Macrophage expression of active MMP-9 induces acute plaque disruption in apoE-deficient mice - Wikidata - awgldk - TriplyDB

Macrophage expression of active MMP-9 induces acute plaque disruption in apoE-deficient mice

Macrophage expression of active MMP-9 induces acute plaque disruption in apoE-deficient mice

http://www.wikidata.org/entity/Q34236802

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Diverse roles of macrophages in atherosclerosis: from inflammatory biology to biomarker discovery Matrix Metalloproteinases in Non-Neoplastic Disorders.Perspectives for Monocyte/Macrophage-Based Diagnostics of Chronic Inflammation Role of vascular smooth muscle cell in the inflammation of atherosclerosis Diverse functions of matrix metalloproteinases during fibrosis Perspectives on reprograming cancer-associated dendritic cells for anti-tumor therapies The Role of Different Monocyte Subsets in the Pathogenesis of Atherosclerosis and Acute Coronary Syndromes Molecular Imaging of Vulnerable Atherosclerotic Plaques in Animal Models.The Roles of CD137 Signaling in Atherosclerosis Biochemistry and molecular biology of gelatinase B or matrix metalloproteinase-9 (MMP-9): the next decade Matrix Metalloproteinase 9 (MMP-9) Regulates Vein Wall Biomechanics in Murine Thrombus Resolution Acrolein activates matrix metalloproteinases by increasing reactive oxygen species in macrophages Macrophages in the pathogenesis of atherosclerosis Molecular mechanisms of collagen isotype-specific modulation of smooth muscle cell phenotype A dynamic model of calcific nodule destabilization in response to monocyte- and oxidized lipid-induced matrix metalloproteinases.GATA3 suppresses metastasis and modulates the tumour microenvironment by regulating microRNA-29b expression.Elastin fragmentation in atherosclerotic mice leads to intraplaque neovascularization, plaque rupture, myocardial infarction, stroke, and sudden death.MMP-9 sheds the beta2 integrin subunit (CD18) from macrophages.Fine ambient air particulate matter exposure induces molecular alterations associated with vascular disease progression within plaques of atherosclerotic susceptible mice.Interleukin-10 overexpression in macrophages suppresses atherosclerosis in hyperlipidemic mice.Allele-specific regulation of matrix metalloproteinase-3 gene by transcription factor NFkappaB Human macrophage foam cells degrade atherosclerotic plaques through cathepsin K mediated processes Accumulation of gene polymorphisms related to plaque disruption and thrombosis is associated with cerebral infarction in subjects with type 2 diabetes.Lysophosphatidic Acid Is Associated with Atherosclerotic Plaque Instability by Regulating NF-κB Dependent Matrix Metalloproteinase-9 Expression via LPA2 in Macrophages.Toll-like receptor 7 deficiency protects apolipoprotein E-deficient mice from diet-induced atherosclerosis A macrophage cell model for selective metalloproteinase inhibitor design Targeting pancreatic cancer with magneto-fluorescent theranostic gold nanoshells.Inflammation and the osteogenic regulation of vascular calcification: a review and perspective.Nitro-fatty acids reduce atherosclerosis in apolipoprotein E-deficient mice.ADAMTS-7 is associated with a high-risk plaque phenotype in human atherosclerosis.Emerging applications of nanotechnology for the diagnosis and management of vulnerable atherosclerotic plaques.Overexpression of urokinase by plaque macrophages causes histological features of plaque rupture and increases vascular matrix metalloproteinase activity in aged apolipoprotein e-null mice Arterial colony stimulating factor-1 influences atherosclerotic lesions by regulating monocyte migration and apoptosis.Suppressed accumulation of cerebral amyloid {beta} peptides in aged transgenic Alzheimer's disease mice by transplantation with wild-type or prostaglandin E2 receptor subtype 2-null bone marrow A comparison of vascular effects from complex and individual air pollutants indicates a role for monoxide gases and volatile hydrocarbons.Inducible transgenes under the control of the hCD68 promoter identifies mouse macrophages with a distribution that differs from the F4/80 - and CSF-1R-expressing populations.Overexpression of MMP-9 and its inhibitors in blood mononuclear cells after myocardial infarction--is it associated with depressive symptomatology?Smooth muscle cell-specific insulin-like growth factor-1 overexpression in Apoe-/- mice does not alter atherosclerotic plaque burden but increases features of plaque stability Size- and charge-dependent non-specific uptake of PEGylated nanoparticles by macrophages.Human CD68 promoter GFP transgenic mice allow analysis of monocyte to macrophage differentiation in vivo.

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Macrophage expression of active MMP-9 induces acute plaque disruption in apoE-deficient mice

http://www.wikidata.org/entity/Q34236802

description

2005 nî lūn-bûn

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2005 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

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2005 թվականի դեկտեմբերին հրատարակված գիտական հոդված

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Macrophage expression of activ ...... ruption in apoE-deficient mice

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Macrophage expression of activ ...... ruption in apoE-deficient mice

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Macrophage expression of activ ...... ruption in apoE-deficient mice

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Macrophage expression of activ ...... ruption in apoE-deficient mice

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Macrophage expression of activ ...... ruption in apoE-deficient mice

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Journal of Clinical Investigation

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Macrophage expression of activ ...... ruption in apoE-deficient mice

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P2093

Elaine W Raines

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Ivan G Gomez

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Paul T Wille

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Peter J Gough

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P2860

SV40-transformed human lung fibroblasts secrete a 92-kDa type IV collagenase which is identical to that secreted by normal human macrophages

Reduction of atherosclerosis in mice by inhibition of CD40 signalling

Targeted gene disruption of matrix metalloproteinase-9 (gelatinase B) suppresses development of experimental abdominal aortic aneurysms

Recruitment of stem and progenitor cells from the bone marrow niche requires MMP-9 mediated release of kit-ligand

Activation of matrix metalloproteinase-9 (MMP-9) via a converging plasmin/stromelysin-1 cascade enhances tumor cell invasion

Increased atherosclerosis in myeloperoxidase-deficient mice

The peroxisome proliferator-activated receptor-gamma is a negative regulator of macrophage activation

S-nitrosylation of matrix metalloproteinases: signaling pathway to neuronal cell death.

Atherosclerotic plaque rupture in the apolipoprotein E knockout mouse.

Plaque pathology and coronary thrombosis in the pathogenesis of acute coronary syndromes.

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10.1172/JCI25074

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1

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1319218

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