Chronic intermittent hypoxia induces NMDA receptor-dependent plasticity and suppresses nitric oxide signaling in the mouse hypothalamic paraventricular nucleus.
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Peripheral chemoreception and arterial pressure responses to intermittent hypoxiaNeural Control of Blood Pressure in Chronic Intermittent HypoxiaSex differences in NMDA GluN1 plasticity in rostral ventrolateral medulla neurons containing corticotropin-releasing factor type 1 receptor following slow-pressor angiotensin II hypertension.Female protection from slow-pressor effects of angiotensin II involves prevention of ROS production independent of NMDA receptor trafficking in hypothalamic neurons expressing angiotensin 1A receptors.Slow-pressor angiotensin II hypertension and concomitant dendritic NMDA receptor trafficking in estrogen receptor β-containing neurons of the mouse hypothalamic paraventricular nucleus are sex and age dependentEndothelin 1-dependent neurovascular dysfunction in chronic intermittent hypoxia.Visualization of oxytocin release that mediates paired pulse facilitation in hypothalamic pathways to brainstem autonomic neurons.Simulating obstructive sleep apnea patients' oxygenation characteristics into a mouse model of cyclical intermittent hypoxia.Dichotomous effects of chronic intermittent hypoxia on focal cerebral ischemic injuryThe lighter side of BDNF.NMDA Receptor Plasticity in the Hypothalamic Paraventricular Nucleus Contributes to the Elevated Blood Pressure Produced by Angiotensin II.Sympatho-adrenal activation by chronic intermittent hypoxia.Cerebrovascular consequences of obstructive sleep apneaNeurogenic mechanisms underlying the rapid onset of sympathetic responses to intermittent hypoxia.Membrane trafficking of NADPH oxidase p47(phox) in paraventricular hypothalamic neurons parallels local free radical production in angiotensin II slow-pressor hypertensionMolecular mechanisms of chronic intermittent hypoxia and hypertensionPost ischemia intermittent hypoxia induces hippocampal neurogenesis and synaptic alterations and alleviates long-term memory impairment.Activation of spinal MrgC-Gi-NR2B-nNOS signaling pathway by Mas oncogene-related gene C receptor agonist bovine adrenal medulla 8-22 attenuates bone cancer pain in mice.Angiotensin II slow-pressor hypertension enhances NMDA currents and NOX2-dependent superoxide production in hypothalamic paraventricular neurons.Oxytocin neuron activation prevents hypertension that occurs with chronic intermittent hypoxia/hypercapnia in rats.Rats selectively bred for differences in aerobic capacity have similar hypertensive responses to chronic intermittent hypoxia.Neonatal Maternal Separation Augments Carotid Body Response to Hypoxia in Adult Males but Not Female Rats.Chronic intermittent hypoxia increases sympathetic control of blood pressure: role of neuronal activity in the hypothalamic paraventricular nucleus.Treatment of intermittent hypoxia increases phosphorylated tau in the hippocampus via biological processes common to agingRedistribution of NMDA Receptors in Estrogen-Receptor-β-Containing Paraventricular Hypothalamic Neurons following Slow-Pressor Angiotensin II Hypertension in Female Mice with Accelerated Ovarian Failure.Altered hypothalamic function in diet-induced obesity.Coupling between respiratory and sympathetic activities as a novel mechanism underpinning neurogenic hypertension.Attention deficit/hyperactivity disorder after neonatal surgery: review of the pathophysiology and risk factors.Alterations in the subcellular distribution of NADPH oxidase p47(phox) in hypothalamic paraventricular neurons following slow-pressor angiotensin II hypertension in female mice with accelerated ovarian failure.Parasympathetic Vagal Control of Cardiac Function.Role of angiotensin-converting enzyme 1 within the median preoptic nucleus following chronic intermittent hypoxia.Heme oxygenase-1-dependent central cardiorespiratory adaptations to chronic intermittent hypoxia in mice.Chronic intermittent hypoxia alters glutamatergic control of sympathetic and respiratory activities in the commissural NTS of rats.Apoε4 disrupts neurovascular regulation and undermines white matter integrity and cognitive function
P2860
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P2860
Chronic intermittent hypoxia induces NMDA receptor-dependent plasticity and suppresses nitric oxide signaling in the mouse hypothalamic paraventricular nucleus.
description
2010 nî lūn-bûn
@nan
2010 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Chronic intermittent hypoxia i ...... lamic paraventricular nucleus.
@ast
Chronic intermittent hypoxia i ...... lamic paraventricular nucleus.
@en
Chronic intermittent hypoxia i ...... lamic paraventricular nucleus.
@nl
type
label
Chronic intermittent hypoxia i ...... lamic paraventricular nucleus.
@ast
Chronic intermittent hypoxia i ...... lamic paraventricular nucleus.
@en
Chronic intermittent hypoxia i ...... lamic paraventricular nucleus.
@nl
prefLabel
Chronic intermittent hypoxia i ...... lamic paraventricular nucleus.
@ast
Chronic intermittent hypoxia i ...... lamic paraventricular nucleus.
@en
Chronic intermittent hypoxia i ...... lamic paraventricular nucleus.
@nl
P2093
P2860
P1476
Chronic intermittent hypoxia i ...... lamic paraventricular nucleus.
@en
P2093
Christal G Coleman
George J Delagrammatikas
Laibaik Park
Virginia M Pickel
P2860
P304
12103-12112
P356
10.1523/JNEUROSCI.3367-10.2010
P407
P577
2010-09-01T00:00:00Z