Evaluation of human leukocyte N-formylpeptide receptor (FPR1) SNPs in aggressive periodontitis patients.
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International Union of Basic and Clinical Pharmacology. LXXIII. Nomenclature for the formyl peptide receptor (FPR) familyThe role of water in activation mechanism of human N-formyl peptide receptor 1 (FPR1) based on molecular dynamics simulationsImpaired phagocytosis in localized aggressive periodontitis: rescue by Resolvin E1Regulation of human formyl peptide receptor 1 synthesis: role of single nucleotide polymorphisms, transcription factors, and inflammatory mediatorsEnamel matrix derivative promotes superoxide production and chemotaxis but reduces matrix metalloproteinase-8 expression by polymorphonuclear leukocytes.Informational paper: implications of genetic technology for the management of periodontal diseases.Peptides derived from HIV-1, HIV-2, Ebola virus, SARS coronavirus and coronavirus 229E exhibit high affinity binding to the formyl peptide receptor.Effect of genetic variability on the inflammatory response to periodontal infection.Identification of genetic risk factors for periodontitis and possible mechanisms of action.The genetic basis of periodontitis.Gene polymorphisms in periodontal health and disease.The role of genetic polymorphisms in periodontitis.Determinants of host susceptibility in aggressive periodontitis.Variable responses of formyl peptide receptor haplotypes toward bacterial peptides.V101L of human formyl peptide receptor 1 (FPR1) increases receptor affinity and augments the antagonism mediated by cyclosporins.Formylpeptide receptor single nucleotide polymorphism 348T>C and its relationship to polymorphonuclear leukocyte chemotaxis in aggressive periodontitis.Role of genetic factors in the pathogenesis of aggressive periodontitis.Enemy attraction: bacterial agonists for leukocyte chemotaxis receptors.Formyl-peptide receptor 2 governs leukocyte influx in local Staphylococcus aureus infections.Honokiol suppresses formyl peptide-induced human neutrophil activation by blocking formyl peptide receptor 1.Assessment of chromosome 19 for genetic association in severe chronic periodontitis.Analysis of a missense variant of the human N-formyl peptide receptor that is associated with agonist-independent beta-arrestin association and indices of inflammation.Normal cell surface expression and selective loss of functions resulting from Phe110 to Ser and Cys126 to Trp substitutions in the formyl peptide receptor.Differential activation of polymorphisms of the formyl peptide receptor by formyl peptides.Human formyl peptide receptor 1 (FPR1) c.32C>T SNP is associated with decreased soluble E-selectin levels.Genetic polymorphisms and periodontal disease in populations of African descent: A review.Clinical and Genomic Correlates of Neutrophil Reactive Oxygen Species Production in Pediatric Patients with Crohn's Disease.Evaluation of RANK/RANKL/OPG gene polymorphisms in aggressive periodontitis.
P2860
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P2860
Evaluation of human leukocyte N-formylpeptide receptor (FPR1) SNPs in aggressive periodontitis patients.
description
2003 nî lūn-bûn
@nan
2003 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2003 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
name
Evaluation of human leukocyte ...... essive periodontitis patients.
@ast
Evaluation of human leukocyte ...... essive periodontitis patients.
@en
Evaluation of human leukocyte N-formylpeptide receptor
@nl
type
label
Evaluation of human leukocyte ...... essive periodontitis patients.
@ast
Evaluation of human leukocyte ...... essive periodontitis patients.
@en
Evaluation of human leukocyte N-formylpeptide receptor
@nl
prefLabel
Evaluation of human leukocyte ...... essive periodontitis patients.
@ast
Evaluation of human leukocyte ...... essive periodontitis patients.
@en
Evaluation of human leukocyte N-formylpeptide receptor
@nl
P2093
P356
P1433
P1476
Evaluation of human leukocyte ...... essive periodontitis patients.
@en
P2093
Cortelli JR
Feingold E
VanDyke TE
P2888
P356
10.1038/SJ.GENE.6363900
P577
2003-01-01T00:00:00Z
P5875
P6179
1050885211