Rationale and design of the LURIC study--a resource for functional genomics, pharmacogenomics and long-term prognosis of cardiovascular disease.
about
The associations of cholesterol metabolism and plasma plant sterols with all-cause and cardiovascular mortalityGenome sequencing elucidates Sardinian genetic architecture and augments association analyses for lipid and blood inflammatory markersLarge-scale association analysis identifies 13 new susceptibility loci for coronary artery disease.Differential network analysis with multiply imputed lipidomic dataLDL-Cholesterol: Standards of Treatment 2016: A German Perspective.Spouses and unrelated friends of probands as controls for stroke genetics studiesStatistical analysis of big data on pharmacogenomicsIs the adiposity-associated FTO gene variant related to all-cause mortality independent of adiposity? Meta-analysis of data from 169,551 Caucasian adultsData on gender and subgroup specific analyses of omega-3 fatty acids in the Ludwigshafen Risk and Cardiovascular Health Study.Genetic variants of the promoter of the heme oxygenase-1 gene and their influence on cardiovascular disease (the Ludwigshafen Risk and Cardiovascular Health study).Association of myeloperoxidase with total and cardiovascular mortality in individuals undergoing coronary angiography--the LURIC study.Effect of the rs2259816 polymorphism in the HNF1A gene on circulating levels of c-reactive protein and coronary artery disease (the ludwigshafen risk and cardiovascular health study).Refining Long-Term Prediction of Cardiovascular Risk in Diabetes - The VILDIA Score.Investigation of 95 variants identified in a genome-wide study for association with mortality after acute coronary syndromeLack of an association between connexin-37, stromelysin-1, plasminogen activator-inhibitor type 1 and lymphotoxin-alpha genes and acute coronary syndrome in Czech CaucasiansCholesteryl ester transfer protein and mortality in patients undergoing coronary angiography: the Ludwigshafen Risk and Cardiovascular Health study.EPIQ-efficient detection of SNP-SNP epistatic interactions for quantitative traitsSerum neopterin is not increased in obese juveniles.A variant in the ABO gene explains the variation in soluble E-selectin levels-results from dense genotyping in two independent populations.Design of the Coronary ARtery DIsease Genome-Wide Replication And Meta-Analysis (CARDIoGRAM) Study: A Genome-wide association meta-analysis involving more than 22 000 cases and 60 000 controls.Glycated hemoglobin predicts all-cause, cardiovascular, and cancer mortality in people without a history of diabetes undergoing coronary angiography.Low free testosterone levels are associated with all-cause and cardiovascular mortality in postmenopausal diabetic women.Additional use of glycated hemoglobin for diagnosis of type 2 diabetes in people undergoing coronary angiography reveals a subgroup at increased cardiovascular riskVitamin D levels predict all-cause and cardiovascular disease mortality in subjects with the metabolic syndrome: the Ludwigshafen Risk and Cardiovascular Health (LURIC) Study.PCSK9 Plasma Concentrations Are Independent of GFR and Do Not Predict Cardiovascular Events in Patients with Decreased GFRGenetic variants primarily associated with type 2 diabetes are related to coronary artery disease riskGalectin-3, Renal Function, and Clinical Outcomes: Results from the LURIC and 4D StudiesUric Acid and Cardiovascular Events: A Mendelian Randomization Study.Neutrophil gelatinase-associated lipocalin levels are U-shaped in the Ludwigshafen Risk and Cardiovascular Health (LURIC) study-Impact for mortality.Nitric Oxide-Related Biological Pathways in Patients with Major DepressionC-peptide levels are associated with mortality and cardiovascular mortality in patients undergoing angiography: the LURIC study.Lymphocytes of type 2 diabetic women carry a high load of stable chromosomal aberrations: a novel risk factor for disease-related early death.Fast and Accurate Construction of Confidence Intervals for Heritability.Glycerophospholipid and sphingolipid species and mortality: the Ludwigshafen Risk and Cardiovascular Health (LURIC) study.Molecular lipids identify cardiovascular risk and are efficiently lowered by simvastatin and PCSK9 deficiency.Immune activation and inflammation in patients with cardiovascular disease are associated with higher phenylalanine to tyrosine ratios: the ludwigshafen risk and cardiovascular health studyHDL cholesterol is not associated with lower mortality in patients with kidney dysfunction.Vitamin D supplements in chronic kidney disease.Identifying new Risk Markers and Potential Targets for Coronary Artery Disease: The Value of the Lipidome and Metabolome.Relations between lipoprotein(a) concentrations, LPA genetic variants, and the risk of mortality in patients with established coronary heart disease: a molecular and genetic association study.
