Cutting edge: CD40-induced expression of recombination activating gene (RAG) 1 and RAG2: a mechanism for the generation of autoaggressive T cells in the periphery.
about
Defining a new biomarker for the autoimmune component of Multiple Sclerosis: Th40 cells.CD40 glycoforms and TNF-receptors 1 and 2 in the formation of CD40 receptor(s) in autoimmunityT-cell receptor revision: friend or foe?Pro-inflammatory T-lymphocytes rapidly infiltrate into the brain and contribute to neuronal injury following cardiac arrest and cardiopulmonary resuscitationModulation of TCRβ surface expression during TCR revision.A CD40-targeted peptide controls and reverses type 1 diabetes in NOD mice.Receptor revision in CD4 T cells is influenced by follicular helper T cell formation and germinal-center interactions.Cutting edge: TCR revision affects predominantly Foxp3 cells and skews them toward the Th17 lineage.TCR revision generates functional CD4+ T cells.Th40 cells (CD4+CD40+ Tcells) drive a more severe form of Experimental Autoimmune Encephalomyelitis than conventional CD4 T cells.High distribution of CD40 and TRAF2 in Th40 T cell rafts leads to preferential survival of this auto-aggressive population in autoimmunityThe dynamic lives of T cells: new approaches and themesDisruption of the homeostatic balance between autoaggressive (CD4+CD40+) and regulatory (CD4+CD25+FoxP3+) T cells promotes diabetes.An alternative role for Foxp3 as an effector T cell regulator controlled through CD40CD40 interacts directly with RAG1 and RAG2 in autoaggressive T cells and Fas prevents CD40-induced RAG expression.Of the multiple mechanisms leading to type 1 diabetes, T cell receptor revision may play a prominent role (is type 1 diabetes more than a single disease?).Overlooked Mechanisms in Type 1 Diabetes Etiology: How Unique Costimulatory Molecules Contribute to DiabetogenesisCD40-mediated signalling influences trafficking, T-cell receptor expression, and T-cell pathogenesis, in the NOD model of type 1 diabetes.The co-evolution of our understanding of CD40 and inflammation.CD40 engagement of CD4+ CD40+ T cells in a neo-self antigen disease model ablates CTLA-4 expression and indirectly impacts tolerance
P2860
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P2860
Cutting edge: CD40-induced expression of recombination activating gene (RAG) 1 and RAG2: a mechanism for the generation of autoaggressive T cells in the periphery.
description
2003 nî lūn-bûn
@nan
2003 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2003 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
2003年の論文
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2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
name
Cutting edge: CD40-induced exp ...... sive T cells in the periphery.
@ast
Cutting edge: CD40-induced exp ...... sive T cells in the periphery.
@en
Cutting edge: CD40-induced expression of recombination activating gene
@nl
type
label
Cutting edge: CD40-induced exp ...... sive T cells in the periphery.
@ast
Cutting edge: CD40-induced exp ...... sive T cells in the periphery.
@en
Cutting edge: CD40-induced expression of recombination activating gene
@nl
prefLabel
Cutting edge: CD40-induced exp ...... sive T cells in the periphery.
@ast
Cutting edge: CD40-induced exp ...... sive T cells in the periphery.
@en
Cutting edge: CD40-induced expression of recombination activating gene
@nl
P2093
P1476
Cutting edge: CD40-induced exp ...... sive T cells in the periphery.
@en
P2093
David H Wagner
Gisela M Vaitaitis
Kathryn Haskins
Michelle Poulin
Richard J Sanderson
P304
P356
10.4049/JIMMUNOL.170.7.3455
P407
P577
2003-04-01T00:00:00Z