Pioglitazone-induced reductions in atherosclerosis occur via smooth muscle cell-specific interaction with PPAR{gamma}.
about
Polyacetylenes from Notopterygium incisum--new selective partial agonists of peroxisome proliferator-activated receptor-gammaKalirin promotes neointimal hyperplasia by activating Rac in smooth muscle cellsSmooth Muscle Peroxisome Proliferator-Activated Receptor γ Plays a Critical Role in Formation and Rupture of Cerebral Aneurysms in Mice In VivoMinireview: Pathophysiological roles of the TR4 nuclear receptor: lessons learned from mice lacking TR4Genetic Variant of Kalirin Gene Is Associated with Ischemic Stroke in a Chinese Han Population.Pioglitazone improves the cardio-ankle vascular index in patients with type 2 diabetes mellitus treated with metformin.Role of vascular smooth muscle PPARγ in regulating AT1 receptor signaling and angiotensin II-dependent hypertension.Interference with peroxisome proliferator-activated receptor-γ in vascular smooth muscle causes baroreflex impairment and autonomic dysfunction.Regional variation in aortic AT1b receptor mRNA abundance is associated with contractility but unrelated to atherosclerosis and aortic aneurysmsDepletion of endothelial or smooth muscle cell-specific angiotensin II type 1a receptors does not influence aortic aneurysms or atherosclerosis in LDL receptor deficient mice.Endothelial cell-specific deficiency of Ang II type 1a receptors attenuates Ang II-induced ascending aortic aneurysms in LDL receptor-/- mice.TR4 nuclear receptor enhances prostate cancer initiation via altering the stem cell population and EMT signals in the PPARG-deleted prostate cells.Regulation of peroxisome proliferator-activated receptor-γ by angiotensin II via transforming growth factor-β1-activated p38 mitogen-activated protein kinase in aortic smooth muscle cells.Thrombospondin-1 (TSP1) contributes to the development of vascular inflammation by regulating monocytic cell motility in mouse models of abdominal aortic aneurysm.Animal models of atherosclerosis.Pro- and antiatherogenic effects of a dominant-negative P465L mutation of peroxisome proliferator-activated receptor-γ in apolipoprotein E-Null mice.Novel mechanisms of abdominal aortic aneurysmsLoss of perivascular adipose tissue on peroxisome proliferator-activated receptor-γ deletion in smooth muscle cells impairs intravascular thermoregulation and enhances atherosclerosisPioglitazone, a PPARγ agonist, attenuates PDGF-induced vascular smooth muscle cell proliferation through AMPK-dependent and AMPK-independent inhibition of mTOR/p70S6K and ERK signaling.Disruption of endothelial peroxisome proliferator-activated receptor γ accelerates diet-induced atherogenesis in LDL receptor-null mice.Dominant negative PPARγ promotes atherosclerosis, vascular dysfunction, and hypertension through distinct effects in endothelium and vascular muscleAtherosclerosis following renal injury is ameliorated by pioglitazone and losartan via macrophage phenotype.Amlodipine reduces AngII-induced aortic aneurysms and atherosclerosis in hypercholesterolemic micePioglitazone Ameliorates Smooth Muscle Cell Proliferation in Cuff-Induced Neointimal Formation by Both Adiponectin-Dependent and -Independent Pathways.Loss of neurotrophin-3 from smooth muscle disrupts vagal gastrointestinal afferent signaling and satiation.Agonistic induction of PPARγ reverses cigarette smoke-induced emphysema.Involvement of the renin-angiotensin system in abdominal and thoracic aortic aneurysms.The Actin-Binding Protein Drebrin Inhibits Neointimal Hyperplasia.G protein-coupled receptor kinase-5 attenuates atherosclerosis by regulating receptor tyrosine kinases and 7-transmembrane receptors.Lipid phosphate phosphatase 3 negatively regulates smooth muscle cell phenotypic modulation to limit intimal hyperplasia.Pioglitazone stabilizes atherosclerotic plaque by regulating the Th17/Treg balance in AMPK-dependent mechanisms.Telmisartan modulates mitochondrial function in vascular smooth muscle cells.Pioglitazone Attenuates Atherosclerosis in Diabetic Mice by Inhibition of Receptor for Advanced Glycation End-Product (RAGE) Signaling.Hypertension-Causing Mutation in Peroxisome Proliferator-Activated Receptor γ Impairs Nuclear Export of Nuclear Factor-κB p65 in Vascular Smooth Muscle.Pioglitazone Attenuates Injury-Induced Neointima Formation in Mouse Femoral Artery Partially through the Activation of AMP-Activated Protein Kinase.Defective autophagy in vascular smooth muscle cells enhances cell death and atherosclerosis
P2860
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P2860
Pioglitazone-induced reductions in atherosclerosis occur via smooth muscle cell-specific interaction with PPAR{gamma}.
description
2010 nî lūn-bûn
@nan
2010 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Pioglitazone-induced reduction ...... interaction with PPAR{gamma}.
@ast
Pioglitazone-induced reduction ...... interaction with PPAR{gamma}.
@en
Pioglitazone-induced reduction ...... interaction with PPAR{gamma}.
@nl
type
label
Pioglitazone-induced reduction ...... interaction with PPAR{gamma}.
@ast
Pioglitazone-induced reduction ...... interaction with PPAR{gamma}.
@en
Pioglitazone-induced reduction ...... interaction with PPAR{gamma}.
@nl
prefLabel
Pioglitazone-induced reduction ...... interaction with PPAR{gamma}.
@ast
Pioglitazone-induced reduction ...... interaction with PPAR{gamma}.
@en
Pioglitazone-induced reduction ...... interaction with PPAR{gamma}.
@nl
P2093
P2860
P1433
P1476
Pioglitazone-induced reduction ...... interaction with PPAR{gamma}.
@en
P2093
Alan Daugherty
Dennis Bruemmer
Talha Ijaz
Venkateswaran Subramanian
P2860
P304
P356
10.1161/CIRCRESAHA.110.219089
P577
2010-08-26T00:00:00Z