Elevated glucose impairs endothelium-dependent relaxation by activating protein kinase C.
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The role of glucosamine-induced ER stress in diabetic atherogenesisVascular and neural mechanisms of ACh-mediated vasodilation in the forearm cutaneous microcirculationEthanol consumption increases blood pressure and alters the responsiveness of the mesenteric vasculature in ratsTwenty-five years since the discovery of endothelium-derived relaxing factor (EDRF): does a dysfunctional endothelium contribute to the development of type 2 diabetes?Reduced Na-K pump but increased Na-K-2Cl cotransporter in aorta of streptozotocin-induced diabetic rat.Glucose scavenging of nitric oxide.Vasodilator responses in the forearm skin of patients with insulin-dependent diabetes mellitus.Intravitreal vs. subtenon triamcinolone acetonide for the treatment of diabetic cystoid macular edema.Spinal cord stimulation in experimental chronic painful diabetic polyneuropathy: Delayed effect of High-frequency stimulation.Effect of glitazones on the progression of coronary artery disease in type 2 diabetes patients.Nitric oxide in the pathogenesis of vascular disease.Endothelial dysfunction in diabetes mellitus: molecular mechanisms and clinical implications.Endothelial dysfunction in diabetes.Molecular insights and therapeutic targets for diabetic endothelial dysfunction.Link between free radicals and protein kinase C in glucose-induced alteration of vascular dilation.Protein kinase C activation and its pharmacological inhibition in vascular disease.Role of vasoactive factors in the pathogenesis of early changes in diabetic retinopathy.Impaired nitric oxide-dependent cyclic guanosine monophosphate generation in glomeruli from diabetic rats. Evidence for protein kinase C-mediated suppression of the cholinergic response.Inositols prevent and reverse endothelial dysfunction in diabetic rat and rabbit vasculature metabolically and by scavenging superoxide.TP receptors and oxidative stress hand in hand from endothelial dysfunction to atherosclerosis.Free radicals, diabetes and endothelial dysfunction.The effect of hyperglycaemia on function of rat isolated mesenteric resistance artery.Effect of protein kinase C and phospholipase A2 inhibitors on the impaired ability of human diabetic platelets to cause vasodilation.Acetylcholine-induced arteriolar dilation is reduced in streptozotocin-induced diabetic rats with motor nerve dysfunctionIn vivo evaluation of ocular inflammatory responses in experimental diabetes.Increased atherothrombotic burden in patients with diabetes mellitus and acute coronary syndrome: a review of antiplatelet therapyEndothelium-dependent inhibition of Na(+)-K+ ATPase activity in rabbit aorta by hyperglycemia. Possible role of endothelium-derived nitric oxide.Endothelium-dependent relaxation and noradrenaline sensitivity in mesenteric resistance arteries of streptozotocin-induced diabetic ratsEffect of diabetes and elevated glucose on nitric oxide-mediated neurotransmission in rat anococcygeus muscle.Endothelial function in the isolated perfused mesentery and aortae of rats with streptozotocin-induced diabetes: effect of treatment with the aldose reductase inhibitor, ponalrestat.Endothelial dysfunction and diabetes: effects on angiogenesis, vascular remodeling, and wound healing.The effect of direct renin inhibition alone and in combination with ACE inhibition on endothelial function, arterial stiffness, and renal function in type 1 diabetes.Advanced glycation end products acutely impair ca(2+) signaling in bovine aortic endothelial cells.Vascular effects of intravenous intralipid and dextrose infusions in obese subjectsPrevalence and risk factors for prolonged QT interval and QT dispersion in patients with type 2 diabetesEndothelium-dependent contractions: when a good guy turns bad!Preferential elevation of protein kinase C isoform beta II and diacylglycerol levels in the aorta and heart of diabetic rats: differential reversibility to glycemic control by islet cell transplantation.Vascular incorporation of alpha-tocopherol prevents endothelial dysfunction due to oxidized LDL by inhibiting protein kinase C stimulation.Preeclampsia selectively impairs endothelium-dependent relaxation and leads to oscillatory activity in small omental arteries.Diabetes status differentiates endothelial function and plasma nitrite response to exercise stress in peripheral arterial disease following supervised training.
P2860
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P2860
Elevated glucose impairs endothelium-dependent relaxation by activating protein kinase C.
description
1991 nî lūn-bûn
@nan
1991 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
1991 թվականի մայիսին հրատարակված գիտական հոդված
@hy
1991年の論文
@ja
1991年論文
@yue
1991年論文
@zh-hant
1991年論文
@zh-hk
1991年論文
@zh-mo
1991年論文
@zh-tw
1991年论文
@wuu
name
Elevated glucose impairs endothelium-dependent relaxation by activating protein kinase C.
@ast
Elevated glucose impairs endothelium-dependent relaxation by activating protein kinase C.
@en
Elevated glucose impairs endothelium-dependent relaxation by activating protein kinase C.
@nl
type
label
Elevated glucose impairs endothelium-dependent relaxation by activating protein kinase C.
@ast
Elevated glucose impairs endothelium-dependent relaxation by activating protein kinase C.
@en
Elevated glucose impairs endothelium-dependent relaxation by activating protein kinase C.
@nl
prefLabel
Elevated glucose impairs endothelium-dependent relaxation by activating protein kinase C.
@ast
Elevated glucose impairs endothelium-dependent relaxation by activating protein kinase C.
@en
Elevated glucose impairs endothelium-dependent relaxation by activating protein kinase C.
@nl
P2093
P2860
P356
P1476
Elevated glucose impairs endothelium-dependent relaxation by activating protein kinase C.
@en
P2093
B Tesfamariam
P2860
P304
P356
10.1172/JCI115179
P407
P577
1991-05-01T00:00:00Z