JS-K, a glutathione/glutathione S-transferase-activated nitric oxide releasing prodrug inhibits androgen receptor and WNT-signaling in prostate cancer cells - Wikidata - awgldk - TriplyDB

JS-K, a glutathione/glutathione S-transferase-activated nitric oxide releasing prodrug inhibits androgen receptor and WNT-signaling in prostate cancer cells

JS-K, a glutathione/glutathione S-transferase-activated nitric oxide releasing prodrug inhibits androgen receptor and WNT-signaling in prostate cancer cells

http://www.wikidata.org/entity/Q34214362

about

Novel strategies to target the ubiquitin proteasome system in multiple myeloma Nitric oxide-mediated sensitization of resistant tumor cells to apoptosis by chemo-immunotherapeutics Adaptive responses of androgen receptor signaling in castration-resistant prostate cancer Stilbene induced inhibition of androgen receptor dimerization: implications for AR and ARΔLBD-signalling in human prostate cancer cells JS-K promotes apoptosis by inducing ROS production in human prostate cancer cells JS-K, a nitric oxide pro-drug, regulates growth and apoptosis through the ubiquitin-proteasome pathway in prostate cancer cells Glutathione S-conjugates as prodrugs to target drug-resistant tumors.Diagnostic value of SFRP1 as a favorable predictive and prognostic biomarker in patients with prostate cancer.A reactive oxygen species activation mechanism contributes to JS-K-induced apoptosis in human bladder cancer cells.Glutathione Transferase (GST)-Activated Prodrugs.The yin and yang of nitric oxide in cancer progression.miRNA and TMPRSS2-ERG do not mind their own business in prostate cancer cells.JS-K, a GST-activated nitric oxide donor prodrug, enhances chemo-sensitivity in renal carcinoma cells and prevents cardiac myocytes toxicity induced by Doxorubicin.Endothelial NO Synthase-Dependent S-Nitrosylation of β-Catenin Prevents Its Association with TCF4 and Inhibits Proliferation of Endothelial Cells Stimulated by Wnt3a.Glutathione S -transferase π: a potential role in antitumor therapy

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P2860

JS-K, a glutathione/glutathione S-transferase-activated nitric oxide releasing prodrug inhibits androgen receptor and WNT-signaling in prostate cancer cells

http://www.wikidata.org/entity/Q34214362

description

2012 nî lūn-bûn

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2012 թուականի Մարտին հրատարակուած գիտական յօդուած

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2012 թվականի մարտին հրատարակված գիտական հոդված

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2012年の論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年论文

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name

JS-K, a glutathione/glutathion ...... aling in prostate cancer cells

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JS-K, a glutathione/glutathion ...... aling in prostate cancer cells

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JS-K, a glutathione/glutathion ...... aling in prostate cancer cells

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JS-K, a glutathione/glutathion ...... aling in prostate cancer cells

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JS-K, a glutathione/glutathion ...... aling in prostate cancer cells

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JS-K, a glutathione/glutathion ...... aling in prostate cancer cells

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JS-K, a glutathione/glutathion ...... aling in prostate cancer cells

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JS-K, a glutathione/glutathion ...... aling in prostate cancer cells

@en

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Andrea Hessenauer

http://www.w3.org/2001/XMLSchema#string

Andres J Schrader

http://www.w3.org/2001/XMLSchema#string

Klaus-Dieter Spindler

http://www.w3.org/2001/XMLSchema#string

Marcus V Cronauer

http://www.w3.org/2001/XMLSchema#string

Mark Schrader

http://www.w3.org/2001/XMLSchema#string

Martin Laschak

http://www.w3.org/2001/XMLSchema#string

Wolfgang Streicher

http://www.w3.org/2001/XMLSchema#string

P2860

Rapid colorimetric assay for cellular growth and survival: Application to proliferation and cytotoxicity assays

Cancer statistics, 2010

Nitric oxide donating nonsteroidal anti-inflammatory drugs induce apoptosis in human prostate cancer cell systems and human prostatic stroma via caspase-3

Limitations of MTT and MTS-based assays for measurement of antiproliferative activity of green tea polyphenols

Analysis of nitrate, nitrite, and [15N]nitrate in biological fluids

Rapid nitric oxide-mediated S-nitrosylation of estrogen receptor: regulation of estrogen-dependent gene transcription

Inhibition of glycogen synthase kinase-3β counteracts ligand-independent activity of the androgen receptor in castration resistant prostate cancer.

JS-K has potent anti-angiogenic activity in vitro and inhibits tumour angiogenesis in a multiple myeloma model in vivo.

Constitutively active androgen receptor splice variants expressed in castration-resistant prostate cancer require full-length androgen receptor

In vivo amplification of the androgen receptor gene and progression of human prostate cancer.

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1471-2407-12-130

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130

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scholarly article

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10.1186/1471-2407-12-130

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12

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2012-03-30T00:00:00Z

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223991429

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1028671494

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22462810

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3376035

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