Contrasting patterns of Bim induction and neuroprotection in Bim-deficient mice between hippocampus and neocortex after status epilepticus.
about
Identification of a novel Bcl-2-interacting mediator of cell death (Bim) E3 ligase, tripartite motif-containing protein 2 (TRIM2), and its role in rapid ischemic tolerance-induced neuroprotectionMitochondrial localization of the forkhead box class O transcription factor FOXO3a in brainBAD-dependent regulation of fuel metabolism and K(ATP) channel activity confers resistance to epileptic seizures.Silencing microRNA-134 produces neuroprotective and prolonged seizure-suppressive effectsBmf upregulation through the AMP-activated protein kinase pathway may protect the brain from seizure-induced cell death.Deletion of Puma protects hippocampal neurons in a model of severe status epilepticus.Transgenic overexpression of 14-3-3 zeta protects hippocampus against endoplasmic reticulum stress and status epilepticus in vivo.Suppression of TNF receptor-1 signaling in an in vitro model of epileptic tolerance.Cell signaling underlying epileptic behavior.miRNA Expression profile after status epilepticus and hippocampal neuroprotection by targeting miR-132Contribution of apoptosis-associated signaling pathways to epileptogenesis: lessons from Bcl-2 family knockouts.In vivo contributions of BH3-only proteins to neuronal death following seizures, ischemia, and traumatic brain injury.A Molecular Approach to Epilepsy Management: from Current Therapeutic Methods to Preconditioning Efforts.De-repression of myelin-regulating gene expression after status epilepticus in mice lacking the C/EBP homologous protein CHOPChopping Out CHOP Chops the Fate of Neurons.Deletion of the BH3-only protein Noxa alters electrographic seizures but does not protect against hippocampal damage after status epilepticus in miceLoss of p53 results in protracted electrographic seizures and development of an aggravated epileptic phenotype following status epilepticus.Differential expression patterns of Puma and Hsp70 following proteasomal stress in the hippocampus are key determinants of neuronal vulnerability.BAD and KATP channels regulate neuron excitability and epileptiform activity.Mice Lacking Functional Fas Death Receptors Are Protected from Kainic Acid-Induced Apoptosis in the Hippocampus.Bcl-2 homology domain 3-only proteins Puma and Bim mediate the vulnerability of CA1 hippocampal neurons to proteasome inhibition in vivo.
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Contrasting patterns of Bim induction and neuroprotection in Bim-deficient mice between hippocampus and neocortex after status epilepticus.
description
2009 nî lūn-bûn
@nan
2009 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
name
Contrasting patterns of Bim in ...... rtex after status epilepticus.
@ast
Contrasting patterns of Bim in ...... rtex after status epilepticus.
@en
Contrasting patterns of Bim in ...... rtex after status epilepticus.
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type
label
Contrasting patterns of Bim in ...... rtex after status epilepticus.
@ast
Contrasting patterns of Bim in ...... rtex after status epilepticus.
@en
Contrasting patterns of Bim in ...... rtex after status epilepticus.
@nl
prefLabel
Contrasting patterns of Bim in ...... rtex after status epilepticus.
@ast
Contrasting patterns of Bim in ...... rtex after status epilepticus.
@en
Contrasting patterns of Bim in ...... rtex after status epilepticus.
@nl
P2093
P2860
P50
P356
P1476
Contrasting patterns of Bim in ...... ortex after status epilepticus
@en
P2093
H P Bonner
S Hatazaki
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P2888
P304
P356
10.1038/CDD.2009.134
P50
P577
2009-09-25T00:00:00Z