Hematopoietic miR155 deficiency enhances atherosclerosis and decreases plaque stability in hyperlipidemic mice. - Wikidata - awgldk - TriplyDB

Hematopoietic miR155 deficiency enhances atherosclerosis and decreases plaque stability in hyperlipidemic mice.

Hematopoietic miR155 deficiency enhances atherosclerosis and decreases plaque stability in hyperlipidemic mice.

http://www.wikidata.org/entity/Q34256848

about

Micromanaging abdominal aortic aneurysms Noncoding RNAs regulate NF-κB signaling to modulate blood vessel inflammation MicroRNAs in flow-dependent vascular remodelling All's well that transcribes well: non-coding RNAs and post-stroke brain damage Biomechanical factors and macrophages in plaque stability MiR-155 has a protective role in the development of non-alcoholic hepatosteatosis in mice.MicroRNA-155 deficiency results in decreased macrophage inflammation and attenuated atherogenesis in apolipoprotein E-deficient mice.Noncoding RNAs and atherosclerosis Role of flow-sensitive microRNAs in endothelial dysfunction and atherosclerosis: mechanosensitive athero-miRs ALK5 and ALK1 play antagonistic roles in transforming growth factor β-induced podosome formation in aortic endothelial cells.Epigenetic enzymes are the therapeutic targets for CD4(+)CD25(+/high)Foxp3(+) regulatory T cells Decreased serum level of miR-146a as sign of chronic inflammation in type 2 diabetic patients Upregulation of miR-142-5p in atherosclerotic plaques and regulation of oxidized low-density lipoprotein-induced apoptosis in macrophages Kruppel-Like Factor 2-Mediated Suppression of MicroRNA-155 Reduces the Proinflammatory Activation of Macrophages.Atherosclerosis.MicroRNA-155 promotes atherosclerosis by repressing Bcl6 in macrophages miR-155 acts as an anti-inflammatory factor in atherosclerosis-associated foam cell formation by repressing calcium-regulated heat stable protein 1.MicroRNA Regulation of Atherosclerosis.MicroRNA in cardiovascular calcification: focus on targets and extracellular vesicle delivery mechanisms.'Atherothrombosis-associated microRNAs in Antiphospholipid syndrome and Systemic Lupus Erythematosus patients'Regulation of SIRT1 by oxidative stress-responsive miRNAs and a systematic approach to identify its role in the endothelium.Endothelial microRNAs and atherosclerosis.Massive analysis of cDNA Ends (MACE) and miRNA expression profiling identifies proatherogenic pathways in chronic kidney disease MicroRNA-492 reverses high glucose-induced insulin resistance in HUVEC cells through targeting resistin.Modulation of macrophage activation and programming in immunity.Pathogenic arterial remodeling: the good and bad of microRNAs.Unlocking the door to new therapies in cardiovascular disease: microRNAs hold the key.MicroRNAs as therapeutic targets in atherosclerosis.miRNAs in atherosclerotic plaque initiation, progression, and rupture.microRNAs in Cerebrovascular Disease.The multifactorial nature of microRNAs in vascular remodelling.MicroRNA regulation of macrophages in human pathologies.Nanomedicine Meets microRNA: Current Advances in RNA-Based Nanotherapies for Atherosclerosis.MicroRNAs in the Pathobiology and Therapy of Atherosclerosis.Cell Death in the Vessel Wall: The Good, the Bad, the Ugly.Bone marrow-specific caspase-1/11 deficiency inhibits atherosclerosis development in Ldlr(-/-) mice.Promising landscape for regulating macrophage polarization: epigenetic viewpoint Elevated plasma miRNA-122, -140-3p, -720, -2861, and -3149 during early period of acute coronary syndrome are derived from peripheral blood mononuclear cells.MicroRNA-155 Regulates ROS Production, NO Generation, Apoptosis and Multiple Functions of Human Brain Microvessel Endothelial Cells Under Physiological and Pathological Conditions.Non-coding RNAs in cardiovascular disease: pathological relevance and emerging role as biomarkers and therapeutics.

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P2860

Hematopoietic miR155 deficiency enhances atherosclerosis and decreases plaque stability in hyperlipidemic mice.

http://www.wikidata.org/entity/Q34256848

description

2012 nî lūn-bûn

@nan

2012 թուականի Ապրիլին հրատարակուած գիտական յօդուած

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2012 թվականի ապրիլին հրատարակված գիտական հոդված

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2012年の論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年论文

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name

Hematopoietic miR155 deficienc ...... bility in hyperlipidemic mice.

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Hematopoietic miR155 deficienc ...... bility in hyperlipidemic mice.

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Hematopoietic miR155 deficienc ...... bility in hyperlipidemic mice.

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type

label

Hematopoietic miR155 deficienc ...... bility in hyperlipidemic mice.

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Hematopoietic miR155 deficienc ...... bility in hyperlipidemic mice.

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Hematopoietic miR155 deficienc ...... bility in hyperlipidemic mice.

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prefLabel

Hematopoietic miR155 deficienc ...... bility in hyperlipidemic mice.

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Hematopoietic miR155 deficienc ...... bility in hyperlipidemic mice.

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Hematopoietic miR155 deficienc ...... bility in hyperlipidemic mice.

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Hematopoietic miR155 deficienc ...... ability in hyperlipidemic mice

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Blanche Schroen

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Chantal C H Pöttgens

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Emiel P C van der Vorst

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Ine M J Wolfs

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Lauran J Stöger

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Marion J J Gijbels

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Marjo M P C Donners

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Stephane Heymans

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P2860

Monocytes in atherosclerosis: subsets and functions

MicroRNA-155 promotes autoimmune inflammation by enhancing inflammatory T cell development

Foxp3-dependent microRNA155 confers competitive fitness to regulatory T cells by targeting SOCS1 protein

Requirement of bic/microRNA-155 for normal immune function

Monocyte and macrophage heterogeneity

Myeloid IκBα deficiency promotes atherogenesis by enhancing leukocyte recruitment to the plaques

MicroRNA-155 silencing enhances inflammatory response and lipid uptake in oxidized low-density lipoprotein-stimulated human THP-1 macrophages.

Cutting edge: the Foxp3 target miR-155 contributes to the development of regulatory T cells.

miR-155 gene: a typical multifunctional microRNA.

MicroRNA-125a-5p partly regulates the inflammatory response, lipid uptake, and ORP9 expression in oxLDL-stimulated monocyte/macrophages.

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10.1371/JOURNAL.PONE.0035877

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4

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Menno de Winther

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224899479

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22558252

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atherosclerosis

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3338496

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