Circadian CLOCK histone acetyl transferase localizes at ND10 nuclear bodies and enables herpes simplex virus gene expression
about
Role of ND10 nuclear bodies in the chromatin repression of HSV-1Infected cell protein 0 functional domains and their coordination in herpes simplex virus replicationKDM1 class flavin-dependent protein lysine demethylasesCircadian proteins CLOCK and BMAL1 in the chromatoid body, a RNA processing granule of male germ cellsHuman-specific transcriptional networks in the brain.The stability of herpes simplex virus 1 ICP0 early after infection is defined by the RING finger and the UL13 protein kinase.Novel roles of cytoplasmic ICP0: proteasome-independent functions of the RING finger are required to block interferon-stimulated gene production but not to promote viral replication.A neuron-specific host microRNA targets herpes simplex virus-1 ICP0 expression and promotes latency.Mutual antagonism between circadian protein period 2 and hepatitis C virus replication in hepatocytes.HSV-1 genome subnuclear positioning and associations with host-cell PML-NBs and centromeres regulate LAT locus transcription during latency in neurons.Cells infected with herpes simplex virus 1 export to uninfected cells exosomes containing STING, viral mRNAs, and microRNAs.The checkpoints of viral gene expression in productive and latent infection: the role of the HDAC/CoREST/LSD1/REST repressor complexThe histone acetyltransferase CLOCK is an essential component of the herpes simplex virus 1 transcriptome that includes TFIID, ICP4, ICP27, and ICP22.DNA mismatch repair proteins are required for efficient herpes simplex virus 1 replicationDynamic modulation of HSV chromatin drives initiation of infection and provides targets for epigenetic therapies.Nuclear sensing of viral DNA, epigenetic regulation of herpes simplex virus infection, and innate immunityOverexpression of the ubiquitin-specific protease 7 resulting from transfection or mutations in the ICP0 binding site accelerates rather than depresses herpes simplex virus 1 gene expressionUnbiased proteomic analysis of proteins interacting with the HIV-1 5'LTR sequence: role of the transcription factor Meis.Use of biotinylated plasmid DNA as a surrogate for HSV DNA to identify proteins that repress or activate viral gene expression.Herpesviral ICP0 Protein Promotes Two Waves of Heterochromatin Removal on an Early Viral Promoter during Lytic Infection.Chromatin Modulation of Herpesvirus Lytic Gene Expression: Managing Nucleosome Density and Heterochromatic Histone Modifications.Cellular Transcriptional Coactivator RanBP10 and Herpes Simplex Virus 1 ICP0 Interact and Synergistically Promote Viral Gene Expression and Replication.Genetics of circadian rhythms in Mammalian model organisms.The dynamics of HCF-1 modulation of herpes simplex virus chromatin during initiation of infection.Interaction of herpes simplex virus ICP0 with ND10 bodies: a sequential process of adhesion, fusion, and retentionCell autonomous regulation of herpes and influenza virus infection by the circadian clock.The nuclear-cytoplasmic shuttling of virion host shutoff RNase is enabled by pUL47 and an embedded nuclear export signal and defines the sites of degradation of AU-rich and stable cellular mRNAsEpigenetic repression of herpes simplex virus infection by the nucleosome remodeler CHD3HSV-1 degrades, stabilizes, requires, or is stung by STING depending on ICP0, the US3 protein kinase, and cell derivation.A targeted RNA interference screen reveals novel epigenetic factors that regulate herpesviral gene expression.Post-translational timing mechanisms of the Drosophila circadian clock.How to control an infectious bead string: nucleosome-based regulation and targeting of herpesvirus chromatin.Checkpoints in productive and latent infections with herpes simplex virus 1: conceptualization of the issues.Histone deacetylases in herpesvirus replication and virus-stimulated host defense.Chromatin dynamics during lytic infection with herpes simplex virus 1.Host-virus interactions: from the perspectives of epigenetics.The 3 facets of regulation of herpes simplex virus gene expression: A critical inquiry.The Life and Times of Parasites: Rhythms in Strategies for Within-host Survival and Between-host Transmission.Evasion of the STING DNA-Sensing Pathway by VP11/12 of Herpes Simplex Virus 1.Impaired STING Pathway in Human Osteosarcoma U2OS Cells Contributes to the Growth of ICP0-Null Mutant Herpes Simplex Virus.
P2860
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P2860
Circadian CLOCK histone acetyl transferase localizes at ND10 nuclear bodies and enables herpes simplex virus gene expression
description
2010 nî lūn-bûn
@nan
2010 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年学术文章
@wuu
2010年学术文章
@zh-cn
2010年学术文章
@zh-hans
2010年学术文章
@zh-my
2010年学术文章
@zh-sg
2010年學術文章
@yue
name
Circadian CLOCK histone acetyl ...... simplex virus gene expression
@ast
Circadian CLOCK histone acetyl ...... simplex virus gene expression
@en
Circadian CLOCK histone acetyl ...... simplex virus gene expression
@nl
type
label
Circadian CLOCK histone acetyl ...... simplex virus gene expression
@ast
Circadian CLOCK histone acetyl ...... simplex virus gene expression
@en
Circadian CLOCK histone acetyl ...... simplex virus gene expression
@nl
prefLabel
Circadian CLOCK histone acetyl ...... simplex virus gene expression
@ast
Circadian CLOCK histone acetyl ...... simplex virus gene expression
@en
Circadian CLOCK histone acetyl ...... simplex virus gene expression
@nl
P2860
P356
P1476
Circadian CLOCK histone acetyl ...... simplex virus gene expression
@en
P2860
P304
17721-17726
P356
10.1073/PNAS.1012991107
P407
P577
2010-09-27T00:00:00Z