Apoptosis repressor with caspase domain inhibits cardiomyocyte apoptosis by reducing K+ currents.
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Apoptosis repressor with caspase recruitment domain protects against cell death by interfering with Bax activationA critical role for the protein apoptosis repressor with caspase recruitment domain in hypoxia-induced pulmonary hypertensionCatalytic mechanism and substrate specificity of the beta-subunit of the voltage-gated potassium channelComparative normal/failing rat myocardium cell membrane chromatographic analysis system for screening specific components that counteract doxorubicin-induced heart failure from Acontium carmichaeli.Endothelial and smooth muscle cell ion channels in pulmonary vasoconstriction and vascular remodelingAttenuation of apoptosis in enterocytes by blockade of potassium channels.Nrf2 enhances myocardial clearance of toxic ubiquitinated proteinsActivation of K+ channels: an essential pathway in programmed cell death.Cell shrinkage and monovalent cation fluxes: role in apoptosisInteractions between the C-terminus of Kv1.5 and Kvβ regulate pyridine nucleotide-dependent changes in channel gatingFunctional Interactions between BKCaα-Subunit and Annexin A5: Implications in Apoptosis.K+ channels in apoptosis.Potassium channels in the regulation of pulmonary artery smooth muscle cell proliferation and apoptosis: pharmacotherapeutic implications.Programmed cell death in cardiac myocytes: strategies to maximize post-ischemic salvage.Impact of intracellular ion channels on cancer development and progression.Expression and modification of ARC (apoptosis repressor with a CARD domain) is distinctly regulated by oxidative stress in cancer cells.Gene expression profiling of human promyelocytic cells in response to infection with Anaplasma phagocytophilum.Regulation of necrosis of H9c2 myogenic cells upon transient energy deprivation. Rapid deenergization of mitochondria precedes necrosis and is controlled by reactive oxygen species, stress kinase JNK, HSP72 and ARC.AMP-activated protein kinase inhibits Kv 1.5 channel currents of pulmonary arterial myocytes in response to hypoxia and inhibition of mitochondrial oxidative phosphorylation.Thiol oxidation causes pulmonary vasodilation by activating K+ channels and inhibiting store-operated Ca2+ channels.Differential expression of a novel voltage gated potassium channel--Kv 1.5 in astrocytomas and its impact on prognosis in glioblastoma.
P2860
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P2860
Apoptosis repressor with caspase domain inhibits cardiomyocyte apoptosis by reducing K+ currents.
description
2003 nî lūn-bûn
@nan
2003 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2003 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
name
Apoptosis repressor with caspa ...... tosis by reducing K+ currents.
@ast
Apoptosis repressor with caspa ...... tosis by reducing K+ currents.
@en
Apoptosis repressor with caspa ...... tosis by reducing K+ currents.
@nl
type
label
Apoptosis repressor with caspa ...... tosis by reducing K+ currents.
@ast
Apoptosis repressor with caspa ...... tosis by reducing K+ currents.
@en
Apoptosis repressor with caspa ...... tosis by reducing K+ currents.
@nl
prefLabel
Apoptosis repressor with caspa ...... tosis by reducing K+ currents.
@ast
Apoptosis repressor with caspa ...... tosis by reducing K+ currents.
@en
Apoptosis repressor with caspa ...... tosis by reducing K+ currents.
@nl
P2093
P2860
P1476
Apoptosis repressor with caspa ...... tosis by reducing K+ currents.
@en
P2093
Carmelle V Remillard
Daryoush Ekhterae
Jason X-J Yuan
Oleksandr Platoshyn
Shen Zhang
P2860
P304
P356
10.1152/AJPCELL.00279.2002
P577
2003-01-29T00:00:00Z