Nutritional recovery promotes hypothalamic inflammation in rats during adulthood - Wikidata - awgldk - TriplyDB

Nutritional recovery promotes hypothalamic inflammation in rats during adulthood

Nutritional recovery promotes hypothalamic inflammation in rats during adulthood

http://www.wikidata.org/entity/Q34199264

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IL-17A-producing CD8(+)T cells as therapeutic targets in autoimmunity.Live Attenuated Leishmania donovani Centrin Gene-Deleted Parasites Induce IL-23-Dependent IL-17-Protective Immune Response against Visceral Leishmaniasis in a Murine Model.

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Nutritional recovery promotes hypothalamic inflammation in rats during adulthood

http://www.wikidata.org/entity/Q34199264

description

2014 nî lūn-bûn

@nan

2014 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի օգոստոսին հրատարակված գիտական հոդված

@hy

2014年の論文

@ja

2014年論文

@yue

2014年論文

@zh-hant

2014年論文

@zh-hk

2014年論文

@zh-mo

2014年論文

@zh-tw

2014年论文

@wuu

name

Nutritional recovery promotes hypothalamic inflammation in rats during adulthood

@ast

Nutritional recovery promotes hypothalamic inflammation in rats during adulthood

@en

Nutritional recovery promotes hypothalamic inflammation in rats during adulthood

@nl

type

label

Nutritional recovery promotes hypothalamic inflammation in rats during adulthood

@ast

Nutritional recovery promotes hypothalamic inflammation in rats during adulthood

@en

Nutritional recovery promotes hypothalamic inflammation in rats during adulthood

@nl

prefLabel

Nutritional recovery promotes hypothalamic inflammation in rats during adulthood

@ast

Nutritional recovery promotes hypothalamic inflammation in rats during adulthood

@en

Nutritional recovery promotes hypothalamic inflammation in rats during adulthood

@nl

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Mediators of Inflammation

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Nutritional recovery promotes hypothalamic inflammation in rats during adulthood

@en

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Ana Paula Carli de Almeida

http://www.w3.org/2001/XMLSchema#string

Hellen Barbosa Farias Silva

http://www.w3.org/2001/XMLSchema#string

Katarine Barbosa Cardoso

http://www.w3.org/2001/XMLSchema#string

Letícia Martins Ignacio-Souza

http://www.w3.org/2001/XMLSchema#string

Marciane Milanski

http://www.w3.org/2001/XMLSchema#string

Marise Auxiliadora de Barros Reis

http://www.w3.org/2001/XMLSchema#string

Silvia Regina de Lima Reis

http://www.w3.org/2001/XMLSchema#string

Vanessa Cristina Arantes

http://www.w3.org/2001/XMLSchema#string

P2860

Brain circuits regulating energy homeostasis

A simple method for the isolation and purification of total lipides from animal tissues

Saturated fatty acids produce an inflammatory response predominantly through the activation of TLR4 signaling in hypothalamus: implications for the pathogenesis of obesity

Protection from obesity-induced insulin resistance in mice lacking TNF-alpha function

Effects of the obese gene product on body weight regulation in ob/ob mice

Transgenerational effects of prenatal exposure to the Dutch famine on neonatal adiposity and health in later life.

Leptin levels reflect body lipid content in mice: evidence for diet-induced resistance to leptin action.

TNF-alpha antagonism with etanercept decreases glucose and increases the proportion of high molecular weight adiponectin in obese subjects with features of the metabolic syndrome.

For debate: Fetal and early postnatal growth restriction lead to diabetes, the metabolic syndrome and renal failure.

Analysis of serial measurements in medical research.

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736506

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scholarly article

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10.1155/2014/736506

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2014

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Márcia Queiroz Latorraca

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2014-08-31T00:00:00Z

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266245607

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25258479

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4166447

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