EMT transcription factors snail and slug directly contribute to cisplatin resistance in ovarian cancer - Wikidata - awgldk - TriplyDB

EMT transcription factors snail and slug directly contribute to cisplatin resistance in ovarian cancer

EMT transcription factors snail and slug directly contribute to cisplatin resistance in ovarian cancer

http://www.wikidata.org/entity/Q34202046

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Targeting Epithelial-Mesenchymal Transition (EMT) to Overcome Drug Resistance in Cancer EMT-Inducing Molecular Factors in Gynecological Cancers Central role of Snail1 in the regulation of EMT and resistance in cancer: a target for therapeutic intervention The molecular fingerprint of high grade serous ovarian cancer reflects its fallopian tube origin Radioactive (125)I seeds inhibit cell growth and epithelial-mesenchymal transition in human glioblastoma multiforme via a ROS-mediated signaling pathway Expression of epidermal growth factor‑like domain 7 may be a predictive marker of the effect of neoadjuvant chemotherapy for locally advanced uterine cervical cancer Resveratrol inhibits cisplatin-induced epithelial-to-mesenchymal transition in ovarian cancer cell lines Gene expression profiling reveals epithelial mesenchymal transition (EMT) genes can selectively differentiate eribulin sensitive breast cancer cells Integrin β4 promotes cell invasion and epithelial-mesenchymal transition through the modulation of Slug expression in hepatocellular carcinoma Non-invasive analysis of acquired resistance to cancer therapy by sequencing of plasma DNA Flow induces epithelial-mesenchymal transition, cellular heterogeneity and biomarker modulation in 3D ovarian cancer nodules.TCRP1 expression is associated with platinum sensitivity in human lung and ovarian cancer cells.Involvement of AF1q/MLLT11 in the progression of ovarian cancer SLUG transcription factor: a pro-survival and prognostic factor in gastrointestinal stromal tumour Downregulation of miR-218 contributes to epithelial-mesenchymal transition and tumor metastasis in lung cancer by targeting Slug/ZEB2 signaling Immunohistochemical analysis of NANOG expression and epithelial-mesenchymal transition in pulmonary sarcomatoid carcinoma.Ethaselen: a novel organoselenium anticancer agent targeting thioredoxin reductase 1 reverses cisplatin resistance in drug-resistant K562 cells by inducing apoptosis.Decreased levels of baseline and drug-induced tubulin polymerisation are hallmarks of resistance to taxanes in ovarian cancer cells and are associated with epithelial-to-mesenchymal transition Reverse epithelial-mesenchymal transition contributes to the regain of drug sensitivity in tyrosine kinase inhibitor-resistant non-small cell lung cancer cells.EMT and EGFR in CTCs cytokeratin negative non-metastatic breast cancer Slug is associated with poor survival in squamous cell carcinoma of the lung.OvMark: a user-friendly system for the identification of prognostic biomarkers in publically available ovarian cancer gene expression datasets.CD151-α3β1 integrin complexes suppress ovarian tumor growth by repressing slug-mediated EMT and canonical Wnt signaling.Slug contributes to cancer progression by direct regulation of ERα signaling pathway Cisplatin-selected resistance is associated with increased motility and stem-like properties via activation of STAT3/Snail axis in atypical teratoid/rhabdoid tumor cells Fibrillin-1, induced by Aurora-A but inhibited by BRCA2, promotes ovarian cancer metastasis The role of photodynamic therapy in overcoming cancer drug resistance.Aberrant expression of Notch1/numb/snail signaling, an epithelial mesenchymal transition related pathway, in adenomyosis.Regulation of the protein stability of EMT transcription factors Inhibition of Snail Family Transcriptional Repressor 2 (SNAI2) Enhances Multidrug Resistance of Hepatocellular Carcinoma Cells Expression of Par3 polarity protein correlates with poor prognosis in ovarian cancer SLUG is a direct transcriptional repressor of PTEN tumor suppressor.Disruption of TWIST1-RELA binding by mutation and competitive inhibition to validate the TWIST1 WR domain as a therapeutic target.NRP1 is targeted by miR-130a and miR-130b, and is associated with multidrug resistance in epithelial ovarian cancer based on integrated gene network analysis.Loss of Scribble Promotes Snail Translation through Translocation of HuR and Enhances Cancer Drug Resistance A multi-stage process including transient polyploidization and EMT precedes the emergence of chemoresistent ovarian carcinoma cells with a dedifferentiated and pro-inflammatory secretory phenotype miR-3188 regulates nasopharyngeal carcinoma proliferation and chemosensitivity through a FOXO1-modulated positive feedback loop with mTOR-p-PI3K/AKT-c-JUN.Migration and invasion is inhibited by silencing ROR1 and ROR2 in chemoresistant ovarian cancer.Inhibition of endotrophin, a cleavage product of collagen VI, confers cisplatin sensitivity to tumours.Roles Each of Snail, Yin Yang 1 and RKIP in the Regulation of Tumor Cells Chemo-immuno-resistance to Apoptosis.

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EMT transcription factors snail and slug directly contribute to cisplatin resistance in ovarian cancer

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Harriet Feilotter

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Jian Chen

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Moyez Dharsee

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Paul C Park

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P2860

Ovarian carcinoma subtypes are different diseases: implications for biomarker studies

Epithelial-mesenchymal transition in cancer: parallels between normal development and tumor progression

The basics of epithelial-mesenchymal transition

Analyzing real-time PCR data by the comparative C(T) method

The mesenchymal cell, its role in the embryo, and the remarkable signaling mechanisms that create it

Promotion of hepatocellular carcinoma metastasis through matrix metalloproteinase activation by epithelial-mesenchymal transition regulator Twist1

Epithelial-mesenchymal transition: at the crossroads of development and tumor metastasis

Snail blocks the cell cycle and confers resistance to cell death

Pathway analysis of gene lists associated with platinum-based chemotherapy resistance in ovarian cancer: the big picture.

Drug transporters of platinum-based anticancer agents and their clinical significance.

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