Inhibition of glycogen synthase kinase-3 activity triggers an apoptotic response in pancreatic cancer cells through JNK-dependent mechanisms.
about
Molecular targets for the treatment of pancreatic cancer: Clinical and experimental studiesGSK-3 as potential target for therapeutic intervention in cancerAdaptive cellular stress pathways as therapeutic targets of dietary phytochemicals: focus on the nervous systemToll-like receptor 4-mediated signaling participates in apoptosis of hippocampal neurons.Lithium inhibits tumorigenic potential of PDA cells through targeting hedgehog-GLI signaling pathway.The roles of Notch3 on the cell proliferation and apoptosis induced by CHIR99021 in NSCLC cell lines: a functional link between Wnt and Notch signaling pathwaysThe MEK/ERK pathway promotes NOTCH signalling in pancreatic cancer cells.Inhibition of glycogen synthase kinase-3 beta induces apoptosis and mitotic catastrophe by disrupting centrosome regulation in cancer cells.Lithium inhibits tumor lymphangiogenesis and metastasis through the inhibition of TGFBIp expression in cancer cellsRole of glycogen synthase kinase-3β inhibitor AZD1080 in ovarian cancer.Beneficial role of overexpression of TFPI-2 on tumour progression in human small cell lung cancer.Lithium chloride and inhibition of glycogen synthase kinase 3β as a potential therapy for serous ovarian cancer.Glycogen Synthase Kinase 3β in Pancreatic Cancer and its Implications in Chemotherapy and Radiation Therapy.The selective PI3Kα inhibitor BYL719 as a novel therapeutic option for neuroendocrine tumors: Results from multiple cell line models.Glycogen synthase kinase-3 (GSK3) inhibition induces prosurvival autophagic signals in human pancreatic cancer cells.GSK-3α promotes oncogenic KRAS function in pancreatic cancer via TAK1-TAB stabilization and regulation of noncanonical NF-κB.Animal models of arrhythmogenic right ventricular cardiomyopathy: what have we learned and where do we go? Insight for therapeutics.C-terminal deletion of NOTCH1 intracellular domain (N1ICD) increases its stability but does not amplify and recapitulate N1ICD-dependent signallingPOPX2 phosphatase regulates apoptosis through the TAK1-IKK-NF-κB pathway.GSK3β mediates pancreatic cancer cell invasion in vitro via the CXCR4/MMP-2 Pathway.Three-dimensional organoid culture reveals involvement of Wnt/β-catenin pathway in proliferation of bladder cancer cells.Glycogen synthase kinase 3 regulates IL-1β mediated iNOS expression in hepatocytes by down-regulating c-Jun.
P2860
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P2860
Inhibition of glycogen synthase kinase-3 activity triggers an apoptotic response in pancreatic cancer cells through JNK-dependent mechanisms.
description
2011 nî lūn-bûn
@nan
2011 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
Inhibition of glycogen synthas ...... ough JNK-dependent mechanisms.
@ast
Inhibition of glycogen synthas ...... ough JNK-dependent mechanisms.
@en
Inhibition of glycogen synthas ...... ough JNK-dependent mechanisms.
@nl
type
label
Inhibition of glycogen synthas ...... ough JNK-dependent mechanisms.
@ast
Inhibition of glycogen synthas ...... ough JNK-dependent mechanisms.
@en
Inhibition of glycogen synthas ...... ough JNK-dependent mechanisms.
@nl
prefLabel
Inhibition of glycogen synthas ...... ough JNK-dependent mechanisms.
@ast
Inhibition of glycogen synthas ...... ough JNK-dependent mechanisms.
@en
Inhibition of glycogen synthas ...... ough JNK-dependent mechanisms.
@nl
P2093
P356
P1433
P1476
Inhibition of glycogen synthas ...... rough JNK-dependent mechanisms
@en
P2093
Benoît Marchand
Isabelle Tremblay
Sébastien Cagnol
P304
P356
10.1093/CARCIN/BGR309
P407
P577
2011-12-26T00:00:00Z