Reactive oxygen species originating from mitochondria regulate the cardiac sodium channel.
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Cellular and Molecular Mechanisms of Arrhythmia by Oxidative StressCardiomyocyte Remodeling in Atrial Fibrillation and Hibernating Myocardium: Shared Pathophysiologic Traits Identify Novel Treatment Strategies?Regulation of ion channels by pyridine nucleotidesHypoxamiRs and cancer: from biology to targeted therapyMechanisms of sudden cardiac death: oxidants and metabolismIon channel macromolecular complexes in cardiomyocytes: roles in sudden cardiac deathPost-translational modifications of the cardiac Na channel: contribution of CaMKII-dependent phosphorylation to acquired arrhythmiasmiR-210: More than a silent player in hypoxiaCross talk between mitochondria and NADPH oxidasesTherapeutic inhibition of mitochondrial reactive oxygen species with mito-TEMPO reduces diabetic cardiomyopathyReduced Na⁺ current density underlies impaired propagation in the diabetic rabbit ventricleIn vitro cardiotoxicity assessment of environmental chemicals using an organotypic human induced pluripotent stem cell-derived model.Cardiac Na Channels: Structure to FunctionInhibition of the cardiac Na⁺ channel Nav1.5 by carbon monoxide.Mitochondrial dysfunction on sinoatrial node and pulmonary vein electrophysiological activitiesMitochondria and arrhythmias.A mutation causing Brugada syndrome identifies a mechanism for altered autonomic and oxidant regulation of cardiac sodium currentsEffective antioxidant therapy for the management of arrhythmiaDiseases caused by mutations in Nav1.5 interacting proteinsMitochondria are sources of metabolic sink and arrhythmias.Mitochondria-derived ROS bursts disturb Ca²⁺ cycling and induce abnormal automaticity in guinea pig cardiomyocytes: a theoretical study.Metabolic stress, reactive oxygen species, and arrhythmia.miR-210: fine-tuning the hypoxic response.Mitochondria-derived superoxide and voltage-gated sodium channels in baroreceptor neurons from chronic heart-failure ratsMouse lung development and NOX1 induction during hyperoxia are developmentally regulated and mitochondrial ROS dependent.Reactive oxygen species-targeted therapeutic interventions for atrial fibrillation.Changes in Intracellular Na+ following Enhancement of Late Na+ Current in Virtual Human Ventricular Myocytes.Mitochondrial dysfunction causing cardiac sodium channel downregulation in cardiomyopathySensory nerve terminal mitochondrial dysfunction activates airway sensory nerves via transient receptor potential (TRP) channelsCardiac sodium channel mutations: why so many phenotypes?Ketamine induces toxicity in human neurons differentiated from embryonic stem cells via mitochondrial apoptosis pathway.Mitochondria oxidative stress, connexin43 remodeling, and sudden arrhythmic deathRegulation of the Na+/Ca2+ exchanger by pyridine nucleotide redox potential in ventricular myocytesReactive species modify NaV1.8 channels and affect action potentials in murine dorsal root ganglion neurons.Control of glutamine metabolism by the tumor suppressor Rb.Cardiac electrophysiological alterations in heart/muscle-specific manganese-superoxide dismutase-deficient mice: prevention by a dietary antioxidant polyphenol.Redox control of cardiac excitability.Redox regulation of sodium and calcium handlingOverview of pyridine nucleotides review series.Cardiac sodium channelopathy associated with SCN5A mutations: electrophysiological, molecular and genetic aspects.
P2860
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P2860
Reactive oxygen species originating from mitochondria regulate the cardiac sodium channel.
description
2010 nî lūn-bûn
@nan
2010 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年学术文章
@wuu
2010年学术文章
@zh-cn
2010年学术文章
@zh-hans
2010年学术文章
@zh-my
2010年学术文章
@zh-sg
2010年學術文章
@yue
name
Reactive oxygen species origin ...... te the cardiac sodium channel.
@ast
Reactive oxygen species origin ...... te the cardiac sodium channel.
@en
Reactive oxygen species origin ...... te the cardiac sodium channel.
@nl
type
label
Reactive oxygen species origin ...... te the cardiac sodium channel.
@ast
Reactive oxygen species origin ...... te the cardiac sodium channel.
@en
Reactive oxygen species origin ...... te the cardiac sodium channel.
@nl
prefLabel
Reactive oxygen species origin ...... te the cardiac sodium channel.
@ast
Reactive oxygen species origin ...... te the cardiac sodium channel.
@en
Reactive oxygen species origin ...... te the cardiac sodium channel.
@nl
P2860
P1433
P1476
Reactive oxygen species originating from mitochondria regulate the cardiac sodium channel
@en
P2093
P2860
P304
P356
10.1161/CIRCRESAHA.110.220673
P577
2010-08-19T00:00:00Z