharyngeal carcinoma to radiation therapy. - Wikidata - awgldk - TriplyDB

Targeting Epstein-Barr virus oncoprotein LMP1-mediated glycolysis sensitizes nasopharyngeal carcinoma to radiation therapy.

Targeting Epstein-Barr virus oncoprotein LMP1-mediated glycolysis sensitizes nasopharyngeal carcinoma to radiation therapy.

http://www.wikidata.org/entity/Q34171822

about

Stealing the Keys to the Kitchen: Viral Manipulation of the Host Cell Metabolic Network Biomarkers for enhancing the radiosensitivity of nasopharyngeal carcinoma Deguelin inhibits non-small cell lung cancer via down-regulating Hexokinases II-mediated glycolysis.Suppression Of β-catenin Nuclear Translocation By CGP57380 Decelerates Poor Progression And Potentiates Radiation-Induced Apoptosis in Nasopharyngeal Carcinoma.Dengue virus induces and requires glycolysis for optimal replication Alpha-enolase promotes cell glycolysis, growth, migration, and invasion in non-small cell lung cancer through FAK-mediated PI3K/AKT pathway Viral activation of cellular metabolism Epstein-Barr Virus MicroRNA Expression Increases Aggressiveness of Solid Malignancies.Gold nano-particles (AuNPs) carrying anti-EBV-miR-BART7-3p inhibit growth of EBV-positive nasopharyngeal carcinoma.Enolase-1 is a therapeutic target in endometrial carcinoma.Repression of Hox genes by LMP1 in nasopharyngeal carcinoma and modulation of glycolytic pathway genes by HoxC8.Discriminating between Terminal- and Non-Terminal Respiratory Unit-Type Lung Adenocarcinoma Based on MicroRNA Profiles.Metabolic stress is a barrier to Epstein-Barr virus-mediated B-cell immortalization.The putative tumor activator ARHGEF3 promotes nasopharyngeal carcinoma cell pathogenesis by inhibiting cellular apoptosis The role of Epstein-Barr virus infection in the pathogenesis of nasopharyngeal carcinoma.Novel roles and therapeutic targets of Epstein-Barr virus-encoded latent membrane protein 1-induced oncogenesis in nasopharyngeal carcinoma.Metabolic reprogramming: a hallmark of viral oncogenesis.Transcriptomic profiling of a chicken lung epithelial cell line (CLEC213) reveals a mitochondrial respiratory chain activity boost during influenza virus infection.Epstein-Barr Virus-Encoded Latent Membrane Protein 1 Upregulates Glucose Transporter 1 Transcription via the mTORC1/NF-κB Signaling Pathways.Pyruvate dehydrogenase B promoted the growth and migration of the nasopharyngeal carcinoma cells.Lactate dehydrogenase inhibition: exploring possible applications beyond cancer treatment.LMP1 Increases Expression of NADPH Oxidase (NOX) and Its Regulatory Subunit p22 in NP69 Nasopharyngeal Cells and Makes Them Sensitive to a Treatment by a NOX Inhibitor.Activation of the FGFR1 signalling pathway by the Epstein-Barr virus-encoded LMP1 promotes aerobic glycolysis and transformation of human nasopharyngeal epithelial cells.Activation of cellular metabolism during latent Kaposi's Sarcoma herpesvirus infection.LMP1 promotes expression of insulin-like growth factor 1 (IGF1) to selectively activate IGF1 receptor and drive cell proliferation.Manipulation of the host cell membrane by human γ-herpesviruses EBV and KSHV for pathogenesis.The role of metabolic reprogramming in γ-herpesvirus-associated oncogenesis.The role of oxidative stress in EBV lytic reactivation, radioresistance and the potential preventive and therapeutic implications.Epstein-Barr Virus LMP1 Mediated Oncogenicity.LMP1-mediated glycolysis induces myeloid-derived suppressor cell expansion in nasopharyngeal carcinoma.Diisopropylamine dichloroacetate enhances radiosensitization in esophageal squamous cell carcinoma by increasing mitochondria-derived reactive oxygen species levels.Viral E6/E7 oncogene and cellular hexokinase 2 expression in HPV-positive cancer cell lines.Modulation of the Tumor Microenvironment by Epstein-Barr virus Latent Membrane Protein-1 in Nasopharyngeal Carcinoma.EBV Infection and Glucose Metabolism in Nasopharyngeal Carcinoma.(-)-Epigallocatechin-3-gallate inhibition of Epstein-Barr virus spontaneous lytic infection involves downregulation of latent membrane protein 1.IDH2 is a novel diagnostic and prognostic serum biomarker for non-small-cell lung cancer.Lactic Acid Downregulates Viral MicroRNA To Promote Epstein-Barr Virus-Immortalized B Lymphoblastic Cell Adhesion and Growth.Upregulation of Glucose Uptake and Hexokinase Activity of Primary Human CD4+ T Cells in Response to Infection with HIV-1.Chibby suppresses aerobic glycolysis and proliferation of nasopharyngeal carcinoma via the Wnt/β-catenin-Lin28/let7-PDK1 cascade.EBV based cancer prevention and therapy in nasopharyngeal carcinoma.

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Targeting Epstein-Barr virus oncoprotein LMP1-mediated glycolysis sensitizes nasopharyngeal carcinoma to radiation therapy.

http://www.wikidata.org/entity/Q34171822

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2014 nî lūn-bûn

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2014 թվականի մարտին հրատարակված գիտական հոդված

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P2860

Hallmarks of Cancer: The Next Generation

Epstein-Barr virus immortalization of human B-cells leads to stabilization of hypoxia-induced factor 1 alpha, congruent with the Warburg effect

Oncometabolite 2-hydroxyglutarate is a competitive inhibitor of α-ketoglutarate-dependent dioxygenases

Hypoxia-inducible factor 1 and dysregulated c-Myc cooperatively induce vascular endothelial growth factor and metabolic switches hexokinase 2 and pyruvate dehydrogenase kinase 1

Temporal Proteome and Lipidome Profiles Reveal Hepatitis C Virus-Associated Reprogramming of Hepatocellular Metabolism and Bioenergetics

Dysregulation of glucose transport, glycolysis, TCA cycle and glutaminolysis by oncogenes and tumor suppressors in cancer cells

TNF-alpha inhibits asbestos-induced cytotoxicity via a NF-kappaB-dependent pathway, a possible mechanism for asbestos-induced oncogenesis

Divergent effects of human cytomegalovirus and herpes simplex virus-1 on cellular metabolism

Hexokinase-mitochondria interaction mediated by Akt is required to inhibit apoptosis in the presence or absence of Bax and Bak

Exome sequencing of head and neck squamous cell carcinoma reveals inactivating mutations in NOTCH1

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radiation therapy

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