NADPH oxidase and PKC contribute to increased Na transport by the thick ascending limb during type 1 diabetes - Wikidata - awgldk - TriplyDB

NADPH oxidase and PKC contribute to increased Na transport by the thick ascending limb during type 1 diabetes

NADPH oxidase and PKC contribute to increased Na transport by the thick ascending limb during type 1 diabetes

http://www.wikidata.org/entity/Q34213306

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NADPH oxidase inhibition reduces tubular sodium transport and improves kidney oxygenation in diabetes.NADPH oxidase 4 mediates flow-induced superoxide production in thick ascending limbs.Predicted consequences of diabetes and SGLT inhibition on transport and oxygen consumption along a rat nephron.Interaction between NO synthase and NADPH oxidase in control of sodium transport by the renal thick ascending limb during diabetes.Oxidative stress in hypertension: role of the kidney.Glucose Homeostasis and Cardiovascular Alterations in Diabetes.NADPH-oxidase-driven oxidative stress during experimental diabetes offsets NO-mediated regulation of renal medullary sodium transport. A potential treatment modality during type 1 diabetes?

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NADPH oxidase and PKC contribute to increased Na transport by the thick ascending limb during type 1 diabetes

http://www.wikidata.org/entity/Q34213306

description

2011 nî lūn-bûn

@nan

2011 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2011 թվականի դեկտեմբերին հրատարակված գիտական հոդված

@hy

2011年の論文

@ja

2011年論文

@yue

2011年論文

@zh-hant

2011年論文

@zh-hk

2011年論文

@zh-mo

2011年論文

@zh-tw

2011年论文

@wuu

name

NADPH oxidase and PKC contribu ...... ng limb during type 1 diabetes

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NADPH oxidase and PKC contribu ...... ng limb during type 1 diabetes

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NADPH oxidase and PKC contribu ...... ng limb during type 1 diabetes

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type

label

NADPH oxidase and PKC contribu ...... ng limb during type 1 diabetes

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NADPH oxidase and PKC contribu ...... ng limb during type 1 diabetes

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NADPH oxidase and PKC contribu ...... ng limb during type 1 diabetes

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prefLabel

NADPH oxidase and PKC contribu ...... ng limb during type 1 diabetes

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NADPH oxidase and PKC contribu ...... ng limb during type 1 diabetes

@en

NADPH oxidase and PKC contribu ...... ng limb during type 1 diabetes

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HYPERTENSIONAHA.111.184796

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NADPH oxidase and PKC contribu ...... ng limb during type 1 diabetes

@en

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Jennifer S Pollock

http://www.w3.org/2001/XMLSchema#string

Jing Yang

http://www.w3.org/2001/XMLSchema#string

Pamela K Carmines

http://www.w3.org/2001/XMLSchema#string

P2860

Tubular sodium handling and tubuloglomerular feedback in experimental diabetes mellitus

Control of NaCl transport in the thick ascending limb

Immunolocalization of protein kinase C isoenzymes alpha, beta1 and betaII in rat kidney.

Reactive oxygen species cause diabetes-induced decrease in renal oxygen tension.

Polyol-pathway-dependent disturbances in renal medullary metabolism in experimental insulin-deficient diabetes mellitus in rats.

Immunolocalization of protein kinase C isoenzymes alpha, beta I, beta II, delta, and epsilon in mouse kidney.

Targeting SGK1 in diabetes

Dopamine-induced exocytosis of Na,K-ATPase is dependent on activation of protein kinase C-epsilon and -delta.

Characterization of SPAK and OSR1, regulatory kinases of the Na-K-2Cl cotransporter.

Is phosphorylation of the alpha1 subunit at Ser-16 involved in the control of Na,K-ATPase activity by phorbol ester-activated protein kinase C?

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431-436

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scholarly article

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10.1161/HYPERTENSIONAHA.111.184796

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2

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59

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2011-12-27T00:00:00Z

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51895895

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22203737

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type-1 diabetes

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4169139

http://www.w3.org/2001/XMLSchema#string