Influence of genetic background on knockout mouse phenotypes.
about
Insufficiently defined genetic background confounds phenotypes in transgenic studies as exemplified by malaria infection in Tlr9 knockout miceDefective extraembryonic angiogenesis in mice lacking LBP-1a, a member of the grainyhead family of transcription factorsStrain-specific modifier genes governing craniofacial phenotypes.An allelic series of mice reveals a role for RERE in the development of multiple organs affected in chromosome 1p36 deletions.The membrane-anchored serine protease prostasin (CAP1/PRSS8) supports epidermal development and postnatal homeostasis independent of its enzymatic activity.Distribution and Severity of Neuropathology in β-Mannosidase-Deficient Mice is Strain Dependent.Secretoglobin superfamily protein SCGB3A2 deficiency potentiates ovalbumin-induced allergic pulmonary inflammation.Reduced pachytene piRNAs and translation underlie spermiogenic arrest in Maelstrom mutant miceNatural genetic variation determines susceptibility to aggregation or toxicity in a C. elegans model for polyglutamine disease.Of mice and models: improved animal models for biomedical research.The relevance of individual genetic background and its role in animal models of epilepsyTransforming growth factor beta signaling in adult cardiovascular diseases and repair.Prolonged Cre expression driven by the α-myosin heavy chain promoter can be cardiotoxic.IgA is important for clearance and critical for protection from rotavirus infection.Roles of uroplakins in plaque formation, umbrella cell enlargement, and urinary tract diseases.Differential regulation of HMG-CoA reductase and Insig-1 by enzymes of the ubiquitin-proteasome system.Influence of genetic background on genetically engineered mouse phenotypes.Genetic Background Limits Generalizability of Genotype-Phenotype Relationships.MicroRNA miR-301a is a novel cardiac regulator of Cofilin-2.Elimination of both CD4+ and CD8+ T cells but not B cells eliminates inflammation and prolongs the survival of TGFbeta1-deficient mice.Congenic D1A dopamine receptor mutants: ethologically based resolution of behavioural topography indicates genetic background as a determinant of knockout phenotype.D1-like dopamine receptor-mediated function in congenic mutants with D1 vs. D5 receptor "knockout".Rodent models in Down syndrome research: impact and future opportunities.14-3-3ζ loss leads to neonatal lethality by microRNA-126 downregulation-mediated developmental defects in lung vasculature.Osteopontin regulates dentin and alveolar bone development and mineralization.Gene expression analysis to identify mechanisms underlying heart failure susceptibility in mice and humans.
P2860
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P2860
Influence of genetic background on knockout mouse phenotypes.
description
2001 nî lūn-bûn
@nan
2001 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2001 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2001年の論文
@ja
2001年論文
@yue
2001年論文
@zh-hant
2001年論文
@zh-hk
2001年論文
@zh-mo
2001年論文
@zh-tw
2001年论文
@wuu
name
Influence of genetic background on knockout mouse phenotypes.
@ast
Influence of genetic background on knockout mouse phenotypes.
@en
Influence of genetic background on knockout mouse phenotypes.
@nl
type
label
Influence of genetic background on knockout mouse phenotypes.
@ast
Influence of genetic background on knockout mouse phenotypes.
@en
Influence of genetic background on knockout mouse phenotypes.
@nl
prefLabel
Influence of genetic background on knockout mouse phenotypes.
@ast
Influence of genetic background on knockout mouse phenotypes.
@en
Influence of genetic background on knockout mouse phenotypes.
@nl
P2093
P1476
Influence of genetic background on knockout mouse phenotypes.
@en
P2093
Doetschman T
Kallapur S
Sanford LP
P304
P356
10.1385/1-59259-220-1:217
P407
P577
2001-01-01T00:00:00Z