Oxygen-coupled redox regulation of the skeletal muscle ryanodine receptor-Ca2+ release channel by NADPH oxidase 4 - Wikidata - awgldk - TriplyDB

Oxygen-coupled redox regulation of the skeletal muscle ryanodine receptor-Ca2+ release channel by NADPH oxidase 4

Oxygen-coupled redox regulation of the skeletal muscle ryanodine receptor-Ca2+ release channel by NADPH oxidase 4

http://www.wikidata.org/entity/Q34213979

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Calcium and ROS: A mutual interplay Biochemistry, physiology, and pathophysiology of NADPH oxidases in the cardiovascular system NADPH oxidases in heart failure: poachers or gamekeepers?Skeletal Muscle Regeneration and Oxidative Stress Are Altered in Chronic Kidney Disease Review of RyR1 pathway and associated pathomechanisms Reactive Oxygen Species/Nitric Oxide Mediated Inter-Organ Communication in Skeletal Muscle Wasting Diseases.Cardiac ryanodine receptor activation by a high Ca²⁺ store load is reversed in a reducing cytoplasmic redox environment.Diaphragm dysfunction caused by sphingomyelinase requires the p47(phox) subunit of NADPH oxidase.Real-time imaging of NADPH oxidase activity in living cells using a novel fluorescent protein reporter Chronic intermittent hypoxia increases rat sternohyoid muscle NADPH oxidase expression with attendant modest oxidative stress.Skeletal muscle contractions induce acute changes in cytosolic superoxide, but slower responses in mitochondrial superoxide and cellular hydrogen peroxide Nox4: a hydrogen peroxide-generating oxygen sensor.Nox4 Is Dispensable for Exercise Induced Muscle Fibre Switch Regulation of cell physiology and pathology by protein S-glutathionylation: lessons learned from the cardiovascular system.Off-target thiol alkylation by the NADPH oxidase inhibitor 3-benzyl-7-(2-benzoxazolyl)thio-1,2,3-triazolo[4,5-d]pyrimidine (VAS2870).NOX2 As a Target for Drug Development: Indications, Possible Complications, and Progress.Excess TGF-β mediates muscle weakness associated with bone metastases in mice.Crosstalk between RyR2 oxidation and phosphorylation contributes to cardiac dysfunction in mice with Duchenne muscular dystrophy.Absence of Dystrophin Disrupts Skeletal Muscle Signaling: Roles of Ca2+, Reactive Oxygen Species, and Nitric Oxide in the Development of Muscular Dystrophy.S-nitrosylation: integrator of cardiovascular performance and oxygen delivery.NADPH oxidase 4 is required for interleukin-1β-mediated activation of protein kinase Cδ and downstream activation of c-jun N-terminal kinase signaling in smooth muscle.The Endoplasmic Reticulum Chaperone Calnexin Is a NADPH Oxidase NOX4 Interacting Protein.Low Po₂ conditions induce reactive oxygen species formation during contractions in single skeletal muscle fibers Interactions between sarco-endoplasmic reticulum and mitochondria in cardiac and skeletal muscle - pivotal roles in Ca²⁺ and reactive oxygen species signaling.Prolonged fasting activates Nrf2 in post-weaned elephant seals.Oxygen-coupled redox regulation of the skeletal muscle ryanodine receptor/Ca2+ release channel (RyR1): sites and nature of oxidative modification.Differential Expression of NADPH Oxidases Depends on Skeletal Muscle Fiber Type in Rats.Angiotensin receptor-mediated oxidative stress is associated with impaired cardiac redox signaling and mitochondrial function in insulin-resistant rats.Regulation of the skeletal muscle ryanodine receptor/Ca2+-release channel RyR1 by S-palmitoylation.TRP channels as sensors of oxygen availability.Nox NADPH oxidases and the endoplasmic reticulum Redox homeostasis and age-related deficits in neuromuscular integrity and function.Regulation of NADPH oxidases in skeletal muscle.Mitochondria in the middle: exercise preconditioning protection of striated muscle.Exercise training decreases NADPH oxidase activity and restores skeletal muscle mass in heart failure rats.Subtype-selective regulation of IP(3) receptors by thimerosal via cysteine residues within the IP(3)-binding core and suppressor domain Mitochondrial ROS versus ER ROS: Which Comes First in Myocardial Calcium Dysregulation?Effect of oxygen tension on bioenergetics and proteostasis in young and old myoblast precursor cells.An olive oil-derived antioxidant mixture ameliorates the age-related decline of skeletal muscle function.Oxidative stress induces stem cell proliferation via TRPA1/RyR-mediated Ca2+ signaling in the Drosophila midgut.

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Oxygen-coupled redox regulation of the skeletal muscle ryanodine receptor-Ca2+ release channel by NADPH oxidase 4

http://www.wikidata.org/entity/Q34213979

description

2011 nî lūn-bûn

@nan

2011 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

@hyw

2011 թվականի սեպտեմբերին հրատարակված գիտական հոդված

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2011年の論文

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2011年学术文章

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2011年学术文章

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2011年学术文章

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2011年学术文章

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2011年学术文章

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2011年學術文章

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Oxygen-coupled redox regulatio ...... ase channel by NADPH oxidase 4

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Oxygen-coupled redox regulatio ...... ase channel by NADPH oxidase 4

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Oxygen-coupled redox regulatio ...... ase channel by NADPH oxidase 4

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Oxygen-coupled redox regulatio ...... ase channel by NADPH oxidase 4

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Oxygen-coupled redox regulatio ...... ase channel by NADPH oxidase 4

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Oxygen-coupled redox regulatio ...... ase channel by NADPH oxidase 4

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Oxygen-coupled redox regulatio ...... ase channel by NADPH oxidase 4

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PNAS.1109546108

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Proceedings of the National Academy of Sciences of the United States of America

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Oxygen-coupled redox regulatio ...... ase channel by NADPH oxidase 4

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P2093

Dawn E Bowles

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Douglas T Hess

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Gerhard Meissner

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Jerry Eu

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Jonathan S Stamler

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Kenneth R Laurita

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Leonardo Nogueira

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Qi-An Sun

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Sandro Yong

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P2860

Leaky RyR2 trigger ventricular arrhythmias in Duchenne muscular dystrophy

Hypernitrosylated ryanodine receptor calcium release channels are leaky in dystrophic muscle

Remodeling of ryanodine receptor complex causes "leaky" channels: a molecular mechanism for decreased exercise capacity

The NOX family of ROS-generating NADPH oxidases: physiology and pathophysiology

Functional analysis of Nox4 reveals unique characteristics compared to other NADPH oxidases

Structure, regulation and evolution of Nox-family NADPH oxidases that produce reactive oxygen species

Identification of hyperreactive cysteines within ryanodine receptor type 1 by mass spectrometry.

Detecting acute changes in oxygen: will the real sensor please stand up?

Identification of cysteines involved in S-nitrosylation, S-glutathionylation, and oxidation to disulfides in ryanodine receptor type 1.

Acute oxygen-sensing mechanisms.

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16098-16103

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scholarly article

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10.1073/PNAS.1109546108

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38

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108

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2011-09-06T00:00:00Z

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51624048

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21896730

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3179127

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