Antagonistic crosstalk between type I and II interferons and increased host susceptibility to bacterial infections
about
The Dialogue of the Host-Parasite Relationship: Leishmania spp. and Trypanosoma cruzi InfectionDifferential effects of type I and II interferons on myeloid cells and resistance to intracellular bacterial infectionsPhenotypic diversity and emerging new tools to study macrophage activation in bacterial infectious diseasesOverlapping, additive and counterregulatory effects of type II and I interferons on myeloid dendritic cell functionsCrosstalk between Mycobacterium tuberculosis and the host cellThe bacterial pathogen Listeria monocytogenes and the interferon family: type I, type II and type III interferonsDown regulation of macrophage IFNGR1 exacerbates systemic L. monocytogenes infectionProduction of IFN-β during Listeria monocytogenes infection is restricted to monocyte/macrophage lineageType I interferons in bacterial infections: taming of myeloid cells and possible implications for autoimmunity.Stat1 negatively regulates immune-mediated injury with Anaplasma phagocytophilum infection.Route of Infection Determines the Impact of Type I Interferons on Innate Immunity to Listeria monocytogenesMPYS is required for IFN response factor 3 activation and type I IFN production in the response of cultured phagocytes to bacterial second messengers cyclic-di-AMP and cyclic-di-GMP.Innate and adaptive interferons suppress IL-1α and IL-1β production by distinct pulmonary myeloid subsets during Mycobacterium tuberculosis infection.Cryptococcus neoformans Infection in Mice Lacking Type I Interferon Signaling Leads to Increased Fungal Clearance and IL-4-Dependent Mucin Production in the Lungs.Analysis of Transcriptional Signatures in Response to Listeria monocytogenes Infection Reveals Temporal Changes That Result from Type I Interferon Signaling.ESX-1 exploits type I IFN-signalling to promote a regulatory macrophage phenotype refractory to IFNγ-mediated autophagy and growth restriction of intracellular mycobacteria.HLA antigen and NK cell activating ligand expression in malignant cells: a story of loss or acquisitionEarly events regulating immunity and pathogenesis during Listeria monocytogenes infectionTranscriptional Profiling in Experimental Visceral Leishmaniasis Reveals a Broad Splenic Inflammatory Environment that Conditions Macrophages toward a Disease-Promoting Phenotype.Innate immune function by Toll-like receptors: distinct responses in newborns and the elderly.Host-directed therapy of tuberculosis based on interleukin-1 and type I interferon crosstalk.Safety and Immunogenicity of the Recombinant BCG Vaccine AERAS-422 in Healthy BCG-naïve Adults: A Randomized, Active-controlled, First-in-human Phase 1 Trial.Type I IFNs downregulate myeloid cell IFN-γ receptor by inducing recruitment of an early growth response 3/NGFI-A binding protein 1 complex that silences ifngr1 transcription.TPL-2-ERK1/2 signaling promotes host resistance against intracellular bacterial infection by negative regulation of type I IFN productionBeneficial innate signaling interference for antibacterial responses by a Toll-like receptor-mediated enhancement of the MKP-IRF3 axis.Type I interferons induced by radiation therapy mediate recruitment and effector function of CD8(+) T cellsThe interferons and their receptors--distribution and regulation.Subversion and Utilization of Host Innate Defense by Leishmania amazonensis.The price of immunity.Age-dependent differences in systemic and cell-autonomous immunity to L. monocytogenes.Tracing innate immune defences along the path of Listeria monocytogenes infection.A trip in the "New Microbiology" with the bacterial pathogen Listeria monocytogenes.Giving CD4+ T cells the slip: viral interference with MHC class II-restricted antigen processing and presentation.Microparticles released from Mycobacterium tuberculosis-infected human macrophages contain increased levels of the type I interferon inducible proteins including ISG15.Critical role of the endogenous interferon ligand-receptors in type I and type II interferons response.Effects of type I interferons in malariaBacteria tune interferon responses by playing with chromatin
P2860
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P2860
Antagonistic crosstalk between type I and II interferons and increased host susceptibility to bacterial infections
description
2010 nî lūn-bûn
@nan
2010 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Antagonistic crosstalk between ...... bility to bacterial infections
@ast
Antagonistic crosstalk between ...... bility to bacterial infections
@en
Antagonistic crosstalk between ...... bility to bacterial infections
@nl
type
label
Antagonistic crosstalk between ...... bility to bacterial infections
@ast
Antagonistic crosstalk between ...... bility to bacterial infections
@en
Antagonistic crosstalk between ...... bility to bacterial infections
@nl
prefLabel
Antagonistic crosstalk between ...... bility to bacterial infections
@ast
Antagonistic crosstalk between ...... bility to bacterial infections
@en
Antagonistic crosstalk between ...... bility to bacterial infections
@nl
P2093
P2860
P356
P1433
P1476
Antagonistic crosstalk between ...... bility to bacterial infections
@en
P2093
Jessica Humann
Krista K Hill
Laurel L Lenz
Manira Rayamajhi
Staci Kearney
P2860
P304
P356
10.4161/VIRU.1.5.12787
P577
2010-09-01T00:00:00Z