Motor deficits are triggered by reperfusion-reoxygenation injury as diagnosed by MRI and by a mechanism involving oxidants.

Motor deficits are triggered by reperfusion-reoxygenation injury as diagnosed by MRI and by a mechanism involving oxidants.

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http://www.wikidata.org/entity/Q34238262
Mn porphyrin-based SOD mimic, MnTnHex-2-PyP(5+), and non-SOD mimic, MnTBAP(3-), suppressed rat spinal cord ischemia/reperfusion injury via NF-κB pathwaysPathophysiology of glia in perinatal white matter injury.Sex differences in brain MRI abnormalities and neurodevelopmental outcomes in a rat model of neonatal hypoxia-ischemia.White matter injury in the preterm infant: pathology and mechanisms.Comprehensive pharmacokinetic studies and oral bioavailability of two Mn porphyrin-based SOD mimics, MnTE-2-PyP5+ and MnTnHex-2-PyP5+.Developmental susceptibility of neurons to transient tetrahydrobiopterin insufficiency and antenatal hypoxia-ischemia in fetal rabbits.Robust rat pulmonary radioprotection by a lipophilic Mn N-alkylpyridylporphyrin, MnTnHex-2-PyP(5+).Design, mechanism of action, bioavailability and therapeutic effects of mn porphyrin-based redox modulators.Therapeutic potential of peroxynitrite decomposition catalysts: a patent review.White Matter Injury of Prematurity: Its Mechanisms and Clinical Features.Tetrahydrobiopterin in antenatal brain hypoxia-ischemia-induced motor impairments and cerebral palsy.Placental perfusion in uterine ischemia model as evaluated by dynamic contrast enhanced MRI.On the use of fluorescence lifetime imaging and dihydroethidium to detect superoxide in intact animals and ex vivo tissues: a reassessment.Spinal cord injury in hypertonic newborns after antenatal hypoxia-ischemia in a rabbit model of cerebral palsy.Concurrent decrease of brain white matter tracts' thicknesses and fractional anisotropy after antenatal hypoxia-ischemia detected with tract-based spatial statistics analysis.Post-Irradiation Treatment with a Superoxide Dismutase Mimic, MnTnHex-2-PyP5+, Mitigates Radiation Injury in the Lungs of Non-Human Primates after Whole-Thorax Exposure to Ionizing Radiation.
P2860

Motor deficits are triggered by reperfusion-reoxygenation injury as diagnosed by MRI and by a mechanism involving oxidants.

Item
http://www.wikidata.org/entity/Q34238262
description
2012 nî lūn-bûn
@nan
2012 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2012 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
name
Motor deficits are triggered b ...... mechanism involving oxidants.
@ast
Motor deficits are triggered b ...... mechanism involving oxidants.
@en
Motor deficits are triggered b ...... mechanism involving oxidants.
@nl
type
label
Motor deficits are triggered b ...... mechanism involving oxidants.
@ast
Motor deficits are triggered b ...... mechanism involving oxidants.
@en
Motor deficits are triggered b ...... mechanism involving oxidants.
@nl
prefLabel
Motor deficits are triggered b ...... mechanism involving oxidants.
@ast
Motor deficits are triggered b ...... mechanism involving oxidants.
@en
Motor deficits are triggered b ...... mechanism involving oxidants.
@nl
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Motor deficits are triggered b ...... mechanism involving oxidants.
@en
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10.1523/JNEUROSCI.5986-11.2012
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