P2860
Q24609527-DAF00860-721A-4952-AAEE-D6F03AC93490Q28267312-4B4F0E5F-B8F1-4EB9-8563-61447DD757DCQ29417023-29287E86-30B9-44F0-8CDB-CDED0F84BA48Q30000348-43B89C05-9692-4E99-B1A6-EE94CD841DE7Q30248925-C380CE14-3B80-41D1-9DFE-E5A2988F3E2AQ30310914-28CFFBA0-C5CD-49AD-9A92-C08E28C8A93BQ30619237-37C87CBE-EB8C-4654-AA09-C7CD24325B45Q30907369-B6C1D076-0198-4C76-827A-312A3B2F69B0Q31124944-66B0C11F-49E6-4366-979E-ACE4DA64653EQ33434167-7A8B2960-E2EF-41F9-B813-DDF9D0410F0FQ33709886-D5763A50-E1BD-48A4-A64E-8577D810086AQ33742763-5C584C49-876B-48DD-ADF7-BE105BD1E1DAQ33875341-F6965763-FC38-4353-ACB4-74A1874DDF30Q34034892-BA280DB9-475F-4C03-AE24-7AA5F39D9EADQ34194254-AB624499-37BC-4AB8-9D6F-AF6711A8D1CBQ34243501-3462AF18-17B0-4D39-9C73-9002BAF26D3DQ34500842-2F83741D-8ED4-41AF-9524-62945EF5E213Q34512802-2D90505F-38A4-4973-9AE7-6502C3902DBDQ34540173-B718CF9C-727B-4032-BCAC-5A3831EBCEA6Q34758752-C75624DC-8BD6-4174-9AD1-0D340157979DQ35043306-D2A18E57-4AD7-4E6C-8FC5-9A2568D227AEQ35123404-16F2B9CD-10B7-4BC5-9EE1-93358C25C66EQ35408499-867C2470-87C9-44D8-B8AB-08A44F043692Q35898186-5DCC749F-966E-4AD4-A39D-E009DC2BD307Q35901123-AADB6DED-DD05-4845-B225-8511478087AEQ35955063-DCE8E7A1-C5AC-45DC-B8E4-85D4C2762B17Q36003071-139A6672-C05A-4410-9B4C-11B7589F7DF1Q36223277-7ACBFEA7-974D-4E02-9E64-075EDF6005F3Q36282562-EFBE34C5-499B-4729-BCCB-0C75F0EE724FQ36296008-9AB7E31B-6BBA-4349-87F0-9C08793365B7Q36629649-DAB3393F-7CA5-4F65-9F76-40B56966480CQ36943704-38905ABF-F6FF-44FD-AD84-A58A7C11C240Q37005099-25C20A2F-23F2-4310-B97F-5A8F13465732Q37490790-D5FEDDEF-2D87-4411-8612-FC9636189F64Q37592818-445284BC-8347-4E94-9B66-7CB64D70918EQ37603096-3584B7C2-BADE-4AB2-8A2B-D3C3552130D6Q37733566-9ECFBD0C-B1BC-46FE-B0DB-B16F62D8A473Q38554471-94060D5C-1725-43D2-AE0D-04B7A69D7C1DQ38741609-C05D30E9-BA3E-4CF2-A9B0-CB3FF51588A6Q38752767-6BA39D4F-118A-4BCE-8971-9C06945DB8B2
P2860
Rationale and design of the LURIC study--a resource for functional genomics, pharmacogenomics and long-term prognosis of cardiovascular disease.
description
2001 nî lūn-bûn
@nan
2001 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2001 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2001年の論文
@ja
2001年論文
@yue
2001年論文
@zh-hant
2001年論文
@zh-hk
2001年論文
@zh-mo
2001年論文
@zh-tw
2001年论文
@wuu
name
Rationale and design of the LU ...... sis of cardiovascular disease.
@ast
Rationale and design of the LU ...... sis of cardiovascular disease.
@en
Rationale and design of the LU ...... sis of cardiovascular disease.
@nl
type
label
Rationale and design of the LU ...... sis of cardiovascular disease.
@ast
Rationale and design of the LU ...... sis of cardiovascular disease.
@en
Rationale and design of the LU ...... sis of cardiovascular disease.
@nl
prefLabel
Rationale and design of the LU ...... sis of cardiovascular disease.
@ast
Rationale and design of the LU ...... sis of cardiovascular disease.
@en
Rationale and design of the LU ...... sis of cardiovascular disease.
@nl
P2093
P2860
P356
P1433
P1476
Rationale and design of the LU ...... sis of cardiovascular disease.
@en
P2093
B R Winkelmann
LURIC Study Group (LUdwigshafen RIsk and Cardiovascular Health)
P Hellstern
P2860
P356
10.1517/14622416.2.1.S1
P433
P577
2001-02-01T00:00:00Z