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Apolipoprotein A-I: insights from redox proteomics for its role in neurodegenerationEarly Postnatal Lipopolysaccharide Exposure Leads to Enhanced Neurogenesis and Impaired Communicative Functions in RatsThe ELGAN study of the brain and related disorders in extremely low gestational age newborns.Inflammation-related proteins in the blood of extremely low gestational age newborns. The contribution of inflammation to the appearance of developmental regulation.Mechanisms of injury to white matter adjacent to a large intraventricular hemorrhage in the preterm brain.Cytokines and perinatal brain damage.Overweight worsens apoptosis, neuroinflammation and blood-brain barrier damage after hypoxic ischemia in neonatal brain through JNK hyperactivation.TNFR1-JNK signaling is the shared pathway of neuroinflammation and neurovascular damage after LPS-sensitized hypoxic-ischemic injury in the immature brain.Animal models of periventricular leukomalacia.Relationships among the concentrations of 25 inflammation-associated proteins during the first postnatal weeks in the blood of infants born before the 28th week of gestationShort and long-term analysis and comparison of neurodegeneration and inflammatory cell response in the ipsilateral and contralateral hemisphere of the neonatal mouse brain after hypoxia/ischemia.JNK signaling is the shared pathway linking neuroinflammation, blood-brain barrier disruption, and oligodendroglial apoptosis in the white matter injury of the immature brainDeterminants of Indices of Cerebral Volume in Former Very Premature Infants at Term Equivalent AgeCXCL5 signaling is a shared pathway of neuroinflammation and blood-brain barrier injury contributing to white matter injury in the immature brain.Low-dose lipopolysaccharide selectively sensitizes hypoxic ischemia-induced white matter injury in the immature brainFetal inflammatory response and brain injury in the preterm newborn.Cerebral microvascular damage occurs early after hypoxia-ischemia via nNOS activation in the neonatal brain.Inflammatory injury to the neonatal brain - what can we do?White matter damage and chemokine induction in developing rat brain after intrauterine infection.Brain injury induced by intracerebral injection of interleukin-1beta and tumor necrosis factor-alpha in the neonatal rat.Effect of erythropoietin combined with hypothermia on serum tau protein levels and neurodevelopmental outcome in neonates with hypoxic-ischemic encephalopathy.Entry and distribution of microglial cells in human embryonic and fetal cerebral cortex.Antenatal mycoplasma infection, the fetal inflammatory response and cerebral white matter damage in very-low-birthweight infants.Adoptive transfer of T regulatory cells inhibits lipopolysaccharide-induced inflammation in fetal brain tissue in a late-pregnancy preterm birth mouse model.Editorial: White blood cells matter in neonatal white-matter injury.
P2860
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P2860
description
2001 nî lūn-bûn
@nan
2001 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2001 թվականի հունիսին հրատարակված գիտական հոդված
@hy
2001年の論文
@ja
2001年論文
@yue
2001年論文
@zh-hant
2001年論文
@zh-hk
2001年論文
@zh-mo
2001年論文
@zh-tw
2001年论文
@wuu
name
Do white cells matter in white matter damage?
@ast
Do white cells matter in white matter damage?
@en
Do white cells matter in white matter damage?
@nl
type
label
Do white cells matter in white matter damage?
@ast
Do white cells matter in white matter damage?
@en
Do white cells matter in white matter damage?
@nl
prefLabel
Do white cells matter in white matter damage?
@ast
Do white cells matter in white matter damage?
@en
Do white cells matter in white matter damage?
@nl
P2093
P1476
Do white cells matter in white matter damage?
@en
P2093
P304
P356
10.1016/S0166-2236(00)01811-7
P577
2001-06-01T00:00:00Z